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APOBEC Enzymes: Mutagenic Fuel for Cancer Evolution and Heterogeneity

Cancer discovery, 2015-07, Vol.5 (7), p.704-712

2015

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Tissue-Specific Distribution of iNKT Cells Impacts Their Cytokine Response

Immunity (Cambridge, Mass.), 2015-09, Vol.43 (3), p.566-578

2015

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Mash Screen: high-throughput sequence containment estimation for genome discovery

Genome Biology, 2019-11, Vol.20 (1), p.232-232, Article 232

2019

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Microhomology‐mediated repair machinery and its relationship with HPV‐mediated oncogenesis

Journal of medical virology, 2024-05, Vol.96 (5), p.e29674-n/a

2024

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Functional Upregulation of the DNA Cytosine Deaminase APOBEC3B by Polyomaviruses

Journal of virology, 2016-07, Vol.90 (14), p.6379-6386

2016

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Structural basis for targeted DNA cytosine deamination and mutagenesis by APOBEC3A and APOBEC3B

Nature structural & molecular biology, 2017-02, Vol.24 (2), p.131-139

2017

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APOBEC3A catalyzes mutation and drives carcinogenesis in vivo

The Journal of experimental medicine, 2020-12, Vol.217 (12)

2020

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Long-read sequencing reveals complex patterns of wraparound transcription in polyomaviruses

PLoS pathogens, 2022-04, Vol.18 (4), p.e1010401

2022

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The DNA cytosine deaminase APOBEC3H haplotype I likely contributes to breast and lung cancer mutagenesis

Nature communications, 2016-09, Vol.7 (1), p.12918-13, Article 12918

2016

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Brief Report: The Virome of Bladder Tumors Arising in People Living With HIV

Journal of acquired immune deficiency syndromes (1999), 2023-12, Vol.94 (4), p.337-340

2023

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Lineage-Specific Effector Signatures of Invariant NKT Cells Are Shared amongst gamma delta T, Innate Lymphoid, and Th Cells

The Journal of immunology (1950), 2016-08, Vol.197 (4), p.1460-1470

2016

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Detection of extensive host-virus DNA hybrids in human polyomavirus 7-associated pruritic rash

British journal of dermatology (1951), 2023-04, Vol.188 (5), p.675-677

2023

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Mutation Processes in 293-Based Clones Overexpressing the DNA Cytosine Deaminase APOBEC3B

PloS one, 2016-05, Vol.11 (5), p.e0155391

2016

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Clinical and molecular characterization of virus-positive and virus-negative Merkel cell carcinoma

Genome medicine, 2020-03, Vol.12 (1), p.30-22, Article 30

2020

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The case for BK polyomavirus as a cause of bladder cancer

Current opinion in virology, 2019-12, Vol.39, p.8-15

2019

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Human papillomavirus E6 triggers upregulation of the antiviral and cancer genomic DNA deaminase APOBEC3B

mBio, 2014-12, Vol.5 (6)

2014

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HPV42, a "Low-Risk" Type, and Digital Papillary Adenocarcinoma

Cancer discovery, 2023-01, Vol.13 (1), p.17-18

2023

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Mutational impact of APOBEC3A and APOBEC3B in a human cell line and comparisons to breast cancer

PLoS genetics, 2023-11, Vol.19 (11), p.e1011043-e1011043

2023

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Sebaceous Carcinoma Epidemiology and Genetics: Emerging Concepts and Clinical Implications for Screening, Prevention, and Treatment

Clinical cancer research, 2021-01, Vol.27 (2), p.389-393

2021

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The PKC/NF-κB signaling pathway induces APOBEC3B expression in multiple human cancers

Cancer research (Chicago, Ill.), 2015-11, Vol.75 (21), p.4538-4547

2015

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Characterization of BK Polyomaviruses from Kidney Transplant Recipients Suggests a Role for APOBEC3 in Driving In-Host Virus Evolution

Cell host & microbe, 2018-05, Vol.23 (5), p.628-635.e7

2018

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Merkel Cell Polyomavirus Exhibits Dominant Control of the Tumor Genome and Transcriptome in Virus-Associated Merkel Cell Carcinoma

mBio, 2017-01, Vol.8 (1)

2017

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The deaminase APOBEC3B triggers the death of cells lacking uracil DNA glycosylase

Proceedings of the National Academy of Sciences - PNAS, 2019-10, Vol.116 (44), p.22158-22163

2019

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Genetic and mechanistic basis for APOBEC3H alternative splicing, retrovirus restriction, and counteraction by HIV-1 protease

Nature communications, 2018-10, Vol.9 (1), p.4137-11, Article 4137

2018

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APOBEC Enzymes: Mutagenic Fuel for Cancer Evolution and Heterogeneity

Tissue-Specific Distribution of iNKT Cells Impacts Their Cytokine Response

Mash Screen: high-throughput sequence containment estimation for genome discovery

Microhomology‐mediated repair machinery and its relationship with HPV‐mediated oncogenesis

Functional Upregulation of the DNA Cytosine Deaminase APOBEC3B by Polyomaviruses

Structural basis for targeted DNA cytosine deamination and mutagenesis by APOBEC3A and APOBEC3B

APOBEC3A catalyzes mutation and drives carcinogenesis in vivo

Long-read sequencing reveals complex patterns of wraparound transcription in polyomaviruses

The DNA cytosine deaminase APOBEC3H haplotype I likely contributes to breast and lung cancer mutagenesis

Discovery of several thousand highly diverse circular DNA viruses

Brief Report: The Virome of Bladder Tumors Arising in People Living With HIV

Lineage-Specific Effector Signatures of Invariant NKT Cells Are Shared amongst gamma delta T, Innate Lymphoid, and Th Cells

Detection of extensive host-virus DNA hybrids in human polyomavirus 7-associated pruritic rash

Mutation Processes in 293-Based Clones Overexpressing the DNA Cytosine Deaminase APOBEC3B

Clinical and molecular characterization of virus-positive and virus-negative Merkel cell carcinoma

The case for BK polyomavirus as a cause of bladder cancer

Human papillomavirus E6 triggers upregulation of the antiviral and cancer genomic DNA deaminase APOBEC3B

HPV42, a "Low-Risk" Type, and Digital Papillary Adenocarcinoma

Mutational impact of APOBEC3A and APOBEC3B in a human cell line and comparisons to breast cancer

Sebaceous Carcinoma Epidemiology and Genetics: Emerging Concepts and Clinical Implications for Screening, Prevention, and Treatment

The PKC/NF-κB signaling pathway induces APOBEC3B expression in multiple human cancers

Characterization of BK Polyomaviruses from Kidney Transplant Recipients Suggests a Role for APOBEC3 in Driving In-Host Virus Evolution

Merkel Cell Polyomavirus Exhibits Dominant Control of the Tumor Genome and Transcriptome in Virus-Associated Merkel Cell Carcinoma

The deaminase APOBEC3B triggers the death of cells lacking uracil DNA glycosylase

Genetic and mechanistic basis for APOBEC3H alternative splicing, retrovirus restriction, and counteraction by HIV-1 protease

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