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Strange Bedfellows: Nuclear Factor, Erythroid 2-Like 2 (Nrf2) and Hypoxia-Inducible Factor 1 (HIF-1) in Tumor Hypoxia

Antioxidants, 2017-04, Vol.6 (2), p.27

2017

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PIM kinase (and Akt) biology and signaling in tumors

Pharmacology & therapeutics (Oxford), 2015-07, Vol.151, p.41-49

2015

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Defining the mechanisms underlying cyclin dependent kinase control of HIF-1α

Oncotarget, 2022, Vol.13 (1), p.454-455

Warfel, Noel A

2022

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p21WAF1 and tumourigenesis: 20 years after

Current opinion in oncology, 2013-01, Vol.25 (1), p.52-58

2013

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Targeting PIM Kinases to Overcome Therapeutic Resistance in Cancer

Molecular cancer therapeutics, 2021-01, Vol.20 (1), p.3-10

2021

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Targeting CDK4/6 to oppose hypoxia-mediated therapeutic resistance

Cell cycle (Georgetown, Tex.), 2017-07, Vol.16 (13), p.1241-1242

Warfel, Noel A.

2017

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PIM kinases alter mitochondrial dynamics and chemosensitivity in lung cancer

Oncogene, 2020-03, Vol.39 (12), p.2597-2611

2020

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Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatase (PHLPP): A New Player in Cell Signaling

The Journal of biological chemistry, 2012-02, Vol.287 (6), p.3610-3616

2012

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PIM Kinase Inhibitors Kill Hypoxic Tumor Cells by Reducing Nrf2 Signaling and Increasing Reactive Oxygen Species

Molecular cancer therapeutics, 2016-07, Vol.15 (7), p.1637-1647

2016

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Small-Molecule NSC59984 Restores p53 Pathway Signaling and Antitumor Effects against Colorectal Cancer via p73 Activation and Degradation of Mutant p53

Cancer research (Chicago, Ill.), 2015-09, Vol.75 (18), p.3842-3852

2015

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Targeting PIM Kinases to Improve the Efficacy of Immunotherapy

Cells (Basel, Switzerland), 2022-11, Vol.11 (22), p.3700

2022

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Hypoxia-Inducible PIM Kinase Expression Promotes Resistance to Antiangiogenic Agents

Clinical cancer research, 2018-01, Vol.24 (1), p.169-180

2018

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CDK1 stabilizes HIF-1α via direct phosphorylation of Ser668 to promote tumor growth

Cell cycle (Georgetown, Tex.), 2013-12, Vol.12 (23), p.3689-3701

2013

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Direct phosphorylation and stabilization of HIF-1α by PIM1 kinase drives angiogenesis in solid tumors

Oncogene, 2021-08, Vol.40 (32), p.5142-5152

2021

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Digital image analysis using video microscopy of human-derived prostate cancer vs normal prostate organoids to assess migratory behavior on extracellular matrix proteins

Frontiers in oncology, 2023-01, Vol.12, p.1083150-1083150

2023

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PIM1 drives lipid droplet accumulation to promote proliferation and survival in prostate cancer

Oncogene, 2024-02, Vol.43 (6), p.406-419

2024

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PIM1 targeted degradation prevents the emergence of chemoresistance in prostate cancer

Cell chemical biology, 2024-02, Vol.31 (2), p.326-337.e11

2024

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Stabilization of PIM Kinases in Hypoxia Is Mediated by the Deubiquitinase USP28

Cells (Basel, Switzerland), 2022-03, Vol.11 (6), p.1006

2022

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Mechanisms Behind Resistance to PI3K Inhibitor Treatment Induced by the PIM Kinase

Molecular cancer therapeutics, 2018-12, Vol.17 (12), p.2710-2721

2018

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Nelfinavir, A Lead HIV Protease Inhibitor, Is a Broad-Spectrum, Anticancer Agent that Induces Endoplasmic Reticulum Stress, Autophagy, and Apoptosis In vitro and In vivo

Clinical cancer research, 2007-09, Vol.13 (17), p.5183-5194

2007

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PIM1 phosphorylates ABI2 to enhance actin dynamics and promote tumor invasion

The Journal of cell biology, 2023-06, Vol.222 (6), p.1

2023

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Disruption of the Interface between the Pleckstrin Homology (PH) and Kinase Domains of Akt Protein Is Sufficient for Hydrophobic Motif Site Phosphorylation in the Absence of mTORC2

The Journal of biological chemistry, 2011-11, Vol.286 (45), p.39122-39129

2011

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Gene Editing of α6 Integrin Inhibits Muscle Invasive Networks and Increases Cell-Cell Biophysical Properties in Prostate Cancer

Cancer research (Chicago, Ill.), 2019-09, Vol.79 (18), p.4703-4714

2019

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GLUT3/SLC2A3 Is an Endogenous Marker of Hypoxia in Prostate Cancer Cell Lines and Patient-Derived Xenograft Tumors

Diagnostics (Basel), 2022-03, Vol.12 (3), p.676

2022

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Strange Bedfellows: Nuclear Factor, Erythroid 2-Like 2 (Nrf2) and Hypoxia-Inducible Factor 1 (HIF-1) in Tumor Hypoxia

PIM kinase (and Akt) biology and signaling in tumors

Defining the mechanisms underlying cyclin dependent kinase control of HIF-1α

p21WAF1 and tumourigenesis: 20 years after

Targeting PIM Kinases to Overcome Therapeutic Resistance in Cancer

Targeting CDK4/6 to oppose hypoxia-mediated therapeutic resistance

PIM kinases alter mitochondrial dynamics and chemosensitivity in lung cancer

Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatase (PHLPP): A New Player in Cell Signaling

PIM Kinase Inhibitors Kill Hypoxic Tumor Cells by Reducing Nrf2 Signaling and Increasing Reactive Oxygen Species

Small-Molecule NSC59984 Restores p53 Pathway Signaling and Antitumor Effects against Colorectal Cancer via p73 Activation and Degradation of Mutant p53

Targeting PIM Kinases to Improve the Efficacy of Immunotherapy

Hypoxia-Inducible PIM Kinase Expression Promotes Resistance to Antiangiogenic Agents

CDK1 stabilizes HIF-1α via direct phosphorylation of Ser668 to promote tumor growth

Direct phosphorylation and stabilization of HIF-1α by PIM1 kinase drives angiogenesis in solid tumors

Digital image analysis using video microscopy of human-derived prostate cancer vs normal prostate organoids to assess migratory behavior on extracellular matrix proteins

PIM1 drives lipid droplet accumulation to promote proliferation and survival in prostate cancer

PIM1 targeted degradation prevents the emergence of chemoresistance in prostate cancer

Stabilization of PIM Kinases in Hypoxia Is Mediated by the Deubiquitinase USP28

Mechanisms Behind Resistance to PI3K Inhibitor Treatment Induced by the PIM Kinase

Nelfinavir, A Lead HIV Protease Inhibitor, Is a Broad-Spectrum, Anticancer Agent that Induces Endoplasmic Reticulum Stress, Autophagy, and Apoptosis In vitro and In vivo

PIM1 phosphorylates ABI2 to enhance actin dynamics and promote tumor invasion

Deficiency of the Deubiquitinase UCHL1 Attenuates Pulmonary Arterial Hypertension

Disruption of the Interface between the Pleckstrin Homology (PH) and Kinase Domains of Akt Protein Is Sufficient for Hydrophobic Motif Site Phosphorylation in the Absence of mTORC2

Gene Editing of α6 Integrin Inhibits Muscle Invasive Networks and Increases Cell-Cell Biophysical Properties in Prostate Cancer

GLUT3/SLC2A3 Is an Endogenous Marker of Hypoxia in Prostate Cancer Cell Lines and Patient-Derived Xenograft Tumors

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