UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Ion Channel Macromolecular Complexes in Cardiomyocytes: Roles in Sudden Cardiac Death

Circulation research, 2015-06, Vol.116 (12), p.1971-1988

2015

Volltextzugriff (PDF)

A detailed analysis of single‐channel Nav1.5 recordings does not reveal any cooperative gating

The Journal of physiology, 2023-09, Vol.601 (17), p.3847-3868

2023

Volltextzugriff (PDF)

Deletion of Trpm4 Alters the Function of the Nav1.5 Channel in Murine Cardiac Myocytes

International journal of molecular sciences, 2021-04, Vol.22 (7), p.3401

2021

Volltextzugriff (PDF)

The dispensability of 14-3-3 proteins for the regulation of human cardiac sodium channel Nav1.5

PloS one, 2024-03, Vol.19 (3), p.e0298820-e0298820

2024

Volltextzugriff (PDF)

Cardiac voltage-gated calcium channel macromolecular complexes

Biochimica et biophysica acta, 2016-07, Vol.1863 (7), p.1806-1812

2016

Volltextzugriff (PDF)

Species-Specific Effects of Cation Channel TRPM4 Small-Molecule Inhibitors

Frontiers in pharmacology, 2021-07, Vol.12, p.712354-712354

2021

Volltextzugriff (PDF)

Multiomics uncover the proinflammatory role of Trpm4 deletion after myocardial infarction in mice

American journal of physiology. Heart and circulatory physiology, 2023-04, Vol.324 (4), p.H504-H518

2023

Volltextzugriff (PDF)

TRPM4 channels in the cardiovascular system: Physiology, pathophysiology, and pharmacology

Biochemical pharmacology, 2012-10, Vol.84 (7), p.873-881

2012

Volltextzugriff (PDF)

Knockdown of the TRPM4 channel alters cardiac electrophysiology and hemodynamics in a sex‐ and age‐dependent manner in mice

Physiological reports, 2023-08, Vol.11 (16), p.e15783-n/a

2023

Volltextzugriff (PDF)

Single-Molecule Localization of the Cardiac Voltage-Gated Sodium Channel Reveals Different Modes of Reorganization at Cardiomyocyte Membrane Domains

Circulation. Arrhythmia and electrophysiology, 2020-07, Vol.13 (7), p.e008241-e008241

2020

Volltextzugriff (PDF)

Bioorthogonal site-selective conjugation of fluorescent dyes to antibodies: method and potential applications

RSC advances, 2022-10, Vol.12 (44), p.2836-28317

2022

Volltextzugriff (PDF)

Dystrophin and calcium current are decreased in cardiomyocytes expressing Cre enzyme driven by αMHC but not TNT promoter

Scientific reports, 2019-12, Vol.9 (1), p.19422-10, Article 19422

2019

Volltextzugriff (PDF)

Functional characterization of a novel SCN5A variant associated with long QT syndrome and sudden cardiac death

International journal of legal medicine, 2019-11, Vol.133 (6), p.1733-1742

2019

Volltextzugriff (PDF)

Identification of a novel loss-of-function calcium channel gene mutation in short QT syndrome (SQTS6)

European heart journal, 2011-05, Vol.32 (9), p.1077-1088

2011

Volltextzugriff (PDF)

Biochemical re-programming of human dermal stem cells to neurons by increasing mitochondrial membrane potential

Cell death and differentiation, 2019-06, Vol.26 (6), p.1048-1061

2019

Volltextzugriff (PDF)

Annotation of functional impact of voltage‐gated sodium channel mutations

Human mutation, 2017-05, Vol.38 (5), p.485-493

2017

Volltextzugriff (PDF)

Variants of Transient Receptor Potential Melastatin Member 4 in Childhood Atrioventricular Block

Journal of the American Heart Association, 2016-05, Vol.5 (5), p.n/a

2016

Volltextzugriff (PDF)

Cardiac Sodium Channel Nav1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin

Circulation research, 2006-08, Vol.99 (4), p.407-414

2006

Volltextzugriff (PDF)

PDZ Domain-Binding Motif Regulates Cardiomyocyte Compartment-Specific Nav1.5 Channel Expression and Function

Circulation (New York, N.Y.), 2014-07, Vol.130 (2), p.147-160

2014

Volltextzugriff (PDF)

Functional Consequences of the SCN5A -p.Y1977N Mutation within the PY Ubiquitylation Motif: Discrepancy between HEK293 Cells and Transgenic Mice

International journal of molecular sciences, 2019-10, Vol.20 (20), p.5033

2019

Volltextzugriff (PDF)

Editorial: Inherited Arrhythmias of the Cardiac Sodium Channel Nav1.5

Frontiers in physiology, 2021-08, Vol.12, p.716553-716553

2021

Volltextzugriff (PDF)

β-Blockers in Congenital Short-QT Syndrome as Ion Channel Blockers

Journal of cardiovascular electrophysiology, 2013-10, Vol.24 (10), p.1172-1174

2013

Volltextzugriff (PDF)

Lateral Membrane-Specific MAGUK CASK Down-Regulates NaV1.5 Channel in Cardiac Myocytes

Circulation research, 2016-08, Vol.119 (4), p.544-556

2016

Volltextzugriff (PDF)

Targeting Ion Channel TRPM4

Chimia, 2022-12, Vol.76 (12), p.1039

2022

Volltextzugriff (PDF)

Cardiac-specific ablation of synapse-associated protein SAP97 in mice decreases potassium currents but not sodium current

Heart rhythm, 2015, Vol.12 (1), p.181-192

2015

Volltextzugriff (PDF)

1 – 25 von 95

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(92)
Mit Volltextzugriff
(92)

Publikationsform

Sprache

Englisch
(95)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Ion Channel Macromolecular Complexes in Cardiomyocytes: Roles in Sudden Cardiac Death

A detailed analysis of single‐channel Nav1.5 recordings does not reveal any cooperative gating

Deletion of Trpm4 Alters the Function of the Nav1.5 Channel in Murine Cardiac Myocytes

The dispensability of 14-3-3 proteins for the regulation of human cardiac sodium channel Nav1.5

Cardiac voltage-gated calcium channel macromolecular complexes

Species-Specific Effects of Cation Channel TRPM4 Small-Molecule Inhibitors

Multiomics uncover the proinflammatory role of Trpm4 deletion after myocardial infarction in mice

TRPM4 channels in the cardiovascular system: Physiology, pathophysiology, and pharmacology

Knockdown of the TRPM4 channel alters cardiac electrophysiology and hemodynamics in a sex‐ and age‐dependent manner in mice

Single-Molecule Localization of the Cardiac Voltage-Gated Sodium Channel Reveals Different Modes of Reorganization at Cardiomyocyte Membrane Domains

Bioorthogonal site-selective conjugation of fluorescent dyes to antibodies: method and potential applications

Dystrophin and calcium current are decreased in cardiomyocytes expressing Cre enzyme driven by αMHC but not TNT promoter

Functional characterization of a novel SCN5A variant associated with long QT syndrome and sudden cardiac death

Identification of a novel loss-of-function calcium channel gene mutation in short QT syndrome (SQTS6)

Biochemical re-programming of human dermal stem cells to neurons by increasing mitochondrial membrane potential

Annotation of functional impact of voltage‐gated sodium channel mutations

Variants of Transient Receptor Potential Melastatin Member 4 in Childhood Atrioventricular Block

Cardiac Sodium Channel Nav1.5 Is Regulated by a Multiprotein Complex Composed of Syntrophins and Dystrophin

PDZ Domain-Binding Motif Regulates Cardiomyocyte Compartment-Specific Nav1.5 Channel Expression and Function

Functional Consequences of the SCN5A -p.Y1977N Mutation within the PY Ubiquitylation Motif: Discrepancy between HEK293 Cells and Transgenic Mice

Editorial: Inherited Arrhythmias of the Cardiac Sodium Channel Nav1.5

β-Blockers in Congenital Short-QT Syndrome as Ion Channel Blockers

Lateral Membrane-Specific MAGUK CASK Down-Regulates NaV1.5 Channel in Cardiac Myocytes

Targeting Ion Channel TRPM4

Cardiac-specific ablation of synapse-associated protein SAP97 in mice decreases potassium currents but not sodium current

Suchergebnisse filtern