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Amyloid polymorphisms constitute distinct clouds of conformational variants in different etiological subtypes of Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2017-12, Vol.114 (49), p.13018-13023

2017

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Review on Alzheimer's disease: Inhibition of amyloid beta and tau tangle formation

International journal of biological macromolecules, 2021-01, Vol.167, p.382-394

2021

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Cerebral amyloid angiopathy and Alzheimer disease - one peptide, two pathways

Nature reviews. Neurology, 2020-01, Vol.16 (1), p.30-42

2020

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Molecular Insight into the Therapeutic Promise of Flavonoids against Alzheimer's Disease

Molecules (Basel, Switzerland), 2020-03, Vol.25 (6), p.1267

2020

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Soluble TREM2 ameliorates pathological phenotypes by modulating microglial functions in an Alzheimer’s disease model

Nature communications, 2019-03, Vol.10 (1), p.1365-1365, Article 1365

2019

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Tau deletion reduces plaque‐associated BACE1 accumulation and decelerates plaque formation in a mouse model of Alzheimer's disease

The EMBO journal, 2019-12, Vol.38 (23), p.e102345-n/a

2019

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The antibody aducanumab reduces Aβ plaques in Alzheimer’s disease

Nature (London), 2016-09, Vol.537 (7618), p.50-56

2016

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Amyloid-β and tau: the trigger and bullet in Alzheimer disease pathogenesis

JAMA neurology, 2014-04, Vol.71 (4), p.505

Bloom, George S

2014

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Scanning ultrasound removes amyloid-β and restores memory in an Alzheimer's disease mouse model

Science translational medicine, 2015-03, Vol.7 (278), p.278ra33

2015

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Targeting of nonlipidated, aggregated apoE with antibodies inhibits amyloid accumulation

The Journal of clinical investigation, 2018-05, Vol.128 (5), p.2144-2155

2018

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Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology

Brain (London, England : 1878), 2016-04, Vol.139 (Pt 4), p.1265-1281

2016

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A new era for understanding amyloid structures and disease

Nature reviews. Molecular cell biology, 2018-12, Vol.19 (12), p.755-773

2018

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Gamma frequency entrainment attenuates amyloid load and modifies microglia

Nature (London), 2016-12, Vol.540 (7632), p.230-235

2016

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The Major Risk Factors for Alzheimer’s Disease: Age, Sex, and Genes Modulate the Microglia Response to Aβ Plaques

Cell reports (Cambridge), 2019-04, Vol.27 (4), p.1293-1306.e6

2019

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Microglia-mediated T cell infiltration drives neurodegeneration in tauopathy

Nature (London), 2023-03, Vol.615 (7953), p.668-677

2023

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Soluble Aβ oligomer production and toxicity

Journal of neurochemistry, 2012-01, Vol.120 (s1), p.125-139

2012

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Clusterin levels are increased in Alzheimer's disease and influence the regional distribution of Aβ

Brain pathology (Zurich, Switzerland), 2017-05, Vol.27 (3), p.305-313

2017

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Neuroinflammation and neuronal loss precede Aβ plaque deposition in the hAPP-J20 mouse model of Alzheimer's disease

PloS one, 2013-04, Vol.8 (4), p.e59586-e59586

2013

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Microglia contributes to plaque growth by cell death due to uptake of amyloid β in the brain of Alzheimer's disease mouse model

Glia, 2016-12, Vol.64 (12), p.2274-2290

2016

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The amyloid hypothesis on trial

Nature (London), 2018-07, Vol.559 (7715), p.S4-S7

Makin, Simon

2018

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Microglia and amyloid plaque formation in Alzheimer's disease – Evidence, possible mechanisms, and future challenges

Journal of neuroimmunology, 2024-05, Vol.390, p.578342, Article 578342

2024

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Intraneuronal beta-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation

The Journal of neuroscience, 2006-10, Vol.26 (40), p.10129-10140

2006

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Amyloid polymorphisms constitute distinct clouds of conformational variants in different etiological subtypes of Alzheimer’s disease

Review on Alzheimer's disease: Inhibition of amyloid beta and tau tangle formation

Cerebral amyloid angiopathy and Alzheimer disease - one peptide, two pathways

Molecular Insight into the Therapeutic Promise of Flavonoids against Alzheimer's Disease

Soluble TREM2 ameliorates pathological phenotypes by modulating microglial functions in an Alzheimer’s disease model

Tau deletion reduces plaque‐associated BACE1 accumulation and decelerates plaque formation in a mouse model of Alzheimer's disease

Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoE

The antibody aducanumab reduces Aβ plaques in Alzheimer’s disease

Amyloid-β and tau: the trigger and bullet in Alzheimer disease pathogenesis

Scanning ultrasound removes amyloid-β and restores memory in an Alzheimer's disease mouse model

Targeting of nonlipidated, aggregated apoE with antibodies inhibits amyloid accumulation

Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology

A new era for understanding amyloid structures and disease

Gamma frequency entrainment attenuates amyloid load and modifies microglia

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

The Major Risk Factors for Alzheimer’s Disease: Age, Sex, and Genes Modulate the Microglia Response to Aβ Plaques

Microglia-mediated T cell infiltration drives neurodegeneration in tauopathy

Soluble Aβ oligomer production and toxicity

Clusterin levels are increased in Alzheimer's disease and influence the regional distribution of Aβ

Neuroinflammation and neuronal loss precede Aβ plaque deposition in the hAPP-J20 mouse model of Alzheimer's disease

Microglia contributes to plaque growth by cell death due to uptake of amyloid β in the brain of Alzheimer's disease mouse model

The amyloid hypothesis on trial

Sex-Specific Association of Apolipoprotein E With Cerebrospinal Fluid Levels of Tau

Microglia and amyloid plaque formation in Alzheimer's disease – Evidence, possible mechanisms, and future challenges

Intraneuronal beta-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation

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