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Nox‐4 deletion reduces oxidative stress and injury by PKC‐α‐associated mechanisms in diabetic nephropathy

Physiological reports, 2014-11, Vol.2 (11), p.e12192-n/a

2014

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Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy

Journal of the American Society of Nephrology, 2014-06, Vol.25 (6), p.1237-1254

2014

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Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy

Diabetologia, 2016-02, Vol.59 (2), p.379-389

2016

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Mapping time-course mitochondrial adaptations in the kidney in experimental diabetes

Clinical science (1979), 2016-05, Vol.130 (9), p.711-720

2016

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RAGE-Induced Cytosolic ROS Promote Mitochondrial Superoxide Generation in Diabetes

Journal of the American Society of Nephrology, 2009-04, Vol.20 (4), p.742-752

2009

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Processed foods drive intestinal barrier permeability and microvascular diseases

Science advances, 2021-03, Vol.7 (14)

2021

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Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product–Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C-α–Dependent Pathway

Diabetes (New York, N.Y.), 2008-02, Vol.57 (2), p.460-469

2008

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Lack of the antioxidant enzyme glutathione peroxidase-1 accelerates atherosclerosis in diabetic apolipoprotein E-deficient mice

Circulation (New York, N.Y.), 2007-04, Vol.115 (16), p.2178-2187

2007

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Targeted reduction of advanced glycation improves renal function in obesity

Kidney international, 2011-07, Vol.80 (2), p.190-198

2011

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Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathy

American journal of physiology. Renal physiology, 2010-03, Vol.298 (3), p.F763-F770

2010

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Modulation of soluble receptor for advanced glycation end products by angiotensin-converting enzyme-1 inhibition in diabetic nephropathy

Journal of the American Society of Nephrology, 2005-08, Vol.16 (8), p.2363-2372

2005

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Targeting Methylglyoxal in Diabetic Kidney Disease Using the Mitochondria-Targeted Compound MitoGamide

Nutrients, 2021-04, Vol.13 (5), p.1457

2021

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Attenuation of Extracellular Matrix Accumulation in Diabetic Nephropathy by the Advanced Glycation End Product Cross-Link Breaker ALT-711 via a Protein Kinase C-α−Dependent Pathway

Diabetes (New York, N.Y.), 2004-11, Vol.53 (11), p.2921-2930

2004

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Targeting the AGE-RAGE axis improves renal function in the context of a healthy diet low in advanced glycation end-product content

Nephrology (Carlton, Vic.), 2013-01, Vol.18 (1), p.47-56

2013

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AT2R agonist, compound 21, is reno-protective against type 1 diabetic nephropathy

Hypertension (Dallas, Tex. 1979), 2015-05, Vol.65 (5), p.1073-1081

2015

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Advanced glycation: how are we progressing to combat this web of sugar anomalies in diabetic nephropathy

Current pharmaceutical design, 2004-01, Vol.10 (27), p.3361-3372

2004

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NADPH Oxidase Nox5 Accelerates Renal Injury in Diabetic Nephropathy

Diabetes (New York, N.Y.), 2017-10, Vol.66 (10), p.2691-2703

2017

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Complement C5a Induces Renal Injury in Diabetic Kidney Disease by Disrupting Mitochondrial Metabolic Agility

Diabetes (New York, N.Y.), 2020-01, Vol.69 (1), p.83-98

2020

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Dedifferentiation of Immortalized Human Podocytes in Response to Transforming Growth Factor-β A Model for Diabetic Podocytopathy

Diabetes (New York, N.Y.), 2011-06, Vol.60 (6), p.1779-1788

2011

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Tandem inhibition of PKC in Diαβetic nephropathy: it takes two to tango?

Diabetes (New York, N.Y.), 2013-04, Vol.62 (4), p.1010-1011

2013

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Delineating a role for the mitochondrial permeability transition pore in diabetic kidney disease by targeting cyclophilin D

Clinical science (1979), 2020-01, Vol.134 (2), p.239-259

2020

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Tandem Inhibition of PKC in Diαβetic Nephropathy

Diabetes (New York, N.Y.), 2013-04, Vol.62 (4), p.1010-1011

2013

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Tandem inhibition of PKC in di[alpha][beta]etic nephropathy: it takes two to tango?

Diabetes (New York, N.Y.), 2013-04, Vol.62 (4), p.1010

2013

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Nox-4 and progressive kidney disease

Current opinion in nephrology and hypertension, 2015-01, Vol.24 (1), p.74-80

2015

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Advanced Glycation End Products Are Direct Modulators of β-Cell Function

Diabetes (New York, N.Y.), 2011-10, Vol.60 (10), p.2523-2532

2011

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Nox‐4 deletion reduces oxidative stress and injury by PKC‐α‐associated mechanisms in diabetic nephropathy

Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy

Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy

Mapping time-course mitochondrial adaptations in the kidney in experimental diabetes

RAGE-Induced Cytosolic ROS Promote Mitochondrial Superoxide Generation in Diabetes

Processed foods drive intestinal barrier permeability and microvascular diseases

Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product–Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C-α–Dependent Pathway

Lack of the antioxidant enzyme glutathione peroxidase-1 accelerates atherosclerosis in diabetic apolipoprotein E-deficient mice

Targeted reduction of advanced glycation improves renal function in obesity

Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathy

Modulation of soluble receptor for advanced glycation end products by angiotensin-converting enzyme-1 inhibition in diabetic nephropathy

Targeting Methylglyoxal in Diabetic Kidney Disease Using the Mitochondria-Targeted Compound MitoGamide

Attenuation of Extracellular Matrix Accumulation in Diabetic Nephropathy by the Advanced Glycation End Product Cross-Link Breaker ALT-711 via a Protein Kinase C-α−Dependent Pathway

Targeting the AGE-RAGE axis improves renal function in the context of a healthy diet low in advanced glycation end-product content

AT2R agonist, compound 21, is reno-protective against type 1 diabetic nephropathy

Advanced glycation: how are we progressing to combat this web of sugar anomalies in diabetic nephropathy

NADPH Oxidase Nox5 Accelerates Renal Injury in Diabetic Nephropathy

Complement C5a Induces Renal Injury in Diabetic Kidney Disease by Disrupting Mitochondrial Metabolic Agility

Dedifferentiation of Immortalized Human Podocytes in Response to Transforming Growth Factor-β A Model for Diabetic Podocytopathy

Tandem inhibition of PKC in Diαβetic nephropathy: it takes two to tango?

Delineating a role for the mitochondrial permeability transition pore in diabetic kidney disease by targeting cyclophilin D

Tandem Inhibition of PKC in Diαβetic Nephropathy

Tandem inhibition of PKC in di[alpha][beta]etic nephropathy: it takes two to tango?

Nox-4 and progressive kidney disease

Advanced Glycation End Products Are Direct Modulators of β-Cell Function

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