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Renal disease in tuberous sclerosis complex: pathogenesis and therapy

Nature reviews. Nephrology, 2018-11, Vol.14 (11), p.704-716

2018

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The TRPP2-dependent channel of renal primary cilia also requires TRPM3

PloS one, 2019-03, Vol.14 (3), p.e0214053-e0214053

2019

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Improvement in Renal Cystic Disease of Tuberous Sclerosis Complex After Treatment with Mammalian Target of Rapamycin Inhibitor

The Journal of pediatrics, 2017-08, Vol.187, p.318-322.e2

2017

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Characterization of Renal Toxicity in Mice Administered the Marine Biotoxin Domoic Acid

Journal of the American Society of Nephrology, 2014-06, Vol.25 (6), p.1187-1197

2014

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Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress

American journal of physiology. Renal physiology, 2012-09, Vol.303 (6), p.F831-F844

2012

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Cell cycle control and DNA damage response of conditionally immortalized urothelial cells

PloS one, 2011-01, Vol.6 (1), p.e16595

2011

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Glomerulocystic kidney disease

Pediatric nephrology (Berlin, West), 2010-10, Vol.25 (10), p.2049-2059

2010

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Primary cilia regulate the osmotic stress response of renal epithelial cells through TRPM3

American journal of physiology. Renal physiology, 2017-04, Vol.312 (4), p.F791-F805

2017

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Clinical and Molecular Insights into Tuberous Sclerosis Complex Renal Disease

Pediatric nephrology (Berlin, West), 2011-06, Vol.26 (6), p.839-852

2011

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Evidence for pericyte origin of TSC-associated renal angiomyolipomas and implications for angiotensin receptor inhibition therapy

American journal of physiology. Renal physiology, 2014-09, Vol.307 (5), p.F560-F570

2014

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Tuberin Regulates Prostaglandin Receptor-Mediated Viability, via Rheb, in mTORC1-Hyperactive Cells

Molecular cancer research, 2017-10, Vol.15 (10), p.1318-1330

2017

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Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage

American journal of physiology. Renal physiology, 2015-07, Vol.309 (1), p.F79-F87

2015

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Tuberous sclerosis complex exhibits a new renal cystogenic mechanism

Physiological reports, 2019-01, Vol.7 (2), p.e13983-n/a

2019

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Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors

American journal of physiology. Renal physiology, 2012-04, Vol.302 (7), p.F801-F808

2012

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The growing importance of mTORC1-S6K1 signaling in kidney

American journal of physiology. Renal physiology, 2009-09, Vol.297 (3), p.F583-F584

2009

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Stressed podocytes fail to fold: a potential new role of ER in FSGS

American journal of physiology. Renal physiology, 2011-09, Vol.301 (3), p.F494-F495

2011

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Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells

American journal of physiology. Renal physiology, 2006-06, Vol.290 (6), p.F1320-F1328

2006

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Renal involvement in tuberous sclerosis complex and von Hippel-Lindau disease: shared disease mechanisms?

Nature clinical practice. Nephrology, 2009-03, Vol.5 (3), p.143-156

2009

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Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress

American journal of physiology. Renal physiology, 2012, Vol.72 (3)

2012

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Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors

American journal of physiology. Renal physiology, 2012, Vol.71 (4)

2012

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Renal Cystic Disease: The Role of the Primary Cilium/Centrosome Complex in Pathogenesis

Advances in chronic kidney disease, 2006-04, Vol.13 (2), p.131-137

2006

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Altered cell volume regulation in a mouse cell‐model of autosomal recessive polycystic kidney disease

The FASEB journal, 2006-03, Vol.20 (4), p.A339-A339

2006

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Increased Cell Proliferation is Attenuated by Ca2+ Chelation in a Mouse Model of ARPKD

The FASEB journal, 2006-03, Vol.20 (4), p.A339-A339

2006

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Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells

American journal of physiology. Renal physiology, 2006-06, Vol.59 (6), p.F1320

2006

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Interrelationship between cell proliferation, actin disruption, and cytosolic calcium in cilia -deficient collecting duct cells

Siroky, Brian J

2006

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Renal disease in tuberous sclerosis complex: pathogenesis and therapy

The TRPP2-dependent channel of renal primary cilia also requires TRPM3

Improvement in Renal Cystic Disease of Tuberous Sclerosis Complex After Treatment with Mammalian Target of Rapamycin Inhibitor

Characterization of Renal Toxicity in Mice Administered the Marine Biotoxin Domoic Acid

Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress

Cell cycle control and DNA damage response of conditionally immortalized urothelial cells

Glomerulocystic kidney disease

Primary cilia regulate the osmotic stress response of renal epithelial cells through TRPM3

Clinical and Molecular Insights into Tuberous Sclerosis Complex Renal Disease

Evidence for pericyte origin of TSC-associated renal angiomyolipomas and implications for angiotensin receptor inhibition therapy

Tuberin Regulates Prostaglandin Receptor-Mediated Viability, via Rheb, in mTORC1-Hyperactive Cells

Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage

Tuberous sclerosis complex exhibits a new renal cystogenic mechanism

Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors

The growing importance of mTORC1-S6K1 signaling in kidney

Stressed podocytes fail to fold: a potential new role of ER in FSGS

Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells

Renal involvement in tuberous sclerosis complex and von Hippel-Lindau disease: shared disease mechanisms?

Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress

Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors

Renal Cystic Disease: The Role of the Primary Cilium/Centrosome Complex in Pathogenesis

Altered cell volume regulation in a mouse cell‐model of autosomal recessive polycystic kidney disease

Increased Cell Proliferation is Attenuated by Ca2+ Chelation in a Mouse Model of ARPKD

Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells

Interrelationship between cell proliferation, actin disruption, and cytosolic calcium in cilia -deficient collecting duct cells

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