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Sex Differences in Nonalcoholic Fatty Liver Disease: State of the Art and Identification of Research Gaps

Hepatology (Baltimore, Md.), 2019-10, Vol.70 (4), p.1457-1469

2019

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c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

Hepatology (Baltimore, Md.), 2016-06, Vol.63 (6), p.1987-2003

2016

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The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

International journal of molecular sciences, 2018-11, Vol.19 (11), p.3657

2018

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Gut microbiota mediates diurnal variation of acetaminophen induced acute liver injury in mice

Journal of hepatology, 2018-07, Vol.69 (1), p.51-59

2018

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c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

The Journal of biological chemistry, 2011-10, Vol.286 (40), p.35071-35078

2011

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Sab (Sh3bp5) dependence of JNK mediated inhibition of mitochondrial respiration in palmitic acid induced hepatocyte lipotoxicity

Journal of hepatology, 2015-06, Vol.62 (6), p.1367-1374

2015

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Regulation of drug-induced liver injury by signal transduction pathways: critical role of mitochondria

Trends in pharmacological sciences (Regular ed.), 2013-04, Vol.34 (4), p.243-253

2013

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Protective role of p53 in acetaminophen hepatotoxicity

Free radical biology & medicine, 2017-05, Vol.106, p.111-117

2017

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Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Frontiers in cell and developmental biology, 2024, Vol.12, p.1359152-1359152

2024

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Hepatic FcRn regulates albumin homeostasis and susceptibility to liver injury

Proceedings of the National Academy of Sciences - PNAS, 2017-04, Vol.114 (14), p.E2862-E2871

2017

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The role of MAP2 kinases and p38 kinase in acute murine liver injury models

Cell death & disease, 2017-06, Vol.8 (6), p.e2903-e2903

2017

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Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

The Journal of biological chemistry, 2011-06, Vol.286 (25), p.22047-22054

2011

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Targeting signal transduction pathways which regulate necrosis in acetaminophen hepatotoxicity

Journal of hepatology, 2015-07, Vol.63 (1), p.5-7

2015

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Response to: ‘Professional identity in clinician‐scientists: brokers between care and science’

Medical education, 2017-12, Vol.51 (12), p.1294-1294

Win, Sanda

2017

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Mechanism and Therapeutic Targets of c-Jun-N-Terminal Kinases Activation in Nonalcoholic Fatty Liver Disease

Biomedicines, 2022-08, Vol.10 (8), p.2035

2022

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c- Jun -N Terminal Kinase-Mediated Degradation of γ-Glutamylcysteine Ligase Catalytic Subunit Inhibits GSH Recovery After Acetaminophen Treatment: Role in Sustaining JNK Activation and Liver Injury

Antioxidants & redox signaling, 2023-06, Vol.38 (16-18), p.1071

2023

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Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

Hepatology (Baltimore, Md.), 2015-12, Vol.62 (6), p.1847-1857

2015

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New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

Hepatology (Baltimore, Md.), 2018-05, Vol.67 (5), p.2013-2024

2018

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Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Hepatology (Baltimore, Md.), 2020-05, Vol.71 (5), p.1530-1532

2020

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Intestinal Epithelial Chemokine (C-C Motif) Ligand 7 Overexpression Enhances Acetaminophen-Induced Hepatotoxicity in Mice

The American journal of pathology, 2020-01, Vol.190 (1), p.57-67

2020

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Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

Antioxidants & redox signaling, 2017-02, Vol.26 (5), p.207

2017

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Endoplasmic Reticulum Stress-Induced Upregulation of STARD1 Promotes Acetaminophen-Induced Acute Liver Failure

Gastroenterology (New York, N.Y. 1943), 2019-08, Vol.157 (2), p.552-568

2019

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Hepatic Mitochondrial SAB Deletion or Knockdown Alleviates Diet‐Induced Metabolic Syndrome, Steatohepatitis, and Hepatic Fibrosis

Hepatology (Baltimore, Md.), 2021-12, Vol.74 (6), p.3127-3145

2021

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Expression of mitochondrial membrane-linked SAB determines severity of sex-dependent acute liver injury

The Journal of clinical investigation, 2019-12, Vol.129 (12), p.5278-5293

2019

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Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

Cell host & microbe, 2024-01, Vol.32 (1), p.48-62.e9

2024

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Sex Differences in Nonalcoholic Fatty Liver Disease: State of the Art and Identification of Research Gaps

c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

Gut microbiota mediates diurnal variation of acetaminophen induced acute liver injury in mice

c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

Sab (Sh3bp5) dependence of JNK mediated inhibition of mitochondrial respiration in palmitic acid induced hepatocyte lipotoxicity

Regulation of drug-induced liver injury by signal transduction pathways: critical role of mitochondria

Protective role of p53 in acetaminophen hepatotoxicity

Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Hepatic FcRn regulates albumin homeostasis and susceptibility to liver injury

The role of MAP2 kinases and p38 kinase in acute murine liver injury models

Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

Targeting signal transduction pathways which regulate necrosis in acetaminophen hepatotoxicity

Response to: ‘Professional identity in clinician‐scientists: brokers between care and science’

Mechanism and Therapeutic Targets of c-Jun-N-Terminal Kinases Activation in Nonalcoholic Fatty Liver Disease

c- Jun -N Terminal Kinase-Mediated Degradation of γ-Glutamylcysteine Ligase Catalytic Subunit Inhibits GSH Recovery After Acetaminophen Treatment: Role in Sustaining JNK Activation and Liver Injury

Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Intestinal Epithelial Chemokine (C-C Motif) Ligand 7 Overexpression Enhances Acetaminophen-Induced Hepatotoxicity in Mice

Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

Endoplasmic Reticulum Stress-Induced Upregulation of STARD1 Promotes Acetaminophen-Induced Acute Liver Failure

Hepatic Mitochondrial SAB Deletion or Knockdown Alleviates Diet‐Induced Metabolic Syndrome, Steatohepatitis, and Hepatic Fibrosis

Expression of mitochondrial membrane-linked SAB determines severity of sex-dependent acute liver injury

Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

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