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Methods Used to Evaluate Pain Behaviors in Rodents

Frontiers in molecular neuroscience, 2017-09, Vol.10, p.284-284

2017

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An animal model of oxaliplatin-induced cold allodynia reveals a crucial role for Nav1.6 in peripheral pain pathways

Pain (Amsterdam), 2013-09, Vol.154 (9), p.1749

2013

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Development of a high-throughput fluorescent no-wash sodium influx assay

PloS one, 2019-03, Vol.14 (3), p.e0213751-e0213751

2019

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The structure, dynamics and selectivity profile of a NaV1.7 potency-optimised huwentoxin-IV variant

PloS one, 2017-03, Vol.12 (3), p.e0173551

2017

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Natural Compounds Purified from the Leaves of Aristotelia chilensis: Makomakinol, a New Alkaloid and the Effect of Aristoteline and Hobartine on NaV Channels

International journal of molecular sciences, 2023-11, Vol.24 (21), p.15504

2023

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Ant venoms contain vertebrate-selective pain-causing sodium channel toxins

Nature communications, 2023-05, Vol.14 (1), p.2977-2977, Article 2977

2023

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Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain

Toxins, 2016-03, Vol.8 (3), p.78

2016

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Venom composition and pain-causing toxins of the Australian great carpenter bee Xylocopa aruana

Scientific reports, 2022-12, Vol.12 (1), p.22168-22168, Article 22168

2022

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Pain-causing stinging nettle toxins target TMEM233 to modulate NaV1.7 function

Nature communications, 2023-04, Vol.14 (1), p.2442-2442, Article 2442

2023

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Discovery and mode of action of a novel analgesic β-toxin from the African spider Ceratogyrus darlingi

PloS one, 2017, Vol.12 (9), p.e0182848-e0182848

2017

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Towards a generic prototyping approach for therapeutically-relevant peptides and proteins in a cell-free translation system

Nature communications, 2022-01, Vol.13 (1), p.260-260, Article 260

2022

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µ-Conotoxins Targeting the Human Voltage-Gated Sodium Channel Subtype NaV1.7

Toxins, 2022-08, Vol.14 (9), p.600

2022

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The pharmacology of voltage-gated sodium channel activators

Neuropharmacology, 2017-12, Vol.127, p.87-108

2017

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NaV1.7 as a pain target – From gene to pharmacology

Pharmacology & therapeutics (Oxford), 2017-04, Vol.172, p.73-100

2017

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The Tarantula Venom Peptide Eo1a Binds to the Domain II S3-S4 Extracellular Loop of Voltage-Gated Sodium Channel NaV1.8 to Enhance Activation

Frontiers in pharmacology, 2022-01, Vol.12, p.789570-789570

2022

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Discovery, Pharmacological Characterisation and NMR Structure of the Novel µ-Conotoxin SxIIIC, a Potent and Irreversible NaV Channel Inhibitor

Biomedicines, 2020-10, Vol.8 (10), p.391

2020

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Inflammatory and Neuropathic Gene Expression Signatures of Chemotherapy-Induced Neuropathy Induced by Vincristine, Cisplatin, and Oxaliplatin in C57BL/6J Mice

The journal of pain, 2020-01, Vol.21 (1-2), p.182-194

2020

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Sodium Channels and Venom Peptide Pharmacology

Advances in pharmacology (1990), 2017, Vol.79, p.67

2017

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Characterization of Synthetic Tf2 as a NaV1.3 Selective Pharmacological Probe

Biomedicines, 2020-06, Vol.8 (6), p.155

2020

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Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)

Human molecular genetics, 2024-01, Vol.33 (2), p.103-109

2024

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Recombinant production, bioconjugation and membrane binding studies ofPn3a, a selective NaV1.7 inhibitor

Biochemical pharmacology, 2020-11, Vol.181, p.114148-114148, Article 114148

2020

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Polygodial, a drimane sesquiterpenoid dialdehyde purified from Drimys winteri , inhibits voltage-gated sodium channels

Natural product research, 2022-12, Vol.36 (24), p.1-6323

2022

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Addition of K22 Converts Spider Venom Peptide Pme2a from an Activator to an Inhibitor of NaV1.7

Biomedicines, 2020-02, Vol.8 (2), p.37

2020

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Toxins as tools: Fingerprinting neuronal pharmacology

Neuroscience letters, 2018-07, Vol.679, p.4-14

2018

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Methods Used to Evaluate Pain Behaviors in Rodents

An animal model of oxaliplatin-induced cold allodynia reveals a crucial role for Nav1.6 in peripheral pain pathways

Development of a high-throughput fluorescent no-wash sodium influx assay

The structure, dynamics and selectivity profile of a NaV1.7 potency-optimised huwentoxin-IV variant

Natural Compounds Purified from the Leaves of Aristotelia chilensis: Makomakinol, a New Alkaloid and the Effect of Aristoteline and Hobartine on NaV Channels

Ant venoms contain vertebrate-selective pain-causing sodium channel toxins

Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain

Venom composition and pain-causing toxins of the Australian great carpenter bee Xylocopa aruana

Pain-causing stinging nettle toxins target TMEM233 to modulate NaV1.7 function

Discovery and mode of action of a novel analgesic β-toxin from the African spider Ceratogyrus darlingi

Towards a generic prototyping approach for therapeutically-relevant peptides and proteins in a cell-free translation system

µ-Conotoxins Targeting the Human Voltage-Gated Sodium Channel Subtype NaV1.7

The Snake with the Scorpion's Sting: Novel Three-Finger Toxin Sodium Channel Activators from the Venom of the Long-Glanded Blue Coral Snake (Calliophis bivirgatus)

The pharmacology of voltage-gated sodium channel activators

NaV1.7 as a pain target – From gene to pharmacology

The Tarantula Venom Peptide Eo1a Binds to the Domain II S3-S4 Extracellular Loop of Voltage-Gated Sodium Channel NaV1.8 to Enhance Activation

Discovery, Pharmacological Characterisation and NMR Structure of the Novel µ-Conotoxin SxIIIC, a Potent and Irreversible NaV Channel Inhibitor

Inflammatory and Neuropathic Gene Expression Signatures of Chemotherapy-Induced Neuropathy Induced by Vincristine, Cisplatin, and Oxaliplatin in C57BL/6J Mice

Sodium Channels and Venom Peptide Pharmacology

Characterization of Synthetic Tf2 as a NaV1.3 Selective Pharmacological Probe

Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)

Recombinant production, bioconjugation and membrane binding studies ofPn3a, a selective NaV1.7 inhibitor

Polygodial, a drimane sesquiterpenoid dialdehyde purified from Drimys winteri , inhibits voltage-gated sodium channels

Addition of K22 Converts Spider Venom Peptide Pme2a from an Activator to an Inhibitor of NaV1.7

Toxins as tools: Fingerprinting neuronal pharmacology

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