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Pancreatic Cancer Chemoresistance to Gemcitabine

Cancers, 2017-11, Vol.9 (11), p.157

2017

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Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic cancer cells

BMC cancer, 2019-06, Vol.19 (1), p.596-596, Article 596

2019

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Stellate Cells Aid Growth-Permissive Metabolic Reprogramming and Promote Gemcitabine Chemoresistance in Pancreatic Cancer

Cancers, 2021-02, Vol.13 (4), p.601

2021

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Genetic Ablation of STE20-Type Kinase MST4 Does Not Alleviate Diet-Induced MASLD Susceptibility in Mice

International journal of molecular sciences, 2024-02, Vol.25 (4), p.2446

2024

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Fatty acids abrogate the growth-suppressive effects induced by inhibition of cholesterol flux in pancreatic cancer cells

Cancer cell international, 2023-11, Vol.23 (1), p.1-276, Article 276

2023

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Neoadjuvant chemotherapy is associated with an altered metabolic profile and increased cancer stemness in patients with pancreatic ductal adenocarcinoma

Molecular oncology, 2023-01, Vol.17 (1), p.59-81

2023

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STK25 is a critical determinant in nonalcoholic steatohepatitis

The FASEB journal, 2016-10, Vol.30 (10), p.3628-3643

2016

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Genetic Disruption of Protein Kinase STK25 Ameliorates Metabolic Defects in a Diet-Induced Type 2 Diabetes Model

Diabetes (New York, N.Y.), 2015-08, Vol.64 (8), p.2791-2804

2015

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Pancreatic stellate cell-induced gemcitabine resistance in pancreatic cancer is associated with LDHA- and MCT4-mediated enhanced glycolysis

Cancer cell international, 2023-01, Vol.23 (1), p.9-9, Article 9

2023

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Morphological Heterogeneity in Pancreatic Cancer Reflects Structural and Functional Divergence

Cancers, 2021-02, Vol.13 (4), p.895

2021

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Differential Gemcitabine Sensitivity in Primary Human Pancreatic Cancer Cells and Paired Stellate Cells Is Driven by Heterogenous Drug Uptake and Processing

Cancers, 2020-12, Vol.12 (12), p.3628

2020

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Commonly Used Pancreatic Stellate Cell Cultures Differ Phenotypically and in Their Interactions with Pancreatic Cancer Cells

Cells (Basel, Switzerland), 2019-01, Vol.8 (1), p.23

2019

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Establishment and Characterization of Paired Primary Cultures of Human Pancreatic Cancer Cells and Stellate Cells Derived from the Same Tumor

Cells (Basel, Switzerland), 2020-01, Vol.9 (1), p.227

2020

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Silencing of STE20‐type kinase MST3 in mice with antisense oligonucleotide treatment ameliorates diet‐induced nonalcoholic fatty liver disease

The FASEB journal, 2021-05, Vol.35 (5), p.e21567-n/a

2021

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Pharmacological activation of AMPK suppresses inflammatory response evoked by IL-6 signalling in mouse liver and in human hepatocytes

Molecular and cellular endocrinology, 2013-08, Vol.375 (1-2), p.68-78

2013

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Protein kinase STK25 regulates hepatic lipid partitioning and progression of liver steatosis and NASH

The FASEB journal, 2015-04, Vol.29 (4), p.1564-1576

2015

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Protein kinase STK25 controls lipid partitioning in hepatocytes and correlates with liver fat content in humans

Diabetologia, 2016-02, Vol.59 (2), p.341-353

2016

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Protein kinase MST3 modulates lipid homeostasis in hepatocytes and correlates with nonalcoholic steatohepatitis in humans

The FASEB journal, 2019-09, Vol.33 (9), p.9974-9989

2019

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STK25 Regulates Cardiovascular Disease Progression in a Mouse Model of Hypercholesterolemia

Arteriosclerosis, thrombosis, and vascular biology, 2018-08, Vol.38 (8), p.1723

2018

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STK25 regulates oxidative capacity and metabolic efficiency in adipose tissue

Journal of endocrinology, 2018-09, Vol.238 (3), p.187-202

2018

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Overexpression of protein kinase STK25 in mice exacerbates ectopic lipid accumulation, mitochondrial dysfunction and insulin resistance in skeletal muscle

Diabetologia, 2017-03, Vol.60 (3), p.553-567

2017

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Comparative quantitative proteomic profiling of neoadjuvantly treated versus treatment-naive human pancreatic ductal adenocarcinoma

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2019-06, Vol.19, p.S76-S76

2019

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Increased expression of STK25 leads to impaired glucose utilization and insulin sensitivity in mice challenged with a high‐fat diet

The FASEB journal, 2013-09, Vol.27 (9), p.3660-3671

2013

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Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic adenocarcinoma

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2018-06, Vol.18 (4), p.S155-S155

2018

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Partial hepatic resistance to IL-6-induced inflammation develops in type 2 diabetic mice, while the anti-inflammatory effect of AMPK is maintained

Molecular and cellular endocrinology, 2014-08, Vol.393 (1-2), p.143-151

2014

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Pancreatic Cancer Chemoresistance to Gemcitabine

Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic cancer cells

Stellate Cells Aid Growth-Permissive Metabolic Reprogramming and Promote Gemcitabine Chemoresistance in Pancreatic Cancer

Genetic Ablation of STE20-Type Kinase MST4 Does Not Alleviate Diet-Induced MASLD Susceptibility in Mice

Fatty acids abrogate the growth-suppressive effects induced by inhibition of cholesterol flux in pancreatic cancer cells

Neoadjuvant chemotherapy is associated with an altered metabolic profile and increased cancer stemness in patients with pancreatic ductal adenocarcinoma

STK25 is a critical determinant in nonalcoholic steatohepatitis

Genetic Disruption of Protein Kinase STK25 Ameliorates Metabolic Defects in a Diet-Induced Type 2 Diabetes Model

Pancreatic stellate cell-induced gemcitabine resistance in pancreatic cancer is associated with LDHA- and MCT4-mediated enhanced glycolysis

Morphological Heterogeneity in Pancreatic Cancer Reflects Structural and Functional Divergence

Differential Gemcitabine Sensitivity in Primary Human Pancreatic Cancer Cells and Paired Stellate Cells Is Driven by Heterogenous Drug Uptake and Processing

Commonly Used Pancreatic Stellate Cell Cultures Differ Phenotypically and in Their Interactions with Pancreatic Cancer Cells

Establishment and Characterization of Paired Primary Cultures of Human Pancreatic Cancer Cells and Stellate Cells Derived from the Same Tumor

Silencing of STE20‐type kinase MST3 in mice with antisense oligonucleotide treatment ameliorates diet‐induced nonalcoholic fatty liver disease

Pharmacological activation of AMPK suppresses inflammatory response evoked by IL-6 signalling in mouse liver and in human hepatocytes

Protein kinase STK25 regulates hepatic lipid partitioning and progression of liver steatosis and NASH

Protein kinase STK25 controls lipid partitioning in hepatocytes and correlates with liver fat content in humans

Protein kinase MST3 modulates lipid homeostasis in hepatocytes and correlates with nonalcoholic steatohepatitis in humans

STK25 Regulates Cardiovascular Disease Progression in a Mouse Model of Hypercholesterolemia

STK25 regulates oxidative capacity and metabolic efficiency in adipose tissue

Overexpression of protein kinase STK25 in mice exacerbates ectopic lipid accumulation, mitochondrial dysfunction and insulin resistance in skeletal muscle

Comparative quantitative proteomic profiling of neoadjuvantly treated versus treatment-naive human pancreatic ductal adenocarcinoma

Increased expression of STK25 leads to impaired glucose utilization and insulin sensitivity in mice challenged with a high‐fat diet

Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic adenocarcinoma

Partial hepatic resistance to IL-6-induced inflammation develops in type 2 diabetic mice, while the anti-inflammatory effect of AMPK is maintained

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