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Force Responses and Sarcomere Dynamics of Cardiac Myofibrils Induced by Rapid Changes in [Pi]

Biophysical journal, 2017-01, Vol.112 (2), p.356-367

Stehle, Robert

2017

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Kinetic coupling of phosphate release, force generation and rate-limiting steps in the cross-bridge cycle

Journal of muscle research and cell motility, 2017-08, Vol.38 (3-4), p.275-289

2017

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Kinetics of cardiac sarcomeric processes and rate-limiting steps in contraction and relaxation

Journal of molecular and cellular cardiology, 2010-05, Vol.48 (5), p.843-850

2010

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Infantile restrictive cardiomyopathy: cTnI-R170G/W impair the interplay of sarcomeric proteins and the integrity of thin filaments

PloS one, 2020-03, Vol.15 (3), p.e0229227-e0229227

2020

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Hypertrophic and Dilated Cardiomyopathy-Associated Troponin T Mutations R130C and ΔK210 Oppositely Affect Length-Dependent Calcium Sensitivity of Force Generation

Frontiers in physiology, 2020-06, Vol.11, p.516-516

2020

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De Novo Missense Mutations in TNNC1 and TNNI3 Causing Severe Infantile Cardiomyopathy Affect Myofilament Structure and Function and Are Modulated by Troponin Targeting Agents

International journal of molecular sciences, 2021-09, Vol.22 (17), p.9625

2021

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Early signs of architectural and biomechanical failure in isolated myofibers and immortalized myoblasts from desmin-mutant knock-in mice

Scientific reports, 2017-05, Vol.7 (1), p.1391-14, Article 1391

2017

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Sarcomeric determinants of striated muscle relaxation kinetics

Pflügers Archiv, 2005-03, Vol.449 (6), p.505-517

2005

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Half-Sarcomere Dynamics in Myofibrils during Activation and Relaxation Studied by Tracking Fluorescent Markers

Biophysical journal, 2006-01, Vol.90 (2), p.514-530

2006

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Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy

Cellular and molecular life sciences : CMLS, 2013-02, Vol.70 (3), p.527-543

2013

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Correction: Winter et al. Z-Disk-Associated Plectin (Isoform 1d): Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis. Cells 2023, 12 , 1259

Cells (Basel, Switzerland), 2023-11, Vol.12 (23), p.2677

2023

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Z-Disk-Associated Plectin (Isoform 1d): Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis

Cells (Basel, Switzerland), 2023-04, Vol.12 (9), p.1259

2023

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Cycling Cross-Bridges Contribute to Thin Filament Activation in Human Slow-Twitch Fibers

Frontiers in physiology, 2020-03, Vol.11, p.144-144

2020

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Developmental changes in contractility and sarcomeric proteins from the early embryonic to the adult stage in the mouse heart

The Journal of physiology, 2003-04, Vol.548 (2), p.493-505

2003

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Cross‐bridges in slow skeletal muscle: not only slow but also soft

The Journal of physiology, 2018-04, Vol.596 (7), p.1121-1122

2018

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Postnatal Development of Right Ventricular Myofibrillar Biomechanics in Relation to the Sarcomeric Protein Phenotype in Pediatric Patients with Conotruncal Heart Defects

Journal of the American Heart Association, 2016-06, Vol.5 (6), p.n/a

2016

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Phosphate has dual roles in cross-bridge kinetics in rabbit psoas single myofibrils

The Journal of general physiology, 2021-03, Vol.153 (3), p.1

2021

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Expression and purification of human cardiac troponin subunits and their functional incorporation into isolated cardiac mouse myofibrils

Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 2003-03, Vol.786 (1), p.287-296

2003

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Insights into the kinetics of Ca2+-regulated contraction and relaxation from myofibril studies

Pflügers Archiv, 2009-06, Vol.458 (2), p.337-357

2009

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Deletion of the titin N2B region accelerates myofibrillar force development but does not alter relaxation kinetics

Journal of cell science, 2014-09, Vol.127 (Pt 17), p.3666-3674

2014

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Does β‐Adrenergic/PKA Signaling Modulate Ca 2+ ‐Sensitivity and Force Kinetics in Myofibrils from Mice Carrying the FHC Related Mutation cTnI‐ΔK184?

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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ATP binding and cross‐bridge detachment steps during full Ca2+ activation: comparison of myofibril and muscle fibre mechanics by sinusoidal analysis

The Journal of physiology, 2012-07, Vol.590 (14), p.3361-3373

2012

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Correction: Winter et al. Z-Disk-Associated Plectin : Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis. ICells/I 2023, I12/I, 1259

Cells (Basel, Switzerland), 2023-11, Vol.12 (23)

2023

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Myosin Effects on Thin Filament Activation in Slow-Twitch Human Soleus Muscle Fibers

Biophysical journal, 2018-02, Vol.114 (3), p.644a-644a

2018

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The cardiac troponin C mutation Leu29Gln found in a patient with hypertrophic cardiomyopathy does not alter contractile parameters in skinned murine myocardium

Basic research in cardiology, 2009-11, Vol.104 (6), p.751-760

2009

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Force Responses and Sarcomere Dynamics of Cardiac Myofibrils Induced by Rapid Changes in [Pi]

Kinetic coupling of phosphate release, force generation and rate-limiting steps in the cross-bridge cycle

Kinetics of cardiac sarcomeric processes and rate-limiting steps in contraction and relaxation

Infantile restrictive cardiomyopathy: cTnI-R170G/W impair the interplay of sarcomeric proteins and the integrity of thin filaments

Hypertrophic and Dilated Cardiomyopathy-Associated Troponin T Mutations R130C and ΔK210 Oppositely Affect Length-Dependent Calcium Sensitivity of Force Generation

De Novo Missense Mutations in TNNC1 and TNNI3 Causing Severe Infantile Cardiomyopathy Affect Myofilament Structure and Function and Are Modulated by Troponin Targeting Agents

Early signs of architectural and biomechanical failure in isolated myofibers and immortalized myoblasts from desmin-mutant knock-in mice

Sarcomeric determinants of striated muscle relaxation kinetics

Half-Sarcomere Dynamics in Myofibrils during Activation and Relaxation Studied by Tracking Fluorescent Markers

Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy

Correction: Winter et al. Z-Disk-Associated Plectin (Isoform 1d): Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis. Cells 2023, 12 , 1259

Z-Disk-Associated Plectin (Isoform 1d): Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis

Cycling Cross-Bridges Contribute to Thin Filament Activation in Human Slow-Twitch Fibers

Developmental changes in contractility and sarcomeric proteins from the early embryonic to the adult stage in the mouse heart

Cross‐bridges in slow skeletal muscle: not only slow but also soft

Postnatal Development of Right Ventricular Myofibrillar Biomechanics in Relation to the Sarcomeric Protein Phenotype in Pediatric Patients with Conotruncal Heart Defects

Phosphate has dual roles in cross-bridge kinetics in rabbit psoas single myofibrils

Expression and purification of human cardiac troponin subunits and their functional incorporation into isolated cardiac mouse myofibrils

Insights into the kinetics of Ca2+-regulated contraction and relaxation from myofibril studies

Deletion of the titin N2B region accelerates myofibrillar force development but does not alter relaxation kinetics

Does β‐Adrenergic/PKA Signaling Modulate Ca 2+ ‐Sensitivity and Force Kinetics in Myofibrils from Mice Carrying the FHC Related Mutation cTnI‐ΔK184?

ATP binding and cross‐bridge detachment steps during full Ca2+ activation: comparison of myofibril and muscle fibre mechanics by sinusoidal analysis

Correction: Winter et al. Z-Disk-Associated Plectin : Spatial Arrangement, Interaction Partners, and Role in Filamin C Homeostasis. ICells/I 2023, I12/I, 1259

Myosin Effects on Thin Filament Activation in Slow-Twitch Human Soleus Muscle Fibers

The cardiac troponin C mutation Leu29Gln found in a patient with hypertrophic cardiomyopathy does not alter contractile parameters in skinned murine myocardium

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