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Neighbourhood Watch: Two-pore-2 channels talking to IP3 receptors

Cell calcium (Edinburgh), 2024-05, Vol.119, p.102868-102868, Article 102868

2024

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Mitochondrial junctions with cellular organelles: Ca2+ signalling perspective

Pflügers Archiv, 2018-08, Vol.470 (8), p.1181-1192

Tepikin, Alexei V.

2018

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Polarity of action in salivary gland acinar cells: Local and preferential Ca2+ signalling

Cell calcium (Edinburgh), 2021-11, Vol.99, p.102471-102471, Article 102471

2021

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Inhibitors of ORAI1 Prevent Cytosolic Calcium-Associated Injury of Human Pancreatic Acinar Cells and Acute Pancreatitis in 3 Mouse Models

Gastroenterology (New York, N.Y. 1943), 2015-08, Vol.149 (2), p.481-492.e7

2015

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Role of phosphoinositides in STIM1 dynamics and store-operated calcium entry

Biochemical journal, 2009-12, Vol.425 (1), p.159-168

2009

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Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shift

The Journal of biological chemistry, 2018-05, Vol.293 (21), p.8032-8047

2018

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Endoplasmic reticulum–plasma membrane junctions: structure, function and dynamics

The Journal of physiology, 2016-06, Vol.594 (11), p.2837-2847

2016

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Caffeine protects against experimental acute pancreatitis by inhibition of inositol 1,4,5-trisphosphate receptor-mediated Ca2+ release

Gut, 2017-02, Vol.66 (2), p.301

2017

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Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitis

The Journal of physiology, 2018-07, Vol.596 (13), p.2547-2564

2018

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Deletion of the WD40 domain of ATG16L1 exacerbates acute pancreatitis, abolishes LAP-like non-canonical autophagy and slows trypsin degradation

Autophagy, 2024-08, p.1-13

2024

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Mitochondrial Targeting of Antioxidants Alters Pancreatic Acinar Cell Bioenergetics and Determines Cell Fate

International journal of molecular sciences, 2019-04, Vol.20 (7), p.1700

2019

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Reactive Oxygen Species Induced by Bile Acid Induce Apoptosis and Protect Against Necrosis in Pancreatic Acinar Cells

Gastroenterology (New York, N.Y. 1943), 2011-06, Vol.140 (7), p.2116-2125

2011

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Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

The Journal of biological chemistry, 2009-07, Vol.284 (31), p.20796-20803

2009

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The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cells

Biochemical journal, 2015-02, Vol.465 (3), p.405-412

2015

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Evidence for an interaction between Golli and STIM1 in store-operated calcium entry

Biochemical journal, 2010-09, Vol.430 (3), p.453-460

2010

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A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

Current biology, 2012-07, Vol.22 (13), p.1241-1246

2012

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Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis

Gut, 2014-08, Vol.63 (8), p.1313-1324

2014

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LAP-like non-canonical autophagy and evolution of endocytic vacuoles in pancreatic acinar cells

Autophagy, 2020-07, Vol.16 (7), p.1314-1331

2020

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Menadione-induced Reactive Oxygen Species Generation via Redox Cycling Promotes Apoptosis of Murine Pancreatic Acinar Cells

The Journal of biological chemistry, 2006-12, Vol.281 (52), p.40485-40492

2006

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Dynamic Changes in Cytosolic and Mitochondrial ATP Levels in Pancreatic Acinar Cells

Gastroenterology (New York, N.Y. 1943), 2010-05, Vol.138 (5), p.1976-1987.e5

2010

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Epithelial-mesenchymal transition, IP3 receptors and ER-PM junctions: translocation of Ca2+ signalling complexes and regulation of migration

Biochemical journal, 2016-03, Vol.473 (6), p.757-767

2016

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Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis

Gastroenterology (New York, N.Y. 1943), 2006-03, Vol.130 (3), p.781-793

2006

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cAMP Induces Stromal Interaction Molecule 1 (STIM1) Puncta but neither Orai1 Protein Clustering nor Store-operated Ca2+ Entry (SOCE) in Islet Cells

The Journal of biological chemistry, 2012-03, Vol.287 (13), p.9862-9872

2012

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Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does Not Reduce Severity of Experimental Acute Pancreatitis

Cells (Basel, Switzerland), 2020-06, Vol.9 (6), p.1407

2020

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Neighbourhood Watch: Two-pore-2 channels talking to IP3 receptors

Mitochondrial junctions with cellular organelles: Ca2+ signalling perspective

Polarity of action in salivary gland acinar cells: Local and preferential Ca2+ signalling

Inhibitors of ORAI1 Prevent Cytosolic Calcium-Associated Injury of Human Pancreatic Acinar Cells and Acute Pancreatitis in 3 Mouse Models

Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP

Role of phosphoinositides in STIM1 dynamics and store-operated calcium entry

Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shift

Endoplasmic reticulum–plasma membrane junctions: structure, function and dynamics

Caffeine protects against experimental acute pancreatitis by inhibition of inositol 1,4,5-trisphosphate receptor-mediated Ca2+ release

Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitis

Deletion of the WD40 domain of ATG16L1 exacerbates acute pancreatitis, abolishes LAP-like non-canonical autophagy and slows trypsin degradation

Mitochondrial Targeting of Antioxidants Alters Pancreatic Acinar Cell Bioenergetics and Determines Cell Fate

Reactive Oxygen Species Induced by Bile Acid Induce Apoptosis and Protect Against Necrosis in Pancreatic Acinar Cells

Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cells

Evidence for an interaction between Golli and STIM1 in store-operated calcium entry

A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis

LAP-like non-canonical autophagy and evolution of endocytic vacuoles in pancreatic acinar cells

Menadione-induced Reactive Oxygen Species Generation via Redox Cycling Promotes Apoptosis of Murine Pancreatic Acinar Cells

Dynamic Changes in Cytosolic and Mitochondrial ATP Levels in Pancreatic Acinar Cells

Epithelial-mesenchymal transition, IP3 receptors and ER-PM junctions: translocation of Ca2+ signalling complexes and regulation of migration

Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis

cAMP Induces Stromal Interaction Molecule 1 (STIM1) Puncta but neither Orai1 Protein Clustering nor Store-operated Ca2+ Entry (SOCE) in Islet Cells

Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does Not Reduce Severity of Experimental Acute Pancreatitis

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