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HDLs inhibit endoplasmic reticulum stress and autophagic response induced by oxidized LDLs

Cell death and differentiation, 2011-05, Vol.18 (5), p.817-828

2011

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Acute coronary syndrome remodels the antiplatelet aggregation properties of HDL particle subclasses

Journal of thrombosis and haemostasis, 2018-05, Vol.16 (5), p.933-945

2018

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Ephrin-B1 transduces signals to activate integrin-mediated migration, attachment and angiogenesis

Journal of cell science, 2002-08, Vol.115 (Pt 15), p.3073-3081

2002

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Role of protein kinase C δ in ER stress and apoptosis induced by oxidized LDL in human vascular smooth muscle cells

Cell death & disease, 2013-02, Vol.4 (2), p.e520-e520

2013

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Mitophagy acts as a safeguard mechanism against human vascular smooth muscle cell apoptosis induced by atherogenic lipids

Atherosclerosis, 2016-09, Vol.252, p.e200-e200

2016

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Oxygen-regulated protein-150 prevents calcium homeostasis deregulation and apoptosis induced by oxidized LDL in vascular cells

Cell death and differentiation, 2008-08, Vol.15 (8), p.1255-1265

2008

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2019 ESC/EAS Guidelines for the management of dyslipidaemias: lipid modification to reduce cardiovascular risk

European heart journal, 2020-01, Vol.41 (1), p.111-188

2020

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Identifying the anti-inflammatory response to lipid lowering therapy: a position paper from the working group on atherosclerosis and vascular biology of the European Society of Cardiology

Cardiovascular research, 2019-01, Vol.115 (1), p.10-19

2019

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Anti-Inflammatory and Antiatherogenic Effects of the NLRP3 Inflammasome Inhibitor Arglabin in ApoE2.Ki Mice Fed a High-Fat Diet

Circulation (New York, N.Y.), 2015-03, Vol.131 (12), p.1061-1070

2015

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Identification of a miRNA based-signature associated with acute coronary syndrome: Evidence from the FLORINF study

Atherosclerosis, 2022-08, Vol.355, p.171

2022

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Interplay between hypercholesterolaemia and inflammation in atherosclerosis: Translating experimental targets into clinical practice

European journal of preventive cardiology, 2018-06, Vol.25 (9), p.948-955

2018

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Mitochondrial quality control is altered in ATG7-deficient vascular smooth muscle cells and linked to unstable atherosclerotic plaque phenotype

Atherosclerosis, 2020-12, Vol.315, p.e18-e18

2020

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EphB1-mediated Cell Migration Requires the Phosphorylation of Paxillin at Tyr-31/Tyr-118

The Journal of biological chemistry, 2004-07, Vol.279 (27), p.27965-27970

2004

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Altered mitochondrial quality control in autophagy-deficient vascular smooth muscle cells is linked to high-risk atherosclerotic plaque phenotype

Journal of the neurological sciences, 2019-10, Vol.405, p.156-156

2019

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Reactive oxygen species production by monoamine oxidases in intact cells

Naunyn-Schmiedeberg's archives of pharmacology, 1999-05, Vol.359 (5), p.428-431

1999

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P677Defective autophagy in vascular smooth muscle cells promotes an unstable atherosclerotic plaque phenotype and increased expression of mitophagy markers in Apo E−/− mice

European heart journal, 2017-08, Vol.38 (suppl_1)

2017

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Future directions for therapeutic strategies in post-ischaemic vascularization: a position paper from European Society of Cardiology Working Group on Atherosclerosis and Vascular Biology

Cardiovascular research, 2018-09, Vol.114 (11), p.1411-1421

2018

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Two distinct calcium-dependent mitochondrial pathways are involved in oxidized LDL-induced apoptosis

Arteriosclerosis, thrombosis, and vascular biology, 2005-03, Vol.25 (3), p.639

2005

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Mitochondrial 4-HNE derived from MAO-A promotes mitoCa2+ overload in chronic postischemic cardiac remodeling

Cell death and differentiation, 2020-06, Vol.27 (6), p.1907-1923

2020

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Antiatherogenic effect of bisvanillyl-hydralazone, a new hydralazine derivative with antioxidant, carbonyl scavenger, and antiapoptotic properties

Antioxidants & redox signaling, 2011-06, Vol.14 (11), p.2093

2011

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Differential substrate specificity of monoamine oxidase in the rat heart and renal cortex

Life sciences (1973), 2003-07, Vol.73 (8), p.955-967

2003

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HDLs inhibit endoplasmic reticulum stress and autophagic response induced by oxidized LDLs

Cell death and differentiation, 2011-05, Vol.18 (5), p.817-828

2011

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Essential role of TRPC1 channels in cardiomyoblasts hypertrophy mediated by 5-HT2A serotonin receptors

Biochemical and biophysical research communications, 2010-01, Vol.391 (1), p.979-983

2010

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Desensitization of Platelet-Derived Growth Factor Receptor-β by Oxidized Lipids in Vascular Cells and Atherosclerotic Lesions: Prevention by Aldehyde Scavengers

Circulation research, 2006-03, Vol.98 (6), p.785-792

2006

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Serotonin 5-HT2A receptor-mediated hypertrophy is negatively regulated by caveolin-3 in cardiomyoblasts and neonatal cardiomyocytes

Journal of molecular and cellular cardiology, 2012-02, Vol.52 (2), p.502-510

2012

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HDLs inhibit endoplasmic reticulum stress and autophagic response induced by oxidized LDLs

Acute coronary syndrome remodels the antiplatelet aggregation properties of HDL particle subclasses

Ephrin-B1 transduces signals to activate integrin-mediated migration, attachment and angiogenesis

Role of protein kinase C δ in ER stress and apoptosis induced by oxidized LDL in human vascular smooth muscle cells

Mitophagy acts as a safeguard mechanism against human vascular smooth muscle cell apoptosis induced by atherogenic lipids

Oxygen-regulated protein-150 prevents calcium homeostasis deregulation and apoptosis induced by oxidized LDL in vascular cells

2019 ESC/EAS Guidelines for the management of dyslipidaemias: lipid modification to reduce cardiovascular risk

Identifying the anti-inflammatory response to lipid lowering therapy: a position paper from the working group on atherosclerosis and vascular biology of the European Society of Cardiology

Anti-Inflammatory and Antiatherogenic Effects of the NLRP3 Inflammasome Inhibitor Arglabin in ApoE2.Ki Mice Fed a High-Fat Diet

Identification of a miRNA based-signature associated with acute coronary syndrome: Evidence from the FLORINF study

Interplay between hypercholesterolaemia and inflammation in atherosclerosis: Translating experimental targets into clinical practice

Mitochondrial quality control is altered in ATG7-deficient vascular smooth muscle cells and linked to unstable atherosclerotic plaque phenotype

EphB1-mediated Cell Migration Requires the Phosphorylation of Paxillin at Tyr-31/Tyr-118

Altered mitochondrial quality control in autophagy-deficient vascular smooth muscle cells is linked to high-risk atherosclerotic plaque phenotype

Reactive oxygen species production by monoamine oxidases in intact cells

P677Defective autophagy in vascular smooth muscle cells promotes an unstable atherosclerotic plaque phenotype and increased expression of mitophagy markers in Apo E−/− mice

Future directions for therapeutic strategies in post-ischaemic vascularization: a position paper from European Society of Cardiology Working Group on Atherosclerosis and Vascular Biology

Two distinct calcium-dependent mitochondrial pathways are involved in oxidized LDL-induced apoptosis

Mitochondrial 4-HNE derived from MAO-A promotes mitoCa2+ overload in chronic postischemic cardiac remodeling

Antiatherogenic effect of bisvanillyl-hydralazone, a new hydralazine derivative with antioxidant, carbonyl scavenger, and antiapoptotic properties

Differential substrate specificity of monoamine oxidase in the rat heart and renal cortex

HDLs inhibit endoplasmic reticulum stress and autophagic response induced by oxidized LDLs

Essential role of TRPC1 channels in cardiomyoblasts hypertrophy mediated by 5-HT2A serotonin receptors

Desensitization of Platelet-Derived Growth Factor Receptor-β by Oxidized Lipids in Vascular Cells and Atherosclerotic Lesions: Prevention by Aldehyde Scavengers

Serotonin 5-HT2A receptor-mediated hypertrophy is negatively regulated by caveolin-3 in cardiomyoblasts and neonatal cardiomyocytes

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