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Role of KCa3.1 Channels in Modulating Ca2+ Oscillations during Glioblastoma Cell Migration and Invasion

International journal of molecular sciences, 2018-09, Vol.19 (10), p.2970

2018

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Voltage-dependent gating in K channels: experimental results and quantitative models

Pflügers Archiv, 2020, Vol.472 (1), p.27-47

2020

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Hypoxia, Ion Channels and Glioblastoma Malignancy

Biomolecules (Basel, Switzerland), 2023-12, Vol.13 (12), p.1742

2023

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Structure, Gating and Basic Functions of the Ca2+-activated K Channel of Intermediate Conductance

Current neuropharmacology, 2018-01, Vol.16 (5), p.608-617

2018

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The Volume-Regulated Anion Channel in Glioblastoma

Cancers, 2019-03, Vol.11 (3), p.307

2019

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The inhibition of KCa3.1 channels activity reduces cell motility in glioblastoma derived cancer stem cells

PloS one, 2012-10, Vol.7 (10), p.e47825-e47825

2012

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The 70‐year search for the voltage‐sensing mechanism of ion channels

The Journal of physiology, 2022-07, Vol.600 (14), p.3227-3247

2022

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Pain Control by Proprioceptive and Exteroceptive Stimulation at the Trigeminal Level

Frontiers in physiology, 2018-08, Vol.9, p.1037-1037

2018

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Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype

Human molecular genetics, 2014-09, Vol.23 (18), p.4875-4886

2014

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Simulation of Gating Currents of the Shaker K Channel Using a Brownian Model of the Voltage Sensor

Biophysical journal, 2019-11, Vol.117 (10), p.2005-2019

2019

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Radiation Increases Functional KCa3.1 Expression and Invasiveness in Glioblastoma

Cancers, 2019-02, Vol.11 (3), p.279

2019

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Update on the implication of potassium channels in autism: K(+) channelautism spectrum disorder

Frontiers in cellular neuroscience, 2015-03, Vol.9, p.34-34

2015

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The differential expression of low-threshold K+ currents generates distinct firing patterns in different subtypes of adult mouse trigeminal ganglion neurones

The Journal of physiology, 2008-11, Vol.586 (21), p.5101-5118

2008

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A Calsequestrin-1 Mutation Associated with a Skeletal Muscle Disease Alters Sarcoplasmic Ca2+ Release

PloS one, 2016-05, Vol.11 (5), p.e0155516-e0155516

2016

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Identification of Key Signaling Molecules Involved in the Activation of the Swelling-Activated Chloride Current in Human Glioblastoma Cells

The Journal of membrane biology, 2014-01, Vol.247 (1), p.45-55

2014

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The CaMKII/MLC1 Axis Confers Ca2+-Dependence to Volume-Regulated Anion Channels (VRAC) in Astrocytes

Cells (Basel, Switzerland), 2022-08, Vol.11 (17), p.2656

2022

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Astrocytes-Derived Small Extracellular Vesicles Hinder Glioma Growth

Biomedicines, 2022-11, Vol.10 (11), p.2952

2022

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M2 Receptors Exert Analgesic Action on DRG Sensory Neurons by Negatively Modulating VR1 Activity

Journal of cellular physiology, 2014-06, Vol.229 (6), p.783-790

2014

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Experimental challenges in ion channel research: uncovering basic principles of permeation and gating in potassium channels

Advances in physics: X, 2022-01, Vol.7 (1)

2022

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Fifty years of gating currents and channel gating

The Journal of general physiology, 2023-08, Vol.155 (8), p.1

2023

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Ca2+‐dependent and Ca2+‐independent somatic release from trigeminal neurons

Journal of cellular physiology, 2019-07, Vol.234 (7), p.10977-10989

2019

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Ca 2+ -dependent and Ca 2+ -independent somatic release from trigeminal neurons

Journal of cellular physiology, 2019-07, Vol.234 (7), p.10977-10989

2019

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Ca2+‐activated K+ channels regulate cell volume in human glioblastoma cells

Journal of cellular physiology, 2023-09, Vol.238 (9), p.2120-2134

2023

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Piezo1 controls cell volume and migration by modulating swelling‐activated chloride current through Ca2+ influx

Journal of cellular physiology, 2022-03, Vol.237 (3), p.1857-1870

2022

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Current Neuropharmacology: Editorial; The Role of Ca2+-activated K+ Channels of Intermediate Conductance in Glioblastoma Malignancy

Current neuropharmacology, 2018, Vol.16 (5), p.607-607

2018

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Role of KCa3.1 Channels in Modulating Ca2+ Oscillations during Glioblastoma Cell Migration and Invasion

Voltage-dependent gating in K channels: experimental results and quantitative models

Hypoxia, Ion Channels and Glioblastoma Malignancy

Structure, Gating and Basic Functions of the Ca2+-activated K Channel of Intermediate Conductance

The Volume-Regulated Anion Channel in Glioblastoma

The inhibition of KCa3.1 channels activity reduces cell motility in glioblastoma derived cancer stem cells

The 70‐year search for the voltage‐sensing mechanism of ion channels

Pain Control by Proprioceptive and Exteroceptive Stimulation at the Trigeminal Level

Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype

Simulation of Gating Currents of the Shaker K Channel Using a Brownian Model of the Voltage Sensor

Radiation Increases Functional KCa3.1 Expression and Invasiveness in Glioblastoma

Update on the implication of potassium channels in autism: K(+) channelautism spectrum disorder

The differential expression of low-threshold K+ currents generates distinct firing patterns in different subtypes of adult mouse trigeminal ganglion neurones

A Calsequestrin-1 Mutation Associated with a Skeletal Muscle Disease Alters Sarcoplasmic Ca2+ Release

Identification of Key Signaling Molecules Involved in the Activation of the Swelling-Activated Chloride Current in Human Glioblastoma Cells

The CaMKII/MLC1 Axis Confers Ca2+-Dependence to Volume-Regulated Anion Channels (VRAC) in Astrocytes

Astrocytes-Derived Small Extracellular Vesicles Hinder Glioma Growth

M2 Receptors Exert Analgesic Action on DRG Sensory Neurons by Negatively Modulating VR1 Activity

Experimental challenges in ion channel research: uncovering basic principles of permeation and gating in potassium channels

Fifty years of gating currents and channel gating

Ca2+‐dependent and Ca2+‐independent somatic release from trigeminal neurons

Ca 2+ -dependent and Ca 2+ -independent somatic release from trigeminal neurons

Ca2+‐activated K+ channels regulate cell volume in human glioblastoma cells

Piezo1 controls cell volume and migration by modulating swelling‐activated chloride current through Ca2+ influx

Current Neuropharmacology: Editorial; The Role of Ca2+-activated K+ Channels of Intermediate Conductance in Glioblastoma Malignancy

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