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Cancer Suppression by the Chromosome Custodians, BRCA1 and BRCA2

Science (American Association for the Advancement of Science), 2014-03, Vol.343 (6178), p.1470-1475

Venkitaraman, Ashok R.

2014

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How do mutations affecting the breast cancer genes BRCA1 and BRCA2 cause cancer susceptibility?

DNA repair, 2019-09, Vol.81, p.102668-102668, Article 102668

Venkitaraman, Ashok R.

2019

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RNA Processing and Genome Stability: Cause and Consequence

Molecular cell, 2016-02, Vol.61 (4), p.496-505

2016

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Cancer Susceptibility and the Functions of BRCA1 and BRCA2

Cell, 2002-01, Vol.108 (2), p.171-182

Venkitaraman, Ashok R.

2002

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Linking the cellular functions of BRCA genes to cancer pathogenesis and treatment

Annual review of pathology, 2009-01, Vol.4 (1), p.461-487

Venkitaraman, Ashok R

2009

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Cancer-causing BRCA2 missense mutations disrupt an intracellular protein assembly mechanism to disable genome maintenance

Nucleic acids research, 2021-06, Vol.49 (10), p.5588-5604

2021

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Enhancing Biochemical Resolution by Hyperdimensional Imaging Microscopy

Biophysical journal, 2019-05, Vol.116 (10), p.1815-1822

2019

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A Class of Environmental and Endogenous Toxins Induces BRCA2 Haploinsufficiency and Genome Instability

Cell, 2017-06, Vol.169 (6), p.1105-1118.e15

2017

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BRCA2 Regulates Transcription Elongation by RNA Polymerase II to Prevent R-Loop Accumulation

Cell reports (Cambridge), 2018-01, Vol.22 (4), p.1031-1039

2018

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Pulsatile MAPK Signaling Modulates p53 Activity to Control Cell Fate Decisions at the G2 Checkpoint for DNA Damage

Cell reports (Cambridge), 2020-02, Vol.30 (7), p.2083-2093.e5

2020

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Impact of Alternative Splicing on the Human Proteome

Cell reports (Cambridge), 2017-08, Vol.20 (5), p.1229-1241

2017

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Rapid recruitment of p53 to DNA damage sites directs DNA repair choice and integrity

Proceedings of the National Academy of Sciences - PNAS, 2022-03, Vol.119 (10), p.e2113233119-e2113233119

2022

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HP1-β mobilization promotes chromatin changes that initiate the DNA damage response

Nature (London), 2008-05, Vol.453 (7195), p.682-686

2008

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High-resolution structure of the presynaptic RAD51 filament on single-stranded DNA by electron cryo-microscopy

Nucleic acids research, 2016-11, Vol.44 (19), p.9017

2016

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Pattern formation in centrosome assembly

Current opinion in cell biology, 2012-02, Vol.24 (1), p.14-23

2012

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A kinase-independent function for AURORA-A in replisome assembly during DNA replication initiation

Nucleic acids research, 2020-08, Vol.48 (14), p.7844-7855

2020

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A-type lamins maintain the positional stability of DNA damage repair foci in mammalian nuclei

PloS one, 2013-05, Vol.8 (5), p.e61893-e61893

2013

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Human Inositol Polyphosphate Multikinase Regulates Transcript-Selective Nuclear mRNA Export to Preserve Genome Integrity

Molecular cell, 2013-09, Vol.51 (6), p.737-750

2013

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Ketogenic diet promotes tumor ferroptosis but induces relative corticosterone deficiency that accelerates cachexia

Cell metabolism, 2023-07, Vol.35 (7), p.1147-1162.e7

2023

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BRCA2 deficiency reveals that oxidative stress impairs RNaseH1 function to cripple mitochondrial DNA maintenance

Cell reports (Cambridge), 2021-08, Vol.36 (5), p.109478-109478, Article 109478

2021

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Oncogenic KRAS triggers MAPK-dependent errors in mitosis and MYC-dependent sensitivity to anti-mitotic agents

Scientific reports, 2016-07, Vol.6 (1), p.29741, Article 29741

2016

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Double Strain-Promoted Macrocyclization for the Rapid Selection of Cell-Active Stapled Peptides

Angewandte Chemie International Edition, 2015-12, Vol.54 (51), p.15410-15413

International ed. in English, 2015

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Maximizing the biochemical resolving power of fluorescence microscopy

PloS one, 2013-10, Vol.8 (10), p.e77392-e77392

2013

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A transcriptional response to replication stress selectively expands a subset of Brca2-mutant mammary epithelial cells

Nature communications, 2023-08, Vol.14 (1), p.5206-5206, Article 5206

2023

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Two modules in the BRC repeats of BRCA2 mediate structural and functional interactions with the RAD51 recombinase

Nucleic acids research, 2010-01, Vol.38 (1), p.82-96

2010

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Cancer Suppression by the Chromosome Custodians, BRCA1 and BRCA2

How do mutations affecting the breast cancer genes BRCA1 and BRCA2 cause cancer susceptibility?

RNA Processing and Genome Stability: Cause and Consequence

Cancer Susceptibility and the Functions of BRCA1 and BRCA2

Linking the cellular functions of BRCA genes to cancer pathogenesis and treatment

Cancer-causing BRCA2 missense mutations disrupt an intracellular protein assembly mechanism to disable genome maintenance

Enhancing Biochemical Resolution by Hyperdimensional Imaging Microscopy

A Class of Environmental and Endogenous Toxins Induces BRCA2 Haploinsufficiency and Genome Instability

BRCA2 Regulates Transcription Elongation by RNA Polymerase II to Prevent R-Loop Accumulation

Pulsatile MAPK Signaling Modulates p53 Activity to Control Cell Fate Decisions at the G2 Checkpoint for DNA Damage

Impact of Alternative Splicing on the Human Proteome

Rapid recruitment of p53 to DNA damage sites directs DNA repair choice and integrity

HP1-β mobilization promotes chromatin changes that initiate the DNA damage response

High-resolution structure of the presynaptic RAD51 filament on single-stranded DNA by electron cryo-microscopy

Pattern formation in centrosome assembly

A kinase-independent function for AURORA-A in replisome assembly during DNA replication initiation

A-type lamins maintain the positional stability of DNA damage repair foci in mammalian nuclei

Human Inositol Polyphosphate Multikinase Regulates Transcript-Selective Nuclear mRNA Export to Preserve Genome Integrity

Ketogenic diet promotes tumor ferroptosis but induces relative corticosterone deficiency that accelerates cachexia

BRCA2 deficiency reveals that oxidative stress impairs RNaseH1 function to cripple mitochondrial DNA maintenance

Oncogenic KRAS triggers MAPK-dependent errors in mitosis and MYC-dependent sensitivity to anti-mitotic agents

Double Strain-Promoted Macrocyclization for the Rapid Selection of Cell-Active Stapled Peptides

Maximizing the biochemical resolving power of fluorescence microscopy

A transcriptional response to replication stress selectively expands a subset of Brca2-mutant mammary epithelial cells

Two modules in the BRC repeats of BRCA2 mediate structural and functional interactions with the RAD51 recombinase

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