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Macrophages and Diabetic Nephropathy

Seminars in nephrology, 2010-05, Vol.30 (3), p.290-301

Tesch, Greg H., PhD

2010

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Diabetic nephropathy - is this an immune disorder?

Clinical science (1979), 2017-08, Vol.131 (16), p.2183

Tesch, Greg H

2017

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Mineralocorticoid Receptor Signaling as a Therapeutic Target for Renal and Cardiac Fibrosis

Frontiers in pharmacology, 2017-05, Vol.8, p.313-313

2017

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Review: Serum and urine biomarkers of kidney disease: A pathophysiological perspective

Nephrology (Carlton, Vic.), 2010-09, Vol.15 (6), p.609-616

TESCH, GREG H

2010

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ASK1/p38 signaling in renal tubular epithelial cells promotes renal fibrosis in the mouse obstructed kidney

American journal of physiology. Renal physiology, 2014-12, Vol.307 (11), p.F1263-F1273

2014

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Rodent models of streptozotocin-induced diabetic nephropathy (Methods in Renal Research)

Nephrology (Carlton, Vic.), 2007-06, Vol.12 (3), p.261-266

2007

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ASK1: a new therapeutic target for kidney disease

American journal of physiology. Renal physiology, 2016-08, Vol.311 (2), p.F373-F381

2016

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ASK1 Inhibitor Halts Progression of Diabetic Nephropathy in Nos3-Deficient Mice

Diabetes (New York, N.Y.), 2015-11, Vol.64 (11), p.3903-3913

2015

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Cardiac Tissue Injury and Remodeling Is Dependent Upon MR Regulation of Activation Pathways in Cardiac Tissue Macrophages

Endocrinology (Philadelphia), 2016-08, Vol.157 (8), p.3213-3223

2016

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miR-378 reduces mesangial hypertrophy and kidney tubular fibrosis via MAPK signalling

Clinical science (1979), 2017-03, Vol.131 (5), p.411

2017

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Macrophage Mineralocorticoid Receptor Signaling Plays a Key Role in Aldosterone-Independent Cardiac Fibrosis

Endocrinology (Philadelphia), 2012-07, Vol.153 (7), p.3416-3425

2012

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Myeloid mineralocorticoid receptor activation contributes to progressive kidney disease

Journal of the American Society of Nephrology, 2014-10, Vol.25 (10), p.2231-2240

2014

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Role of macrophages in the fibrotic phase of rat crescentic glomerulonephritis

American journal of physiology. Renal physiology, 2013-04, Vol.304 (8), p.F1043-F1053

2013

Volltextzugriff (PDF)

TGF-β1-activated kinase-1 regulates inflammation and fibrosis in the obstructed kidney

American journal of physiology. Renal physiology, 2011-06, Vol.300 (6), p.F1410-F1421

2011

Volltextzugriff (PDF)

Quantification of renal pathology by image analysis (Methods in Renal Research)

Nephrology (Carlton, Vic.), 2007-12, Vol.12 (6), p.553-558

2007

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Targeting apoptosis signal‐regulating kinase 1 in acute and chronic kidney disease

Anatomical record (Hoboken, N.J. : 2007), 2020-10, Vol.303 (10), p.2553-2560

2020

Volltextzugriff (PDF)

Corrigendum: c-Jun amino terminal kinase signaling promotes aristolochic acid-induced acute kidney injury

Frontiers in physiology, 2023-01, Vol.13, p.1123475-1123475

2023

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Design and pharmacology of a highly specific dual FMS and KIT kinase inhibitor

Proceedings of the National Academy of Sciences - PNAS, 2013-04, Vol.110 (14), p.5689-5694

2013

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c-Jun Amino Terminal Kinase Signaling Promotes Aristolochic Acid-Induced Acute Kidney Injury

Frontiers in physiology, 2021-02, Vol.12, p.599114-599114

2021

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Suppression of Rapidly Progressive Mouse Glomerulonephritis with the Non-Steroidal Mineralocorticoid Receptor Antagonist BR-4628

PloS one, 2015-12, Vol.10 (12), p.e0145666-e0145666

2015

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A pathogenic role for c-jun amino-terminal kinase signaling in renal fibrosis and tubular cell apoptosis

Journal of the American Society of Nephrology, 2007-02, Vol.18 (2), p.472-484

2007

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Establishing equivalent diabetes in male and female Nos3‐deficient mice results in a comparable onset of diabetic kidney injury

Physiological reports, 2019-09, Vol.7 (18), p.e14197-n/a

2019

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Blockade of the c-Jun amino terminal kinase prevents crescent formation and halts established anti-GBM glomerulonephritis in the rat

Laboratory investigation, 2009-04, Vol.89 (4), p.470-484

2009

Volltextzugriff (PDF)

MKK3-p38 signaling promotes apoptosis and the early inflammatory response in the obstructed mouse kidney

American journal of physiology. Renal physiology, 2007-11, Vol.293 (5), p.F1556-F1563

2007

Volltextzugriff (PDF)

ASK1 inhibitor treatment suppresses p38/JNK signalling with reduced kidney inflammation and fibrosis in rat crescentic glomerulonephritis

Journal of cellular and molecular medicine, 2018-09, Vol.22 (9), p.4522-4533

2018

Volltextzugriff (PDF)

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Macrophages and Diabetic Nephropathy

Diabetic nephropathy - is this an immune disorder?

Mineralocorticoid Receptor Signaling as a Therapeutic Target for Renal and Cardiac Fibrosis

Review: Serum and urine biomarkers of kidney disease: A pathophysiological perspective

ASK1/p38 signaling in renal tubular epithelial cells promotes renal fibrosis in the mouse obstructed kidney

Rodent models of streptozotocin-induced diabetic nephropathy (Methods in Renal Research)

ASK1: a new therapeutic target for kidney disease

ASK1 Inhibitor Halts Progression of Diabetic Nephropathy in Nos3-Deficient Mice

Cardiac Tissue Injury and Remodeling Is Dependent Upon MR Regulation of Activation Pathways in Cardiac Tissue Macrophages

miR-378 reduces mesangial hypertrophy and kidney tubular fibrosis via MAPK signalling

Macrophage Mineralocorticoid Receptor Signaling Plays a Key Role in Aldosterone-Independent Cardiac Fibrosis

Myeloid mineralocorticoid receptor activation contributes to progressive kidney disease

Role of macrophages in the fibrotic phase of rat crescentic glomerulonephritis

TGF-β1-activated kinase-1 regulates inflammation and fibrosis in the obstructed kidney

Quantification of renal pathology by image analysis (Methods in Renal Research)

Targeting apoptosis signal‐regulating kinase 1 in acute and chronic kidney disease

Corrigendum: c-Jun amino terminal kinase signaling promotes aristolochic acid-induced acute kidney injury

Design and pharmacology of a highly specific dual FMS and KIT kinase inhibitor

c-Jun Amino Terminal Kinase Signaling Promotes Aristolochic Acid-Induced Acute Kidney Injury

Suppression of Rapidly Progressive Mouse Glomerulonephritis with the Non-Steroidal Mineralocorticoid Receptor Antagonist BR-4628

A pathogenic role for c-jun amino-terminal kinase signaling in renal fibrosis and tubular cell apoptosis

Establishing equivalent diabetes in male and female Nos3‐deficient mice results in a comparable onset of diabetic kidney injury

Blockade of the c-Jun amino terminal kinase prevents crescent formation and halts established anti-GBM glomerulonephritis in the rat

MKK3-p38 signaling promotes apoptosis and the early inflammatory response in the obstructed mouse kidney

ASK1 inhibitor treatment suppresses p38/JNK signalling with reduced kidney inflammation and fibrosis in rat crescentic glomerulonephritis

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