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Assessment of pain induced by castration in piglets: behavioral and physiological responses over the subsequent 5 days

Applied animal behaviour science, 2003-07, Vol.82 (3), p.201-218

2003

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AAV-mediated delivery of a mutated myostatin propeptide ameliorates calpain 3 but not α-sarcoglycan deficiency

Gene therapy, 2007-05, Vol.14 (9), p.733-740

2007

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P.20.2 Restoration of dystrophin expression after skipping of single and double exon DMD duplications in patient-derived cell lines using antisense oligonucleotide and AAV-U7snRNA approaches

Neuromuscular disorders : NMD, 2013-10, Vol.23 (9), p.839-839

2013

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Early expression of ΔCH1 dystrophin isoform reverses or prevents muscular dystrophy in the Dup2 mouse

Neuromuscular disorders : NMD, 2015-10, Vol.25, p.S292-S293

2015

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Novel Roles of Retinoid X Receptor (RXR) and RXR Ligand in Dynamically Modulating the Activity of the Thyroid Hormone Receptor/RXR Heterodimer

The Journal of biological chemistry, 2004-02, Vol.279 (9), p.7427-7437

2004

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O.13 Using out-of-frame exon skipping to induce IRES-driven expression of an N-truncated dystrophin isoform for 5’ DMD mutations

Neuromuscular disorders : NMD, 2013-10, Vol.23 (9), p.848-848

2013

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T.P.17 Alternate translational initiation and amelioration of phenotype in the DMD gene

Neuromuscular disorders : NMD, 2012-10, Vol.22 (9), p.857-858

2012

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G.P.94

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9), p.823-823

2014

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P1.27 Mutation-directed studies on the function of the dystrophin ZZ domain

Neuromuscular disorders : NMD, 2011-10, Vol.21 (9), p.649-649

2011

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P16 Towards a consensus on biochemical outcome measures for Duchenne muscular dystrophy clinical trials

Neuromuscular disorders : NMD, 2014-03, Vol.24, p.S11-S11

2014

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O.12 Studies in antisense oligonucleotide induced exon skipping of single or double exon duplications

Neuromuscular disorders : NMD, 2011-10, Vol.21 (9), p.703-703

2011

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Rescue of Dystrophic Muscle through U7 snRNA-Mediated Exon Skipping

Science (American Association for the Advancement of Science), 2004-12, Vol.306 (5702), p.1796-1799

2004

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P5545Predictors of 10-year mortality and re-intervention in patients with multivessel coronary disease, reduced systolic left ventricular function, after complete revascularization by PCI or CABG

European heart journal, 2019-10, Vol.40 (Supplement_1)

2019

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G.P.96

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9), p.823-824

2014

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P696Etiology and outcome of patients undergoing pericardiocentesis due to cardiac tamponade

European heart journal, 2018-08, Vol.39 (suppl_1)

2018

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P3553Right ventricular echocardiographic composite parameters in predicting major adverse cardiac events in patients with heart failure with reduced ejection fraction

European heart journal, 2019-10, Vol.40 (Supplement_1)

2019

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G.P.1.15 Clinical description and molecular analysis of a promising model of DMD, the LRMD dog

Neuromuscular disorders : NMD, 2007, Vol.17 (9), p.776-776

2007

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T.O.1 Functional evaluation of dystrophic dogs treated by exon-skipping

Neuromuscular disorders : NMD, 2007, Vol.17 (9), p.898-898

2007

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Immortalized skin fibroblasts expressing conditional MyoD as a renewable and reliable source of converted human muscle cells to assess therapeutic strategies for muscular dystrophies: validation of an exon-skipping approach to restore dystrophin in Duchenne muscular dystrophy cells

Human gene therapy, 2009-07, Vol.20 (7), p.784-790

2009

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G.P.357 - Early expression of ΔCH1 dystrophin isoform reverses or prevents muscular dystrophy in the Dup2 mouse

Neuromuscular disorders : NMD, 2015, Vol.25, p.S292-S293

2015

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Les fibroblastes de patients Gorlin présentent une sénescence réplicative accélérée et une radiosensibilité dépendantes du type de mutation Ptch1

Annales de dermatologie et de vénéréologie, 2015-06, Vol.142 (6-7), p.S283-S284

2015

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G.P.94 : Induction of the N-truncated dystrophin by out-of-frame exon 2 skipping restores muscle function in the Dup2 mouse, providing further support for a therapeutic pathway for 5′ DMD mutations

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9-10), p.823-823

2014

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Oxidative Stress Activates the Plasminogen Activator Inhibitor Type 1 (PAI-1) Promoter through an AP-1 Response Element and Cooperates with Insulin for Additive Effects on PAI-1 Transcription

The Journal of biological chemistry, 2004-06, Vol.279 (24), p.25172-25178

2004

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G.P.96 : Dose escalation studies of rAAV9 U7snRNA targeting exon 2 show highly efficient skipping in the Dup2 mouse

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9-10), p.823-824

2014

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AAV-mediated delivery of a mutated myostatin propeptide ameliorates calpain 3 but not -sarcoglycan deficiency

Gene therapy, 2007-05, Vol.14 (9), p.733-740

2007

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Assessment of pain induced by castration in piglets: behavioral and physiological responses over the subsequent 5 days

AAV-mediated delivery of a mutated myostatin propeptide ameliorates calpain 3 but not α-sarcoglycan deficiency

P.20.2 Restoration of dystrophin expression after skipping of single and double exon DMD duplications in patient-derived cell lines using antisense oligonucleotide and AAV-U7snRNA approaches

Early expression of ΔCH1 dystrophin isoform reverses or prevents muscular dystrophy in the Dup2 mouse

Novel Roles of Retinoid X Receptor (RXR) and RXR Ligand in Dynamically Modulating the Activity of the Thyroid Hormone Receptor/RXR Heterodimer

O.13 Using out-of-frame exon skipping to induce IRES-driven expression of an N-truncated dystrophin isoform for 5’ DMD mutations

T.P.17 Alternate translational initiation and amelioration of phenotype in the DMD gene

G.P.94

P1.27 Mutation-directed studies on the function of the dystrophin ZZ domain

P16 Towards a consensus on biochemical outcome measures for Duchenne muscular dystrophy clinical trials

O.12 Studies in antisense oligonucleotide induced exon skipping of single or double exon duplications

Rescue of Dystrophic Muscle through U7 snRNA-Mediated Exon Skipping

P5545Predictors of 10-year mortality and re-intervention in patients with multivessel coronary disease, reduced systolic left ventricular function, after complete revascularization by PCI or CABG

G.P.96

P696Etiology and outcome of patients undergoing pericardiocentesis due to cardiac tamponade

P3553Right ventricular echocardiographic composite parameters in predicting major adverse cardiac events in patients with heart failure with reduced ejection fraction

G.P.1.15 Clinical description and molecular analysis of a promising model of DMD, the LRMD dog

T.O.1 Functional evaluation of dystrophic dogs treated by exon-skipping

Immortalized skin fibroblasts expressing conditional MyoD as a renewable and reliable source of converted human muscle cells to assess therapeutic strategies for muscular dystrophies: validation of an exon-skipping approach to restore dystrophin in Duchenne muscular dystrophy cells

G.P.357 - Early expression of ΔCH1 dystrophin isoform reverses or prevents muscular dystrophy in the Dup2 mouse

Les fibroblastes de patients Gorlin présentent une sénescence réplicative accélérée et une radiosensibilité dépendantes du type de mutation Ptch1

G.P.94 : Induction of the N-truncated dystrophin by out-of-frame exon 2 skipping restores muscle function in the Dup2 mouse, providing further support for a therapeutic pathway for 5′ DMD mutations

Oxidative Stress Activates the Plasminogen Activator Inhibitor Type 1 (PAI-1) Promoter through an AP-1 Response Element and Cooperates with Insulin for Additive Effects on PAI-1 Transcription

G.P.96 : Dose escalation studies of rAAV9 U7snRNA targeting exon 2 show highly efficient skipping in the Dup2 mouse

AAV-mediated delivery of a mutated myostatin propeptide ameliorates calpain 3 but not -sarcoglycan deficiency

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