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Herpes Simplex Virus 1 (HSV-1) Infected Cell Protein 0 (ICP0) Targets of Ubiquitination during Productive Infection of Primary Adult Sensory Neurons

International journal of molecular sciences, 2023-02, Vol.24 (3), p.2931

2023

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Genome Sequence of Herpes Simplex Virus 1 Strain KOS

Journal of Virology, 2012-06, Vol.86 (11), p.6371-6372

2012

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Efficacy of an HSV-1 Neuro-Attenuated Vaccine in Mice Is Reduced by Preventing Viral DNA Replication

Viruses, 2022-04, Vol.14 (5), p.869

2022

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Genome Sequence of Herpes Simplex Virus 1 Strain McKrae

Journal of Virology, 2012-09, Vol.86 (17), p.9540-9541

2012

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HSV-1 strain McKrae is more neuroinvasive than HSV-1 KOS after corneal or vaginal inoculation in mice

Virus research, 2013-05, Vol.173 (2), p.436-440

2013

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hTERT extends the life of human fibroblasts without compromising type I interferon signaling

PloS one, 2013-03, Vol.8 (3), p.e58233-e58233

2013

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HSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate Immunity

Cells, 2014-05, Vol.3 (2), p.438-454

2014

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N-Terminal Phosphorylation Sites of Herpes Simplex Virus 1 ICP0 Differentially Regulate Its Activities and Enhance Viral Replication

Journal of Virology, 2013-02, Vol.87 (4), p.2109-2119

2013

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Two Overlapping Regions within the N-Terminal Half of the Herpes Simplex Virus 1 E3 Ubiquitin Ligase ICP0 Facilitate the Degradation and Dissociation of PML and Dissociation of Sp100 from ND10

Journal of Virology, 2013-12, Vol.87 (24), p.13287-13296

2013

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Inhibitors of One or More Cellular Aurora Kinases Impair the Replication of Herpes Simplex Virus 1 and Other DNA and RNA Viruses with Diverse Genomes and Life Cycles

Microbiology spectrum, 2023-02, Vol.11 (1), p.e0194322-e0194322

2023

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A 77 Amino Acid Region in the N-Terminal Half of the HSV-1 E3 Ubiquitin Ligase ICP0 Contributes to Counteracting an Established Type 1 Interferon Response

Microbiology spectrum, 2022-08, Vol.10 (4), p.e0059322-e0059322

2022

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HSV-1 ICP0 dimer domain adopts a novel β-barrel fold

Proteins, structure, function, and bioinformatics, 2024-02

2024

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Role of Nuclear Factor Y in Stress-Induced Activation of the Herpes Simplex Virus Type 1 ICP0 Promoter

Journal of Virology, 2010-01, Vol.84 (1), p.188-200

2010

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Simple and rapid high-throughput assay to identify HSV-1 ICP0 transactivation inhibitors

Antiviral research, 2021-10, Vol.194, p.105160-105160, Article 105160

2021

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A quantitative assay to monitor HSV-1 ICP0 ubiquitin ligase activity in vitro

Methods (San Diego, Calif.), 2015-11, Vol.90, p.3-7

2015

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Herpes Simplex Virus 1 ICP0 Phosphorylation Site Mutants Are Attenuated for Viral Replication and Impaired for Explant-Induced Reactivation

Journal of Virology, 2011-12, Vol.85 (23), p.12631-12637

2011

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Transient Expression of Herpes Simplex Virus Type 1 ICP22 Represses Viral Promoter Activity and Complements the Replication of an ICP22 Null Virus

Journal of Virology, 2009-09, Vol.83 (17), p.8733-8743

2009

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Herpes Simplex Virus 1 Mutant with Point Mutations in UL39 Is Impaired for Acute Viral Replication in Mice, Establishment of Latency, and Explant-Induced Reactivation

Journal of virology, 2018-04, Vol.92 (7)

2018

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Herpes Simplex Virus 1 ICP22 but Not US1.5 Is Required for Efficient Acute Replication in Mice and VICE Domain Formation

Journal of Virology, 2013-12, Vol.87 (24), p.13510-13519

2013

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Attenuated Herpes Simplex Virus 1 (HSV-1) Expressing a Mutant Form of ICP6 Stimulates a Strong Immune Response That Protects Mice against HSV-1-Induced Corneal Disease

Journal of virology, 2018-09, Vol.92 (17)

2018

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Phosphorylation Site Mutations Affect Herpes Simplex Virus Type 1 ICP0 Function

Journal of Virology, 2005-01, Vol.79 (2), p.1232-1243

2005

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The Cyclin-Dependent Kinase Inhibitor Roscovitine Inhibits the Transactivating Activity and Alters the Posttranslational Modification of Herpes Simplex Virus Type 1 ICP0

Journal of Virology, 2002-02, Vol.76 (3), p.1077-1088

2002

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A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes

Cells (Basel, Switzerland), 2014-12, Vol.3 (4), p.1131-1158

2014

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Herpes simplex virus 1 upregulates p35, alters CDK-5 localization, and stimulates CDK-5 kinase activity during acute infection in neurons

Journal of virology, 2015-05, Vol.89 (9), p.5171-5175

2015

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The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0

Journal of Virology, 2003-12, Vol.77 (23), p.12603-12616

2003

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Herpes Simplex Virus 1 (HSV-1) Infected Cell Protein 0 (ICP0) Targets of Ubiquitination during Productive Infection of Primary Adult Sensory Neurons

Genome Sequence of Herpes Simplex Virus 1 Strain KOS

Efficacy of an HSV-1 Neuro-Attenuated Vaccine in Mice Is Reduced by Preventing Viral DNA Replication

Genome Sequence of Herpes Simplex Virus 1 Strain McKrae

HSV-1 strain McKrae is more neuroinvasive than HSV-1 KOS after corneal or vaginal inoculation in mice

hTERT extends the life of human fibroblasts without compromising type I interferon signaling

HSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate Immunity

N-Terminal Phosphorylation Sites of Herpes Simplex Virus 1 ICP0 Differentially Regulate Its Activities and Enhance Viral Replication

Two Overlapping Regions within the N-Terminal Half of the Herpes Simplex Virus 1 E3 Ubiquitin Ligase ICP0 Facilitate the Degradation and Dissociation of PML and Dissociation of Sp100 from ND10

Inhibitors of One or More Cellular Aurora Kinases Impair the Replication of Herpes Simplex Virus 1 and Other DNA and RNA Viruses with Diverse Genomes and Life Cycles

A 77 Amino Acid Region in the N-Terminal Half of the HSV-1 E3 Ubiquitin Ligase ICP0 Contributes to Counteracting an Established Type 1 Interferon Response

HSV-1 ICP0 dimer domain adopts a novel β-barrel fold

Role of Nuclear Factor Y in Stress-Induced Activation of the Herpes Simplex Virus Type 1 ICP0 Promoter

Simple and rapid high-throughput assay to identify HSV-1 ICP0 transactivation inhibitors

A quantitative assay to monitor HSV-1 ICP0 ubiquitin ligase activity in vitro

Herpes Simplex Virus 1 ICP0 Phosphorylation Site Mutants Are Attenuated for Viral Replication and Impaired for Explant-Induced Reactivation

Transient Expression of Herpes Simplex Virus Type 1 ICP22 Represses Viral Promoter Activity and Complements the Replication of an ICP22 Null Virus

Herpes Simplex Virus 1 Mutant with Point Mutations in UL39 Is Impaired for Acute Viral Replication in Mice, Establishment of Latency, and Explant-Induced Reactivation

Herpes Simplex Virus 1 ICP22 but Not US1.5 Is Required for Efficient Acute Replication in Mice and VICE Domain Formation

Attenuated Herpes Simplex Virus 1 (HSV-1) Expressing a Mutant Form of ICP6 Stimulates a Strong Immune Response That Protects Mice against HSV-1-Induced Corneal Disease

Phosphorylation Site Mutations Affect Herpes Simplex Virus Type 1 ICP0 Function

The Cyclin-Dependent Kinase Inhibitor Roscovitine Inhibits the Transactivating Activity and Alters the Posttranslational Modification of Herpes Simplex Virus Type 1 ICP0

A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes

Herpes simplex virus 1 upregulates p35, alters CDK-5 localization, and stimulates CDK-5 kinase activity during acute infection in neurons

The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0

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