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Modeling Pathogenesis of Primary Liver Cancer in Lineage-Specific Mouse Cell Types

Gastroenterology (New York, N.Y. 1943), 2013-07, Vol.145 (1), p.221-231

2013

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MYC Activates Stem-like Cell Potential in Hepatocarcinoma by a p53-Dependent Mechanism

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (20), p.5903-5913

2014

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DNMT1 is a required genomic regulator for murine liver histogenesis and regeneration

Hepatology (Baltimore, Md.), 2016-08, Vol.64 (2), p.582-598

2016

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Inhibition of cytoplasmic EZH2 induces antitumor activity through stabilization of the DLC1 tumor suppressor protein

Nature communications, 2021-12, Vol.12 (1), p.6941-15, Article 6941

2021

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DLC1 is the principal biologically-relevant down-regulated DLC family member in several cancers

Oncotarget, 2016-07, Vol.7 (29), p.45144-45157

2016

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Definition of Ubiquitination Modulator COP1 as a Novel Therapeutic Target in Human Hepatocellular Carcinoma

Cancer research (Chicago, Ill.), 2010-11, Vol.70 (21), p.8264-8269

2010

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GTF2IRD1 in Craniofacial Development of Humans and Mice

Science (American Association for the Advancement of Science), 2005-11, Vol.310 (5751), p.1184-1187

2005

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Cancer-Associated Point Mutations in the DLC1 Tumor Suppressor and Other Rho-GAPs Occur Frequently and Are Associated with Decreased Function

Cancer research (Chicago, Ill.), 2020-09, Vol.80 (17), p.3568-3579

2020

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DLC-1 gene inhibits human breast cancer cell growth and in vivo tumorigenicity

Oncogene, 2003-01, Vol.22 (3), p.445-450

2003

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MYC activates stem-like cell potential in hepatocarcinoma by a p53-dependent mechanism

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (20), p.5903-5913

2014

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DLC‐1:a Rho GTPase‐activating protein and tumour suppressor

Journal of cellular and molecular medicine, 2007-09, Vol.11 (5), p.1185-1207

2007

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Abstract 888: Inactivation of the DLC1 RhoGAP tumor suppressor by point mutation occurs commonly in human cancer and can result from Rho-dependent or Rho-independent mechanisms

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.888-888

2016

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Amplification and Overexpression of the EMS 1 Oncogene, a Possible Prognostic Marker, in Human Hepatocellular Carcinoma

The Journal of molecular diagnostics : JMD, 2003-02, Vol.5 (1), p.48-53

2003

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Cooperative antiproliferative effect of coordinated ectopic expression of DLC1 tumor suppressor protein and silencing of MYC oncogene expression in liver cancer cells: Therapeutic implications

Oncology letters, 2016-08, Vol.12 (2), p.1591-1596

2016

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DLC-1, a Rho GTPase-activating protein with tumor suppressor function, is essential for embryonic development

FEBS letters, 2005-02, Vol.579 (5), p.1191-1196

2005

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DLC1 suppresses distant dissemination of human hepatocellular carcinoma cells in nude mice through reduction of RhoA GTPase activity, actin cytoskeletal disruption and down-regulation of genes involved in metastasis

International journal of oncology, 2008-06, Vol.32 (6), p.1285-1291

2008

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SMAD5 Gene Expression, Rearrangements, Copy Number, Amplification at Fragile Site FRA5C in Human Hepatocellular Carcinoma

Neoplasia (New York, N.Y.), 2003-09, Vol.5 (5), p.390-396

2003

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Loss of miR-122 expression in liver cancer correlates with suppression of the hepatic phenotype and gain of metastatic properties

Oncogene, 2009-10, Vol.28 (40), p.3526-3536

2009

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Inactivation of the Dlc1 Gene Cooperates with Downregulation of p15INK4b and p16Ink4a, Leading to Neoplastic Transformation and Poor Prognosis in Human Cancer

Cancer research (Chicago, Ill.), 2012-11, Vol.72 (22), p.5900-5911

2012

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Mice with a Targeted Deletion of the Tetranectin Gene Exhibit a Spinal Deformity

Molecular and Cellular Biology, 2001-11, Vol.21 (22), p.7817-7825

2001

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Abstract 2508: Targeting mutant KRAS tumors by inhibiting cytoplasmic EZH2, which directly methylates and destabilizes the DLC1 tumor suppressor protein

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.2508-2508

2021

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Abstract 4359: Several cancer types are associated with increased expression and activity of the ECT2 RhoGEF and decreased expression and activity of the DLC1 RhoGAP, leading to increased RhoA activity

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.4359-4359

2018

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Rho GTPase activating protein cDNA on chromosome 13q12 is the deleted in liver cancer (DLC2) gene

Biochemical and biophysical research communications, 2004-03, Vol.315 (4), p.781-781

2004

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A Potential Role for Tetranectin in Mineralization during Osteogenesis

The Journal of cell biology, 1994-12, Vol.127 (6), p.1767-1775

1994

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DNA variants of DLC-1, a candidate tumor suppressor gene in human hepatocellular carcinoma

International journal of oncology, 2003-07, Vol.23 (1), p.133

2003

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Modeling Pathogenesis of Primary Liver Cancer in Lineage-Specific Mouse Cell Types

MYC Activates Stem-like Cell Potential in Hepatocarcinoma by a p53-Dependent Mechanism

DNMT1 is a required genomic regulator for murine liver histogenesis and regeneration

Inhibition of cytoplasmic EZH2 induces antitumor activity through stabilization of the DLC1 tumor suppressor protein

DLC1 is the principal biologically-relevant down-regulated DLC family member in several cancers

Definition of Ubiquitination Modulator COP1 as a Novel Therapeutic Target in Human Hepatocellular Carcinoma

GTF2IRD1 in Craniofacial Development of Humans and Mice

Cancer-Associated Point Mutations in the DLC1 Tumor Suppressor and Other Rho-GAPs Occur Frequently and Are Associated with Decreased Function

DLC-1 gene inhibits human breast cancer cell growth and in vivo tumorigenicity

MYC activates stem-like cell potential in hepatocarcinoma by a p53-dependent mechanism

DLC‐1:a Rho GTPase‐activating protein and tumour suppressor

Abstract 888: Inactivation of the DLC1 RhoGAP tumor suppressor by point mutation occurs commonly in human cancer and can result from Rho-dependent or Rho-independent mechanisms

Amplification and Overexpression of the EMS 1 Oncogene, a Possible Prognostic Marker, in Human Hepatocellular Carcinoma

Cooperative antiproliferative effect of coordinated ectopic expression of DLC1 tumor suppressor protein and silencing of MYC oncogene expression in liver cancer cells: Therapeutic implications

DLC-1, a Rho GTPase-activating protein with tumor suppressor function, is essential for embryonic development

DLC1 suppresses distant dissemination of human hepatocellular carcinoma cells in nude mice through reduction of RhoA GTPase activity, actin cytoskeletal disruption and down-regulation of genes involved in metastasis

SMAD5 Gene Expression, Rearrangements, Copy Number, Amplification at Fragile Site FRA5C in Human Hepatocellular Carcinoma

Loss of miR-122 expression in liver cancer correlates with suppression of the hepatic phenotype and gain of metastatic properties

Inactivation of the Dlc1 Gene Cooperates with Downregulation of p15INK4b and p16Ink4a, Leading to Neoplastic Transformation and Poor Prognosis in Human Cancer

Mice with a Targeted Deletion of the Tetranectin Gene Exhibit a Spinal Deformity

Abstract 2508: Targeting mutant KRAS tumors by inhibiting cytoplasmic EZH2, which directly methylates and destabilizes the DLC1 tumor suppressor protein

Abstract 4359: Several cancer types are associated with increased expression and activity of the ECT2 RhoGEF and decreased expression and activity of the DLC1 RhoGAP, leading to increased RhoA activity

Rho GTPase activating protein cDNA on chromosome 13q12 is the deleted in liver cancer (DLC2) gene

A Potential Role for Tetranectin in Mineralization during Osteogenesis

DNA variants of DLC-1, a candidate tumor suppressor gene in human hepatocellular carcinoma

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