UB Paderborn / Katalog / Suche / in CDI

Dysbiosis in intestinal inflammation: Cause or consequence

International journal of medical microbiology, 2016-08, Vol.306 (5), p.302-309

2016

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Functional relevance of microbiome signatures: The correlation era requires tools for consolidation

Journal of allergy and clinical immunology, 2017-04, Vol.139 (4), p.1092-1098

2017

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Neutralization of IL-1α ameliorates Crohn’s disease-like ileitis by functional alterations of the gut microbiome

Proceedings of the National Academy of Sciences - PNAS, 2019-12, Vol.116 (52), p.26717-26726

2019

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Mechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont Selection

Frontiers in immunology, 2015-11, Vol.6, p.555-555

2015

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Mechanism of protection of transepithelial barrier function by Lactobacillus salivarius: strain dependence and attenuation by bacteriocin production

American journal of physiology: Gastrointestinal and liver physiology, 2012-11, Vol.303 (9), p.G1029-G1041

2012

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Dysbiosis in Crohn's disease - Joint action of stochastic injuries and focal inflammation in the gut

Gut microbes, 2017-01, Vol.8 (1), p.53-58

2017

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Intestinal Stem Cell Niche Defects Result in Impaired 3D Organoid Formation in Mouse Models of Crohn's Disease-like Ileitis

Stem cell reports, 2020-08, Vol.15 (2), p.389-407

2020

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Death Receptor 3 Signaling Controls the Balance between Regulatory and Effector Lymphocytes in SAMP1/YitFc Mice with Crohn's Disease-Like Ileitis

Frontiers in immunology, 2018-03, Vol.9, p.362-362

2018

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Exposure of Bifidobacterium longum subsp. infantis to Milk Oligosaccharides Increases Adhesion to Epithelial Cells and Induces a Substantial Transcriptional Response

PloS one, 2013-06, Vol.8 (6), p.e67224

2013

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Molecular dialogue between the human gut microbiota and the host: a Lactobacillus and Bifidobacterium perspective

Cellular and molecular life sciences : CMLS, 2014, Vol.71 (2), p.183-203

2014

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Macrophage cytokine responses to commensal Gram-positive Lactobacillus salivarius strains are TLR2-independent and Myd88-dependent

Scientific reports, 2021-03, Vol.11 (1), p.5896-5896, Article 5896

2021

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Influence of Adhesion and Bacteriocin Production by Lactobacillus salivarius on the Intestinal Epithelial Cell Transcriptional Response

Applied and Environmental Microbiology, 2012-08, Vol.78 (15), p.5196-5203

2012

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Human Gut Microbiome Transplantation in Ileitis Prone Mice: A Tool for the Functional Characterization of the Microbiota in Inflammatory Bowel Disease Patients

Inflammatory bowel diseases, 2020-02, Vol.26 (3), p.347-359

2020

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Interleukin 33 Triggers Early Eosinophil-Dependent Events Leading to Metaplasia in a Chronic Model of Gastritis-Prone Mice

Gastroenterology (New York, N.Y. 1943), 2021-01, Vol.160 (1), p.302-316.e7

2021

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Enteroid Formation is Defective in SAMP/YitFc Mice with Crohn’s Disease-Like Ileitis Independently of Disease Severity and Microbial Colonization

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S623-S623

2017

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Death-Domain-Receptor 3 Deletion Normalizes Inflammatory Gene Expression and Prevents Ileitis in Experimental Crohn's Disease

Inflammatory bowel diseases, 2019-01, Vol.25 (1), p.14-26

2019

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Su1879 Spatial 3D-Stereomicroscopic, Microbial and Metabolic Characterization of Intestinal Villous Erosions and Ulcerations in Mice

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S578-S578

2016

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Prebiotic supplementation in frail older people affects specific gut microbiota taxa but not global diversity

Microbiome, 2019-03, Vol.7 (1), p.39-17, Article 39

2019

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Tu1770 - Role of Tl1A and its Receptor Dr3 in Regulating Intestinal Th9 Cell Differentiation in Crohn's Disease-Like Ileitis

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-1015-S-1015

2018

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P104 ROLE OF TL1A AND ITS RECEPTOR DR3 IN REGULATING INTESTINAL TH9 CELL DIFFERENTIATION IN CROHN’S DISEASE (CD)-LIKE ILEITIS

Gastroenterology (New York, N.Y. 1943), 2018-01, Vol.154 (1), p.S53-S53

2018

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P104 ROLE OF TL1A AND ITS RECEPTOR DR3 IN REGULATING INTESTINAL TH9 CELL DIFFERENTIATION IN CROHN'S DISEASE (CD)-LIKE ILEITIS

Inflammatory bowel diseases, 2018-01, Vol.24 (suppl_1), p.S37-S37

2018

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Tu1767 - Blockade of IL-1α Ameliorates Ileitis and DSS Induced Colitis in Samp1/Yitfc Mice by a Mechanism Involving Direct Modulation of the Gut Microbiome

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-1014-S-1014

2018

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Tu1812 - Imbalance of Effector and Suppressor T Cells in Samp1/Yitfc and TNFdare Mouse Models of Crohn's Disease-Like Ileitis

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-1026-S-1027

2018

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Su1997 - Enteroid Formation is Defective in SAMP/YitFc Mice with Crohn’s Disease-Like Ileitis Independently of Disease Severity and Microbial Colonization

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S623-S623

2017

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Su1890 – Predicted Fecal Microbiota Signatures are Associated with Response to Il-1α Neutralization in Samp1/Yitfc Mice

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-650-S-650

2019

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Dysbiosis in intestinal inflammation: Cause or consequence

Functional relevance of microbiome signatures: The correlation era requires tools for consolidation

Neutralization of IL-1α ameliorates Crohn’s disease-like ileitis by functional alterations of the gut microbiome

Mechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont Selection

Mechanism of protection of transepithelial barrier function by Lactobacillus salivarius: strain dependence and attenuation by bacteriocin production

Dysbiosis in Crohn's disease - Joint action of stochastic injuries and focal inflammation in the gut

Intestinal Stem Cell Niche Defects Result in Impaired 3D Organoid Formation in Mouse Models of Crohn's Disease-like Ileitis

Death Receptor 3 Signaling Controls the Balance between Regulatory and Effector Lymphocytes in SAMP1/YitFc Mice with Crohn's Disease-Like Ileitis

Exposure of Bifidobacterium longum subsp. infantis to Milk Oligosaccharides Increases Adhesion to Epithelial Cells and Induces a Substantial Transcriptional Response

Molecular dialogue between the human gut microbiota and the host: a Lactobacillus and Bifidobacterium perspective

Macrophage cytokine responses to commensal Gram-positive Lactobacillus salivarius strains are TLR2-independent and Myd88-dependent

Influence of Adhesion and Bacteriocin Production by Lactobacillus salivarius on the Intestinal Epithelial Cell Transcriptional Response

Human Gut Microbiome Transplantation in Ileitis Prone Mice: A Tool for the Functional Characterization of the Microbiota in Inflammatory Bowel Disease Patients

Interleukin 33 Triggers Early Eosinophil-Dependent Events Leading to Metaplasia in a Chronic Model of Gastritis-Prone Mice

Enteroid Formation is Defective in SAMP/YitFc Mice with Crohn’s Disease-Like Ileitis Independently of Disease Severity and Microbial Colonization

Death-Domain-Receptor 3 Deletion Normalizes Inflammatory Gene Expression and Prevents Ileitis in Experimental Crohn's Disease

Su1879 Spatial 3D-Stereomicroscopic, Microbial and Metabolic Characterization of Intestinal Villous Erosions and Ulcerations in Mice

Prebiotic supplementation in frail older people affects specific gut microbiota taxa but not global diversity

Tu1770 - Role of Tl1A and its Receptor Dr3 in Regulating Intestinal Th9 Cell Differentiation in Crohn's Disease-Like Ileitis

P104 ROLE OF TL1A AND ITS RECEPTOR DR3 IN REGULATING INTESTINAL TH9 CELL DIFFERENTIATION IN CROHN’S DISEASE (CD)-LIKE ILEITIS

P104 ROLE OF TL1A AND ITS RECEPTOR DR3 IN REGULATING INTESTINAL TH9 CELL DIFFERENTIATION IN CROHN'S DISEASE (CD)-LIKE ILEITIS

Tu1767 - Blockade of IL-1α Ameliorates Ileitis and DSS Induced Colitis in Samp1/Yitfc Mice by a Mechanism Involving Direct Modulation of the Gut Microbiome

Tu1812 - Imbalance of Effector and Suppressor T Cells in Samp1/Yitfc and TNFdare Mouse Models of Crohn's Disease-Like Ileitis

Su1997 - Enteroid Formation is Defective in SAMP/YitFc Mice with Crohn’s Disease-Like Ileitis Independently of Disease Severity and Microbial Colonization

Su1890 – Predicted Fecal Microbiota Signatures are Associated with Response to Il-1α Neutralization in Samp1/Yitfc Mice

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