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Molecular Mimicry by Herpes Simplex Virus-Type 1: Autoimmune Disease After Viral Infection

Science (American Association for the Advancement of Science), 1998-02, Vol.279 (5355), p.1344-1347

1998

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A new paradigm for transcription factor TFIIB functionality

Scientific reports, 2014-01, Vol.4 (1), p.3664-3664, Article 3664

2014

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Role of Nuclear Factor Y in Stress-Induced Activation of the Herpes Simplex Virus Type 1 ICP0 Promoter

Journal of Virology, 2010-01, Vol.84 (1), p.188-200

2010

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Point Mutations in Herpes Simplex Virus Type 1 oriL, but Not in oriS, Reduce Pathogenesis during Acute Infection of Mice and Impair Reactivation from Latency

Journal of Virology, 2006-01, Vol.80 (1), p.440-450

2006

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Herpes Simplex Virus 1 ICP0 Phosphorylation Site Mutants Are Attenuated for Viral Replication and Impaired for Explant-Induced Reactivation

Journal of Virology, 2011-12, Vol.85 (23), p.12631-12637

2011

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Transient Expression of Herpes Simplex Virus Type 1 ICP22 Represses Viral Promoter Activity and Complements the Replication of an ICP22 Null Virus

Journal of Virology, 2009-09, Vol.83 (17), p.8733-8743

2009

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Pharmacological Cyclin-Dependent Kinase Inhibitors Inhibit Replication of Wild-Type and Drug-Resistant Strains of Herpes Simplex Virus and Human Immunodeficiency Virus Type 1 by Targeting Cellular, Not Viral, Proteins

Journal of Virology, 2002-08, Vol.76 (15), p.7874-7882

2002

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Origin of Expression of the Herpes Simplex Virus Type 1 Protein US1.5

Journal of Virology, 2009-09, Vol.83 (18), p.9183-9194

2009

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Glutamine Deprivation Causes Enhanced Plating Efficiency of a Herpes Simplex Virus Type 1 ICP0-Null Mutant

Journal of Virology, 2008-11, Vol.82 (22), p.11472-11475

2008

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Herpes Simplex Virus Type 1 C-Terminal Variants of the Origin Binding Protein (OBP), OBPC-1 and OBPC-2, Cooperatively Regulate Viral DNA Levels In Vitro, and OBPC-2 Affects Mortality in Mice

Journal of Virology, 2007-10, Vol.81 (19), p.10699-10711

2007

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Phosphorylation Site Mutations Affect Herpes Simplex Virus Type 1 ICP0 Function

Journal of Virology, 2005-01, Vol.79 (2), p.1232-1243

2005

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ICP22 Is Required for Wild-Type Composition and Infectivity of Herpes Simplex Virus Type 1 Virions

Journal of Virology, 2006-10, Vol.80 (19), p.9381-9390

2006

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Cellular Stress Rather than Stage of the Cell Cycle Enhances the Replication and Plating Efficiencies of Herpes Simplex Virus Type 1 ICP0− Viruses

Journal of Virology, 2006-05, Vol.80 (9), p.4528-4537

2006

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Cathepsin B Mediates Cleavage of Herpes Simplex Virus Type 1 Origin Binding Protein (OBP) To Yield OBPC-1, and Cleavage Is Dependent upon Viral DNA Replication

Journal of Virology, 2007-09, Vol.81 (17), p.9175-9182

2007

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Explant-Induced Reactivation of Herpes Simplex Virus Occurs in Neurons Expressing Nuclear cdk2 and cdk4

Journal of Virology, 2002-08, Vol.76 (15), p.7724-7735

2002

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New anti-HSV therapeutics target the helicase-primase complex

Nature medicine, 2002-04, Vol.8 (4), p.327-328

2002

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Recruitment of activated IRF-3 and CBP/p300 to herpes simplex virus ICP0 nuclear foci: Potential role in blocking IFN-β induction

Virology (New York, N.Y.), 2007-04, Vol.360 (2), p.305-321

2007

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The Cyclin-Dependent Kinase Inhibitor Roscovitine Inhibits the Transactivating Activity and Alters the Posttranslational Modification of Herpes Simplex Virus Type 1 ICP0

Journal of Virology, 2002-02, Vol.76 (3), p.1077-1088

2002

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Neither LAT nor Open Reading Frame P Mutations Increase Expression of Spliced or Intron-Containing ICP0 Transcripts in Mouse Ganglia Latently Infected with Herpes Simplex Virus

Journal of Virology, 2002-05, Vol.76 (10), p.4764-4772

2002

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The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0

Journal of Virology, 2003-12, Vol.77 (23), p.12603-12616

2003

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Site-Directed Mutagenesis of Large DNA Palindromes: Construction and In Vitro Characterization of Herpes Simplex Virus Type 1 Mutants Containing Point Mutations That Eliminate the oriL or oriS Initiation Function

Journal of Virology, 2005-10, Vol.79 (20), p.12783-12797

2005

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The Products of the Herpes Simplex Virus Type 1 Immediate-Early US1/US1.5 Genes Downregulate Levels of S-Phase-Specific Cyclins and Facilitate Virus Replication in S-Phase Vero Cells

Journal of Virology, 2006-04, Vol.80 (8), p.4005-4016

2006

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Origin of expression of the herpes simplex virus type 1 protein U(S)1.5

Journal of virology, 2009-09, Vol.83 (18), p.9183-9194

2009

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ICP0 Is Required for Efficient Reactivation of Herpes Simplex Virus Type 1 from Neuronal Latency

Journal of Virology, 2001-04, Vol.75 (7), p.3240-3249

2001

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Construction and characterization of a herpes simplex virus type I recombinant expressing green fluorescent protein: Acute phase replication and reactivation in mice

Virology (New York, N.Y.), 2007-05, Vol.361 (2), p.372-383

2007

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Molecular Mimicry by Herpes Simplex Virus-Type 1: Autoimmune Disease After Viral Infection

A new paradigm for transcription factor TFIIB functionality

Role of Nuclear Factor Y in Stress-Induced Activation of the Herpes Simplex Virus Type 1 ICP0 Promoter

Point Mutations in Herpes Simplex Virus Type 1 oriL, but Not in oriS, Reduce Pathogenesis during Acute Infection of Mice and Impair Reactivation from Latency

Herpes Simplex Virus 1 ICP0 Phosphorylation Site Mutants Are Attenuated for Viral Replication and Impaired for Explant-Induced Reactivation

Transient Expression of Herpes Simplex Virus Type 1 ICP22 Represses Viral Promoter Activity and Complements the Replication of an ICP22 Null Virus

Pharmacological Cyclin-Dependent Kinase Inhibitors Inhibit Replication of Wild-Type and Drug-Resistant Strains of Herpes Simplex Virus and Human Immunodeficiency Virus Type 1 by Targeting Cellular, Not Viral, Proteins

Origin of Expression of the Herpes Simplex Virus Type 1 Protein US1.5

Glutamine Deprivation Causes Enhanced Plating Efficiency of a Herpes Simplex Virus Type 1 ICP0-Null Mutant

Herpes Simplex Virus Type 1 C-Terminal Variants of the Origin Binding Protein (OBP), OBPC-1 and OBPC-2, Cooperatively Regulate Viral DNA Levels In Vitro, and OBPC-2 Affects Mortality in Mice

Phosphorylation Site Mutations Affect Herpes Simplex Virus Type 1 ICP0 Function

ICP22 Is Required for Wild-Type Composition and Infectivity of Herpes Simplex Virus Type 1 Virions

Cellular Stress Rather than Stage of the Cell Cycle Enhances the Replication and Plating Efficiencies of Herpes Simplex Virus Type 1 ICP0− Viruses

Cathepsin B Mediates Cleavage of Herpes Simplex Virus Type 1 Origin Binding Protein (OBP) To Yield OBPC-1, and Cleavage Is Dependent upon Viral DNA Replication

Explant-Induced Reactivation of Herpes Simplex Virus Occurs in Neurons Expressing Nuclear cdk2 and cdk4

New anti-HSV therapeutics target the helicase-primase complex

Recruitment of activated IRF-3 and CBP/p300 to herpes simplex virus ICP0 nuclear foci: Potential role in blocking IFN-β induction

The Cyclin-Dependent Kinase Inhibitor Roscovitine Inhibits the Transactivating Activity and Alters the Posttranslational Modification of Herpes Simplex Virus Type 1 ICP0

Neither LAT nor Open Reading Frame P Mutations Increase Expression of Spliced or Intron-Containing ICP0 Transcripts in Mouse Ganglia Latently Infected with Herpes Simplex Virus

The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0

Site-Directed Mutagenesis of Large DNA Palindromes: Construction and In Vitro Characterization of Herpes Simplex Virus Type 1 Mutants Containing Point Mutations That Eliminate the oriL or oriS Initiation Function

The Products of the Herpes Simplex Virus Type 1 Immediate-Early US1/US1.5 Genes Downregulate Levels of S-Phase-Specific Cyclins and Facilitate Virus Replication in S-Phase Vero Cells

Origin of expression of the herpes simplex virus type 1 protein U(S)1.5

ICP0 Is Required for Efficient Reactivation of Herpes Simplex Virus Type 1 from Neuronal Latency

Construction and characterization of a herpes simplex virus type I recombinant expressing green fluorescent protein: Acute phase replication and reactivation in mice

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