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Modest overexpression of FOXO maintains cardiac proteostasis and ameliorates age‐associated functional decline

Aging cell, 2017-02, Vol.16 (1), p.93-103

2017

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Pseudo-acetylation of K326 and K328 of actin disrupts Drosophila melanogaster indirect flight muscle structure and performance

Frontiers in physiology, 2015-04, Vol.6, p.116-116

2015

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Biophysical characterization of G-quadruplex forming FMR1 mRNA and of its interactions with different fragile X mental retardation protein isoforms

RNA (Cambridge), 2014-01, Vol.20 (1), p.103-114

2014

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Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs Drosophila Heart Structure and Impairs Function

Journal of cardiovascular development and disease, 2016-06, Vol.3 (2), p.19

2016

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As time flies by: Investigating cardiac aging in the short-lived Drosophila model

Biochimica et biophysica acta. Molecular basis of disease, 2019-07, Vol.1865 (7), p.1831-1844

2019

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Quantifying Tissue-Specific Overexpression of FOXO in Drosophila via mRNA Fluorescence In Situ Hybridization Using Branched DNA Probe Technology

Methods in molecular biology (Clifton, N.J.), 2019, Vol.1890, p.171

2019

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Analysis of the Fragile X mental retardation protein isoforms 1, 2 and 3 interactions with the G-quadruplex forming semaphorin 3F mRNA

Molecular bioSystems, 2012-01, Vol.8 (2), p.642-649

2012

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Ameliorative Effects of Transcription Factor Dfoxo Over-Expression in a Drosophila Cardiovascular Disease Model

Biophysical journal, 2020-02, Vol.118 (3), p.453a-453a

2020

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Cardiac-Specific Expression of VCP/TER94 RNAi or Disease Alleles Disrupts Drosophila Heart Structure and Impairs Function

Biophysical journal, 2017-02, Vol.112 (3), p.559a-560a

2017

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Pseudo-Acetylation of Actin Residues K326 and K328 Disrupts Drosophila Flight Performance and Muscle Structure

Biophysical journal, 2015-01, Vol.108 (2), p.421a-422a

2015

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Overexpression of Foxo in the Heart Ameliorates Performance Decline through Enhanced UPS Processing in Aging Drosophila

Biophysical journal, 2015-01, Vol.108 (2), p.361a-361a

2015

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Analysis of the Fragile X mental retardation protein isoforms 1, 2 and 3 interactions with the G-quadruplex forming semaphorin 3F mRNAThis work was supported by the NIH grant GM074660-02A1 to M.R.M.Electronic supplementary information (ESI) available. See DOI: 10.1039/c1mb05322a

2012-02

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Fragile X mental retardation protein: Self-regulation and miRNA pathway involvement

Blice-Baum, Anna C

2013

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Cardiac-Specific Overexpression of FOXO Affords Protection Against Age-Associated Decline in Cardiac Performance in the Drosophila Model

Biophysical journal, 2014-01, Vol.106 (2), p.777a-777a

2014

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Modest overexpression of FOXO maintains cardiac proteostasis and ameliorates age‐associated functional decline

Pseudo-acetylation of K326 and K328 of actin disrupts Drosophila melanogaster indirect flight muscle structure and performance

Biophysical characterization of G-quadruplex forming FMR1 mRNA and of its interactions with different fragile X mental retardation protein isoforms

Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs Drosophila Heart Structure and Impairs Function

As time flies by: Investigating cardiac aging in the short-lived Drosophila model

Quantifying Tissue-Specific Overexpression of FOXO in Drosophila via mRNA Fluorescence In Situ Hybridization Using Branched DNA Probe Technology

Analysis of the Fragile X mental retardation protein isoforms 1, 2 and 3 interactions with the G-quadruplex forming semaphorin 3F mRNA

Ameliorative Effects of Transcription Factor Dfoxo Over-Expression in a Drosophila Cardiovascular Disease Model

Cardiac-Specific Expression of VCP/TER94 RNAi or Disease Alleles Disrupts Drosophila Heart Structure and Impairs Function

Pseudo-Acetylation of Actin Residues K326 and K328 Disrupts Drosophila Flight Performance and Muscle Structure

Overexpression of Foxo in the Heart Ameliorates Performance Decline through Enhanced UPS Processing in Aging Drosophila

Analysis of the Fragile X mental retardation protein isoforms 1, 2 and 3 interactions with the G-quadruplex forming semaphorin 3F mRNAThis work was supported by the NIH grant GM074660-02A1 to M.R.M.Electronic supplementary information (ESI) available. See DOI: 10.1039/c1mb05322a

Fragile X mental retardation protein: Self-regulation and miRNA pathway involvement

Cardiac-Specific Overexpression of FOXO Affords Protection Against Age-Associated Decline in Cardiac Performance in the Drosophila Model

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