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Defective proteostasis in celiac disease as a new therapeutic target

Cell death & disease, 2019-02, Vol.10 (2), p.114-114, Article 114

2019

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The gliadin-CFTR connection: new perspectives for the treatment of celiac disease

Italian journal of pediatrics, 2019-03, Vol.45 (1), p.40-40, Article 40

2019

Volltextzugriff (PDF)

Cystic fibrosis: A disorder with defective autophagy

Autophagy, 2011-01, Vol.7 (1), p.104-106

2011

Volltextzugriff (PDF)

To Be or Not to Be a Pathogen: Candida albicans and Celiac Disease

Frontiers in immunology, 2019-12, Vol.10, p.2844-2844

2019

Volltextzugriff (PDF)

Thymosin alpha 1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Nature medicine, 2017-05, Vol.23 (5), p.590-600

2017

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Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Nature medicine, 2017-05, Vol.23 (5), p.590-600

2017

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A pathogenic role for cystic fibrosis transmembrane conductance regulator in celiac disease

The EMBO journal, 2019-01, Vol.38 (2), p.n/a

2019

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Restoration of CFTR function in patients with cystic fibrosis carrying the F508del-CFTR mutation

Autophagy, 2014-11, Vol.10 (11), p.2053-2074

2014

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Cysteamine re-establishes the clearance of Pseudomonas aeruginosa by macrophages bearing the cystic fibrosis-relevant F508del-CFTR mutation

Cell death & disease, 2017-01, Vol.8 (1), p.e2544-e2544

2017

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Manipulating proteostasis to repair the F508del-CFTR defect in cystic fibrosis

Molecular and cellular pediatrics, 2016-03, Vol.3 (1), p.13-5, Article 13

2016

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Autophagy suppresses the pathogenic immune response to dietary antigens in cystic fibrosis

Cell death & disease, 2019-03, Vol.10 (4), p.258, Article 258

2019

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Reduced caveolin-1 promotes hyperinflammation due to abnormal heme oxygenase-1 localization in lipopolysaccharide-challenged macrophages with dysfunctional cystic fibrosis transmembrane conductance regulator

The Journal of immunology (1950), 2013-05, Vol.190 (10), p.5196-5206

2013

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Nebulized Hyaluronan Ameliorates lung inflammation in cystic fibrosis mice

Pediatric pulmonology, 2013-08, Vol.48 (8), p.761-771

2013

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Towards a rational combination therapy of cystic fibrosis: How cystamine restores the stability of mutant CFTR

Autophagy, 2013-09, Vol.9 (9), p.1431-1434

2013

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Reply to 'F508del-CFTR is not corrected by thymosin α1'

Nature medicine, 2018-07, Vol.24 (7), p.891-893

2018

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Author Correction: Thymosin [alpha]1 represents a potential potent single-molecule-based therapy for cystic fibrosis.(Correction notice)

Nature medicine, 2018-09, Vol.24 (9), p.1481

2018

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Inhaled medications in cystic fibrosis beyond antibiotics

Minerva pediatrica, 2019-08, Vol.71 (4), p.371

2019

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Personalization of therapies in rare diseases: a translational approach for the treatment of cystic fibrosis

Minerva pediatrica, 2019-08, Vol.71 (4), p.362

2019

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Anakinra restores cellular proteostasis by coupling mitochondrial redox balance to autophagy

The Journal of clinical investigation, 2022-01, Vol.132 (2), p.0_1-16

2022

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Tissue Transglutaminase Activation Modulates Inflammation in Cystic Fibrosis via PPARγ Down-Regulation

The Journal of immunology (1950), 2008-06, Vol.180 (11), p.7697-7705

2008

Volltextzugriff (PDF)

Author Correction: Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Nature medicine, 2018-09, Vol.24 (9), p.1481-1481

2018

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Publisher Correction: Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Nature medicine, 2018-09, Vol.24 (9), p.1482-1482

2018

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Repurposing therapies for the personalised treatment of cystic fibrosis

Expert opinion on orphan drugs, 2018-06, Vol.6 (6), p.361-373

2018

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Autophagy
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Cystic Fibrosis Transmembrane Conductance Regulator - Genetics
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Celiac Disease
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Anakinra restores cellular proteostasis by coupling mitochondrial redox balance to autophagy

Defective proteostasis in celiac disease as a new therapeutic target

The gliadin-CFTR connection: new perspectives for the treatment of celiac disease

Cystic fibrosis: A disorder with defective autophagy

To Be or Not to Be a Pathogen: Candida albicans and Celiac Disease

Thymosin alpha 1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

A pathogenic role for cystic fibrosis transmembrane conductance regulator in celiac disease

Restoration of CFTR function in patients with cystic fibrosis carrying the F508del-CFTR mutation

Cysteamine re-establishes the clearance of Pseudomonas aeruginosa by macrophages bearing the cystic fibrosis-relevant F508del-CFTR mutation

Manipulating proteostasis to repair the F508del-CFTR defect in cystic fibrosis

Autophagy suppresses the pathogenic immune response to dietary antigens in cystic fibrosis

Reduced caveolin-1 promotes hyperinflammation due to abnormal heme oxygenase-1 localization in lipopolysaccharide-challenged macrophages with dysfunctional cystic fibrosis transmembrane conductance regulator

Nebulized Hyaluronan Ameliorates lung inflammation in cystic fibrosis mice

Towards a rational combination therapy of cystic fibrosis: How cystamine restores the stability of mutant CFTR

Reply to 'F508del-CFTR is not corrected by thymosin α1'

Author Correction: Thymosin [alpha]1 represents a potential potent single-molecule-based therapy for cystic fibrosis.(Correction notice)

Inhaled medications in cystic fibrosis beyond antibiotics

Personalization of therapies in rare diseases: a translational approach for the treatment of cystic fibrosis

Anakinra restores cellular proteostasis by coupling mitochondrial redox balance to autophagy

Anakinra restores cellular proteostasis by coupling mitochondrial redox balance to autophagy

Tissue Transglutaminase Activation Modulates Inflammation in Cystic Fibrosis via PPARγ Down-Regulation

Author Correction: Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Publisher Correction: Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis

Repurposing therapies for the personalised treatment of cystic fibrosis

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