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Protective role of p53 in acetaminophen hepatotoxicity

Free radical biology & medicine, 2017-05, Vol.106, p.111-117

2017

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New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

Hepatology (Baltimore, Md.), 2018-05, Vol.67 (5), p.2013-2024

2018

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The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

International journal of molecular sciences, 2018-11, Vol.19 (11), p.3657

2018

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Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Hepatology (Baltimore, Md.), 2020-05, Vol.71 (5), p.1530-1532

2020

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c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

Hepatology (Baltimore, Md.), 2016-06, Vol.63 (6), p.1987-2003

2016

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c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

The Journal of biological chemistry, 2011-10, Vol.286 (40), p.35071-35078

2011

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Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

Antioxidants & redox signaling, 2017-02, Vol.26 (5), p.207

2017

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Sab (Sh3bp5) dependence of JNK mediated inhibition of mitochondrial respiration in palmitic acid induced hepatocyte lipotoxicity

Journal of hepatology, 2015-06, Vol.62 (6), p.1367-1374

2015

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Regulation of drug-induced liver injury by signal transduction pathways: critical role of mitochondria

Trends in pharmacological sciences (Regular ed.), 2013-04, Vol.34 (4), p.243-253

2013

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Endoplasmic Reticulum Stress and Liver Injury

Seminars in liver disease, 2007-11, Vol.27 (4), p.367-377

2007

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Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Frontiers in cell and developmental biology, 2024, Vol.12, p.1359152-1359152

2024

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Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

The Journal of biological chemistry, 2011-06, Vol.286 (25), p.22047-22054

2011

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Targeting signal transduction pathways which regulate necrosis in acetaminophen hepatotoxicity

Journal of hepatology, 2015-07, Vol.63 (1), p.5-7

2015

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Tsunami heights and damage along the Myanmar coast from the December 2004 Sumatra-Andaman earthquake

Earth, planets, and space, 2006-01, Vol.58 (2), p.243-252

2006

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Aperiodic recurrence of geologically recorded tsunamis during the past 5500 years in eastern Hokkaido, Japan

Journal of Geophysical Research - Solid Earth, 2009-01, Vol.114 (B1), p.B01319-n/a

2009

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Effect of Transgenic Extrahepatic Expression of Betaine-Homocysteine Methyltransferase on Alcohol or Homocysteine-Induced Fatty Liver

Alcoholism, clinical and experimental research, 2008-06, Vol.32 (6), p.1049-1058

2008

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Historical tsunamis and storms recorded in a coastal lowland, Shizuoka Prefecture, along the Pacific Coast of Japan

Sedimentology, 2008-12, Vol.55 (6), p.1703-1716

2008

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c- Jun -N Terminal Kinase-Mediated Degradation of γ-Glutamylcysteine Ligase Catalytic Subunit Inhibits GSH Recovery After Acetaminophen Treatment: Role in Sustaining JNK Activation and Liver Injury

Antioxidants & redox signaling, 2023-06, Vol.38 (16-18), p.1071

2023

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Contrasting Genome-Wide Distribution of 8-Hydroxyguanine and Acrolein-Modified Adenine during Oxidative Stress-Induced Renal Carcinogenesis

The American journal of pathology, 2006-10, Vol.169 (4), p.1328-1342

2006

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Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

Cell host & microbe, 2024-01, Vol.32 (1), p.48-62.e9

2024

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Silencing Glycogen Synthase Kinase-3β Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.8244-8255

2010

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JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src

Hepatology (Baltimore, Md.), 2016-03, Vol.63 (6), p.1987-2003

2016

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The central role of mitochondrial metabolism in hepatic steatosis

Exploration of digestive diseases, 2024-02, Vol.3 (1), p.42-68

2024

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A continuum theory of grain size evolution and damage

Journal of Geophysical Research - Solid Earth, 2009-01, Vol.114 (B1), p.B01204-n/a

2009

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Physiological Oxidants Induce Apoptosis and Cell Cycle Arrest in a Multidrug-resistant Natural Killer Cell Line, NK-YS

Leukemia & lymphoma, 2003-12, Vol.44 (12), p.2109-2116

2003

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Protective role of p53 in acetaminophen hepatotoxicity

New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

Sab (Sh3bp5) dependence of JNK mediated inhibition of mitochondrial respiration in palmitic acid induced hepatocyte lipotoxicity

Regulation of drug-induced liver injury by signal transduction pathways: critical role of mitochondria

Endoplasmic Reticulum Stress and Liver Injury

Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

Targeting signal transduction pathways which regulate necrosis in acetaminophen hepatotoxicity

Tsunami heights and damage along the Myanmar coast from the December 2004 Sumatra-Andaman earthquake

Aperiodic recurrence of geologically recorded tsunamis during the past 5500 years in eastern Hokkaido, Japan

Effect of Transgenic Extrahepatic Expression of Betaine-Homocysteine Methyltransferase on Alcohol or Homocysteine-Induced Fatty Liver

Historical tsunamis and storms recorded in a coastal lowland, Shizuoka Prefecture, along the Pacific Coast of Japan

c- Jun -N Terminal Kinase-Mediated Degradation of γ-Glutamylcysteine Ligase Catalytic Subunit Inhibits GSH Recovery After Acetaminophen Treatment: Role in Sustaining JNK Activation and Liver Injury

Contrasting Genome-Wide Distribution of 8-Hydroxyguanine and Acrolein-Modified Adenine during Oxidative Stress-Induced Renal Carcinogenesis

Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

Silencing Glycogen Synthase Kinase-3β Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src

The central role of mitochondrial metabolism in hepatic steatosis

A continuum theory of grain size evolution and damage

Physiological Oxidants Induce Apoptosis and Cell Cycle Arrest in a Multidrug-resistant Natural Killer Cell Line, NK-YS

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