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OPA1 Isoforms in the Hierarchical Organization of Mitochondrial Functions

Cell reports (Cambridge), 2017-06, Vol.19 (12), p.2557-2571

2017

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Mitochondria: Biogenesis and mitophagy balance in segregation and clonal expansion of mitochondrial DNA mutations

The international journal of biochemistry & cell biology, 2015-06, Vol.63, p.21-24

2015

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Learning from oncocytic tumors: Why choose inefficient mitochondria?

Biochimica et biophysica acta, 2011-06, Vol.1807 (6), p.633-642

2011

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Molecular mechanisms of superoxide production by complex III: A bacterial versus human mitochondrial comparative case study

Biochimica et biophysica acta, 2013-11, Vol.1827 (11-12), p.1332-1339

2013

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The effects of idebenone on mitochondrial bioenergetics

Biochimica et biophysica acta, 2012-02, Vol.1817 (2), p.363-369

2012

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OPA1 links human mitochondrial genome maintenance to mtDNA replication and distribution

Genome research, 2011-01, Vol.21 (1), p.12-20

2011

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Organization of the Respiratory Supercomplexes in Cells with Defective Complex III: Structural Features and Metabolic Consequences

Life (Basel, Switzerland), 2021-04, Vol.11 (4), p.351

2021

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Disruptive mitochondrial DNA mutations in complex I subunits are markers of oncocytic phenotype in thyroid tumors

Proceedings of the National Academy of Sciences - PNAS, 2007-05, Vol.104 (21), p.9001-9006

2007

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Respiratory Complex I Dysfunction Due to Mitochondrial DNA Mutations Shifts the Voltage Threshold for Opening of the Permeability Transition Pore toward Resting Levels

The Journal of biological chemistry, 2009-01, Vol.284 (4), p.2045-2052

2009

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The background of mitochondrial DNA haplogroup J increases the sensitivity of Leber's hereditary optic neuropathy cells to 2,5-hexanedione toxicity

PloS one, 2009-11, Vol.4 (11), p.e7922-e7922

2009

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The cytochrome b p.278Y>C mutation causative of a multisystem disorder enhances superoxide production and alters supramolecular interactions of respiratory chain complexes

Human molecular genetics, 2013-06, Vol.22 (11), p.2141-2151

2013

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Leber's Hereditary Optic Neuropathy (LHON) Pathogenic Mutations Induce Mitochondrial-dependent Apoptotic Death in Transmitochondrial Cells Incubated with Galactose Medium

The Journal of biological chemistry, 2003-02, Vol.278 (6), p.4145-4150

2003

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Bioenergetics shapes cellular death pathways in Leber's hereditary optic neuropathy: a model of mitochondrial neurodegeneration

BBA - Bioenergetics, 2004-07, Vol.1658 (1), p.172-179

2004

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Eight human OPA1 isoforms, long and short: What are they for?

Biochimica et biophysica acta. Bioenergetics, 2018-04, Vol.1859 (4), p.263-269

2018

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Respiratory complex I is essential to induce a Warburg profile in mitochondria-defective tumor cells

Cancer & metabolism, 2013-03, Vol.1 (1), p.11-11, Article 11

2013

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OPA1: How much do we know to approach therapy?

Pharmacological research, 2018-05, Vol.131, p.199-210

2018

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Why mitochondria must fuse to maintain their genome integrity

Antioxidants & redox signaling, 2013-08, Vol.19 (4), p.379-388

2013

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Glucose transport activation in human hematopoietic cells M07e is modulated by cytosolic calcium and calmodulin

Cell calcium (Edinburgh), 2006-10, Vol.40 (4), p.373-381

2006

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Cellular and mitochondrial determination of low molecular mass organic acids by LC–MS/MS

Journal of pharmaceutical and biomedical analysis, 2018-02, Vol.150, p.33-38

2018

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Syndromic parkinsonism and dementia associated with OPA1 missense mutations

Annals of neurology, 2015-07, Vol.78 (1), p.21-38

2015

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Drug repositioning as a therapeutic strategy for neurodegenerations associated with OPA1 mutations

Human molecular genetics, 2021-01, Vol.29 (22), p.3631-3645

2021

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Coenzyme Q biosynthesis inhibition induces HIF‐1α stabilization and metabolic switch toward glycolysis

The FEBS journal, 2021-03, Vol.288 (6), p.1956-1974

2021

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Complex II phosphorylation is triggered by unbalanced redox homeostasis in cells lacking complex III

Biochimica et biophysica acta, 2018-03, Vol.1859 (3), p.182-190

2018

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Deciphering OPA1 mutations pathogenicity by combined analysis of human, mouse and yeast cell models

Biochimica et biophysica acta. Molecular basis of disease, 2018-10, Vol.1864 (10), p.3496-3514

2018

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Different mtDNA mutations modify tumor progression in dependence of the degree of respiratory complex I impairment

Human molecular genetics, 2014-03, Vol.23 (6), p.1453-1466

2014

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OPA1 Isoforms in the Hierarchical Organization of Mitochondrial Functions

Mitochondria: Biogenesis and mitophagy balance in segregation and clonal expansion of mitochondrial DNA mutations

Learning from oncocytic tumors: Why choose inefficient mitochondria?

Molecular mechanisms of superoxide production by complex III: A bacterial versus human mitochondrial comparative case study

The effects of idebenone on mitochondrial bioenergetics

OPA1 links human mitochondrial genome maintenance to mtDNA replication and distribution

Organization of the Respiratory Supercomplexes in Cells with Defective Complex III: Structural Features and Metabolic Consequences

Disruptive mitochondrial DNA mutations in complex I subunits are markers of oncocytic phenotype in thyroid tumors

Respiratory Complex I Dysfunction Due to Mitochondrial DNA Mutations Shifts the Voltage Threshold for Opening of the Permeability Transition Pore toward Resting Levels

The background of mitochondrial DNA haplogroup J increases the sensitivity of Leber's hereditary optic neuropathy cells to 2,5-hexanedione toxicity

The cytochrome b p.278Y>C mutation causative of a multisystem disorder enhances superoxide production and alters supramolecular interactions of respiratory chain complexes

Leber's Hereditary Optic Neuropathy (LHON) Pathogenic Mutations Induce Mitochondrial-dependent Apoptotic Death in Transmitochondrial Cells Incubated with Galactose Medium

Bioenergetics shapes cellular death pathways in Leber's hereditary optic neuropathy: a model of mitochondrial neurodegeneration

Eight human OPA1 isoforms, long and short: What are they for?

Respiratory complex I is essential to induce a Warburg profile in mitochondria-defective tumor cells

OPA1: How much do we know to approach therapy?

Why mitochondria must fuse to maintain their genome integrity

Glucose transport activation in human hematopoietic cells M07e is modulated by cytosolic calcium and calmodulin

Cellular and mitochondrial determination of low molecular mass organic acids by LC–MS/MS

Syndromic parkinsonism and dementia associated with OPA1 missense mutations

Drug repositioning as a therapeutic strategy for neurodegenerations associated with OPA1 mutations

Coenzyme Q biosynthesis inhibition induces HIF‐1α stabilization and metabolic switch toward glycolysis

Complex II phosphorylation is triggered by unbalanced redox homeostasis in cells lacking complex III

Deciphering OPA1 mutations pathogenicity by combined analysis of human, mouse and yeast cell models

Different mtDNA mutations modify tumor progression in dependence of the degree of respiratory complex I impairment

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