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Cardiac Dysfunction Promotes Cancer Progression via Multiple Secreted Factors

Cancer research (Chicago, Ill.), 2022-05, Vol.82 (9), p.1753-1761

2022

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ATF3 and JDP2 deficiency in cancer associated fibroblasts promotes tumor growth via SDF-1 transcription

Oncogene, 2019-05, Vol.38 (20), p.3812-3823

2019

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Heparanase is required for activation and function of macrophages

Proceedings of the National Academy of Sciences - PNAS, 2016-11, Vol.113 (48), p.E7808-E7817

2016

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Early Cardiac Remodeling Promotes Tumor Growth and Metastasis

Circulation (New York, N.Y.), 2020-08, Vol.142 (7), p.670-683

2020

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Tumor Progression Reverses Cardiac Hypertrophy and Fibrosis in a Tetracycline-Regulated ATF3 Transgenic Mouse Model

Cells (Basel, Switzerland), 2023-09, Vol.12 (18), p.2289

2023

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Tumor Growth Ameliorates Cardiac Dysfunction and Suppresses Fibrosis in a Mouse Model for Duchenne Muscular Dystrophy

International journal of molecular sciences, 2023-08, Vol.24 (16), p.12595

2023

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Adult cardiac expression of the activating transcription factor 3, ATF3, promotes ventricular hypertrophy

PloS one, 2013-07, Vol.8 (7), p.e68396-e68396

2013

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Heart Failure Promotes Cancer Progression in an Integrin β1-Dependent Manner

International journal of molecular sciences, 2023-12, Vol.24 (24), p.17367

2023

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Tumor Growth Ameliorates Cardiac Dysfunction

Cells (Basel, Switzerland), 2023-07, Vol.12 (14), p.1853

2023

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JDP2 and ATF3 deficiencies dampen maladaptive cardiac remodeling and preserve cardiac function

PloS one, 2019-02, Vol.14 (2), p.e0213081-e0213081

2019

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Cardiac Remodeling in the Absence of Cardiac Contractile Dysfunction Is Sufficient to Promote Cancer Progression

Cells (Basel, Switzerland), 2022-03, Vol.11 (7), p.1108

2022

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A novel c-Jun N-terminal kinase (JNK)-binding protein WDR62 is recruited to stress granules and mediates a nonclassical JNK activation

Molecular biology of the cell, 2010-01, Vol.21 (1), p.117-130

2010

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IL-31 induces antitumor immunity in breast carcinoma

Journal for immunotherapy of cancer, 2020-08, Vol.8 (2), p.e001010

2020

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The AP-1 repressor protein, JDP2, potentiates hepatocellular carcinoma in mice

Molecular cancer, 2010-03, Vol.9 (1), p.54-54, Article 54

2010

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The Ras Recruitment System (RRS) for the Identification and Characterization of Protein-Protein Interactions

Methods in molecular biology (Clifton, N.J.), 2018, Vol.1794, p.61

Aronheim, Ami

2018

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Dimerization of the adaptor Gads facilitates antigen receptor signaling by promoting the cooperative binding of Gads to the adaptor LAT

Science signaling, 2017-09, Vol.10 (498)

2017

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ATF3 expression in cardiomyocytes and myofibroblasts following transverse aortic constriction displays distinct phenotypes

International journal of cardiology. Heart & vasculature, 2021-02, Vol.32, p.100706-100706, Article 100706

2021

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Increased risk of non-hematological cancer in young patients with aortic stenosis: a retrospective cohort study

Cardio-oncology (London, England), 2021-10, Vol.7 (1), p.1-37, Article 37

2021

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Docking interactions of the JNK scaffold protein WDR62

Biochemical journal, 2011-11, Vol.439 (3), p.381-392

2011

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Monocytes Do Not Transdifferentiate into Proper Osteoblasts

TheScientificWorld, 2012-01, Vol.2012, p.384936-11

2012

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Correction: Adult Cardiac Expression of the Activating Transcription Factor 3, ATF3, Promotes Ventricular Hypertrophy

PloS one, 2013, Vol.8 (9)

2013

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Autophosphorylation-dependent degradation of Pak1, triggered by the Rho-family GTPase, Chp

Biochemical journal, 2007-06, Vol.404 (3), p.487-497

2007

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Isolation of an AP-1 repressor by a novel method for detecting protein-protein interactions

Molecular and Cellular Biology, 1997-06, Vol.17 (6), p.3094-3102

1997

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The C-terminal (BRCT) Domains of BRCA1 Interact in Vivo with CtIP, a Protein Implicated in the CtBP Pathway of Transcriptional Repression

The Journal of biological chemistry, 1998-09, Vol.273 (39), p.25388-25392

1998

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The c-Jun Dimerization Protein 2 Inhibits Cell Transformation and Acts as a Tumor Suppressor Gene

The Journal of biological chemistry, 2004-02, Vol.279 (7), p.5708-5715

2004

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Cardiac Dysfunction Promotes Cancer Progression via Multiple Secreted Factors

ATF3 and JDP2 deficiency in cancer associated fibroblasts promotes tumor growth via SDF-1 transcription

Heparanase is required for activation and function of macrophages

Early Cardiac Remodeling Promotes Tumor Growth and Metastasis

Tumor Progression Reverses Cardiac Hypertrophy and Fibrosis in a Tetracycline-Regulated ATF3 Transgenic Mouse Model

Tumor Growth Ameliorates Cardiac Dysfunction and Suppresses Fibrosis in a Mouse Model for Duchenne Muscular Dystrophy

Adult cardiac expression of the activating transcription factor 3, ATF3, promotes ventricular hypertrophy

Heart Failure Promotes Cancer Progression in an Integrin β1-Dependent Manner

Tumor Growth Ameliorates Cardiac Dysfunction

JDP2 and ATF3 deficiencies dampen maladaptive cardiac remodeling and preserve cardiac function

Cardiac Remodeling in the Absence of Cardiac Contractile Dysfunction Is Sufficient to Promote Cancer Progression

A novel c-Jun N-terminal kinase (JNK)-binding protein WDR62 is recruited to stress granules and mediates a nonclassical JNK activation

IL-31 induces antitumor immunity in breast carcinoma

The AP-1 repressor protein, JDP2, potentiates hepatocellular carcinoma in mice

The Ras Recruitment System (RRS) for the Identification and Characterization of Protein-Protein Interactions

Dimerization of the adaptor Gads facilitates antigen receptor signaling by promoting the cooperative binding of Gads to the adaptor LAT

ATF3 expression in cardiomyocytes and myofibroblasts following transverse aortic constriction displays distinct phenotypes

Increased risk of non-hematological cancer in young patients with aortic stenosis: a retrospective cohort study

Docking interactions of the JNK scaffold protein WDR62

Monocytes Do Not Transdifferentiate into Proper Osteoblasts

Correction: Adult Cardiac Expression of the Activating Transcription Factor 3, ATF3, Promotes Ventricular Hypertrophy

Autophosphorylation-dependent degradation of Pak1, triggered by the Rho-family GTPase, Chp

Isolation of an AP-1 repressor by a novel method for detecting protein-protein interactions

The C-terminal (BRCT) Domains of BRCA1 Interact in Vivo with CtIP, a Protein Implicated in the CtBP Pathway of Transcriptional Repression

The c-Jun Dimerization Protein 2 Inhibits Cell Transformation and Acts as a Tumor Suppressor Gene

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