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A tale of two endings: Modulation of satiation by NMDA receptors on or near central and peripheral vagal afferent terminals

Physiology & behavior, 2011-11, Vol.105 (1), p.94-99

Ritter, Robert C

2011

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Gastrointestinal mechanisms of satiation for food

Physiology & behavior, 2004-04, Vol.81 (2), p.249-273

Ritter, Robert C

2004

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Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms

American journal of physiology. Regulatory, integrative and comparative physiology, 2012-01, Vol.302 (2), p.R264-R273

2012

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NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation

American journal of physiology. Regulatory, integrative and comparative physiology, 2015-01, Vol.308 (1), p.R1-R9

2015

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Modulation of vagal afferent excitation and reduction of food intake by leptin and cholecystokinin

Physiology & behavior, 2006-11, Vol.89 (4), p.477-485

2006

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CCK-Induced Reduction of Food Intake and Hindbrain MAPK Signaling Are Mediated by NMDA Receptor Activation

Endocrinology (Philadelphia), 2012-06, Vol.153 (6), p.2633-2646

2012

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Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors

American journal of physiology. Regulatory, integrative and comparative physiology, 2011-08, Vol.301 (2), p.R448-R455

2011

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Chemogenetic activation of ventral medullary astrocytes enhances feeding and corticosterone release in response to mild glucoprivation

American journal of physiology. Regulatory, integrative and comparative physiology, 2023-09, Vol.325 (3), p.R229-R237

2023

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Leptin Sensitizes NTS Neurons to Vagal Input by Increasing Postsynaptic NMDA Receptor Currents

The Journal of neuroscience, 2020-09, Vol.40 (37), p.7054-7064

2020

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CCK-sensitive C fibers activate NTS leptin receptor-expressing neurons via NMDA receptors

American journal of physiology. Regulatory, integrative and comparative physiology, 2024-05, Vol.326 (5), p.R383-R400

2024

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Lesion of NPY Receptor-expressing Neurons in Perifornical Lateral Hypothalamus Attenuates Glucoprivic Feeding

Endocrinology (Philadelphia), 2024-03, Vol.165 (5)

2024

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Blockade of hindbrain NMDA receptors containing NR2 subunits increases sucrose intake

American journal of physiology. Regulatory, integrative and comparative physiology, 2009-04, Vol.296 (4), p.R921-R928

2009

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Cholecystokinin and leptin act synergistically to reduce body weight

American journal of physiology. Regulatory, integrative and comparative physiology, 2000-04, Vol.278 (4), p.882-R890

2000

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Vagal afferent neurons projecting to the stomach and small intestine exhibit multiple N-methyl- d-aspartate receptor subunit phenotypes

Brain research, 2006-11, Vol.1119 (1), p.86-93

2006

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NPY2 receptor activation in the dorsal vagal complex increases food intake and attenuates CCK-induced satiation in male rats

American journal of physiology. Regulatory, integrative and comparative physiology, 2019-04, Vol.316 (4), p.R406-R416

2019

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N-methyl-D-aspartate receptor subunit phenotypes of vagal afferent neurons in nodose ganglia of the rat

Journal of comparative neurology (1911), 2006-06, Vol.496 (6), p.877-885

2006

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Reduced CCK-induced Fos expression in the hindbrain, nodose ganglia, and enteric neurons of rats lacking CCK-1 receptors

Brain research, 2005-07, Vol.1051 (1), p.155-163

2005

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Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist

The Journal of neuroscience, 2014-09, Vol.34 (38), p.12636-12645

2014

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Linking drug and food addiction via compulsive appetite

British journal of pharmacology, 2022-06, Vol.179 (11), p.2589-2609

2022

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Rats maintained on high-fat diets exhibit reduced satiety in response to CCK and bombesin

Peptides (New York, N.Y. : 1980), 1998, Vol.19 (8), p.1407-1415

1998

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Frequency‐dependent facilitation of synaptic throughput via postsynaptic NMDA receptors in the nucleus of the solitary tract

The Journal of physiology, 2015-01, Vol.593 (1), p.111-125

2015

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Vagal Afferent NMDA Receptors Modulate CCK-Induced Reduction of Food Intake Through Synapsin I Phosphorylation in Adult Male Rats

Endocrinology (Philadelphia), 2013-08, Vol.154 (8), p.2613-2625

2013

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Cholecystokinin increases cytosolic calcium in a subpopulation of cultured vagal afferent neurons

American journal of physiology. Regulatory, integrative and comparative physiology, 2002-12, Vol.283 (6), p.1303-R1313

2002

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High fat maintenance diet attenuates hindbrain neuronal response to CCK

Regulatory peptides, 2000-01, Vol.86 (1), p.83-88

2000

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Increased Hypothalamic Signal Transducer and Activator of Transcription 3 Phosphorylation after Hindbrain Leptin Injection

Endocrinology (Philadelphia), 2010-04, Vol.151 (4), p.1509-1519

2010

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A tale of two endings: Modulation of satiation by NMDA receptors on or near central and peripheral vagal afferent terminals

Gastrointestinal mechanisms of satiation for food

Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms

NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation

Modulation of vagal afferent excitation and reduction of food intake by leptin and cholecystokinin

CCK-Induced Reduction of Food Intake and Hindbrain MAPK Signaling Are Mediated by NMDA Receptor Activation

Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors

Chemogenetic activation of ventral medullary astrocytes enhances feeding and corticosterone release in response to mild glucoprivation

Leptin Sensitizes NTS Neurons to Vagal Input by Increasing Postsynaptic NMDA Receptor Currents

CCK-sensitive C fibers activate NTS leptin receptor-expressing neurons via NMDA receptors

Lesion of NPY Receptor-expressing Neurons in Perifornical Lateral Hypothalamus Attenuates Glucoprivic Feeding

Blockade of hindbrain NMDA receptors containing NR2 subunits increases sucrose intake

Cholecystokinin and leptin act synergistically to reduce body weight

Vagal afferent neurons projecting to the stomach and small intestine exhibit multiple N-methyl- d-aspartate receptor subunit phenotypes

NPY2 receptor activation in the dorsal vagal complex increases food intake and attenuates CCK-induced satiation in male rats

N-methyl-D-aspartate receptor subunit phenotypes of vagal afferent neurons in nodose ganglia of the rat

Reduced CCK-induced Fos expression in the hindbrain, nodose ganglia, and enteric neurons of rats lacking CCK-1 receptors

Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist

Linking drug and food addiction via compulsive appetite

Rats maintained on high-fat diets exhibit reduced satiety in response to CCK and bombesin

Frequency‐dependent facilitation of synaptic throughput via postsynaptic NMDA receptors in the nucleus of the solitary tract

Vagal Afferent NMDA Receptors Modulate CCK-Induced Reduction of Food Intake Through Synapsin I Phosphorylation in Adult Male Rats

Cholecystokinin increases cytosolic calcium in a subpopulation of cultured vagal afferent neurons

High fat maintenance diet attenuates hindbrain neuronal response to CCK

Increased Hypothalamic Signal Transducer and Activator of Transcription 3 Phosphorylation after Hindbrain Leptin Injection

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