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Pristane-Accelerated Autoimmune Disease in (SWR X NZB) F1 Mice Leads to Prominent Tubulointerstitial Inflammation and Human Lupus Nephritis-Like Fibrosis

PloS one, 2016-10, Vol.11 (10), p.e0164423-e0164423

2016

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Bad-Deficient Mice Develop Diffuse Large B Cell Lymphoma

Proceedings of the National Academy of Sciences - PNAS, 2003-08, Vol.100 (16), p.9324-9329

2003

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Survival Factor-Mediated BAD Phosphorylation Raises the Mitochondrial Threshold for Apoptosis

Developmental cell, 2002-11, Vol.3 (5), p.631-643

2002

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Activation of BAD by Therapeutic Inhibition of Epidermal Growth Factor Receptor and Transactivation by Insulin-like Growth Factor Receptor

The Journal of biological chemistry, 2002-08, Vol.277 (31), p.27643-27650

2002

Volltextzugriff (PDF)

Repeated administration of dapirolizumab pegol in a randomised phase I study is well tolerated and accompanied by improvements in several composite measures of systemic lupus erythematosus disease activity and changes in whole blood transcriptomic profiles

Annals of the rheumatic diseases, 2017-11, Vol.76 (11), p.1837

2017

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The transcription factor NF-ATc is essential for cardiac valve formation

Nature (London), 1998-03, Vol.392 (6672), p.186-190

1998

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B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: Application to autoimmune disease therapy

Cell, 1995-03, Vol.80 (5), p.707-718

1995

Volltextzugriff (PDF)

Anti‐BDCA2 monoclonal antibody inhibits plasmacytoid dendritic cell activation through Fc‐dependent and Fc‐independent mechanisms

EMBO molecular medicine, 2015-04, Vol.7 (4), p.464-476

2015

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BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

Nature, 2003-08, Vol.424 (6951), p.952-956

2003

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BAD Is a Pro-survival Factor Prior to Activation of Its Pro-apoptotic Function

The Journal of biological chemistry, 2004-10, Vol.279 (40), p.42240-42249

2004

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B7–2 (CD86) is essential for the development of IL-4-producing T cells

International immunology, 1996-10, Vol.8 (10), p.1549-1560

1996

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BAFF (B cell activating factor) transcript level in peripheral blood of patients with SLE is associated with same-day disease activity as well as global activity over the next year

Lupus science & medicine, 2015-06, Vol.2 (1), p.e000063-e000063

2015

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NFATc1 and NFATc2 Together Control Both T and B Cell Activation and Differentiation

Immunity (Cambridge, Mass.), 2001, Vol.14 (1), p.13-20

2001

Volltextzugriff (PDF)

Mouse models of cell death

Nature genetics, 2001-06, Vol.28 (2), p.113-118

2001

Volltextzugriff (PDF)

Inhibitory Function of Two NFAT Family Members in Lymphoid Homeostasis and Th2 Development

Immunity (Cambridge, Mass.), 1998-11, Vol.9 (5), p.627-635

1998

Volltextzugriff (PDF)

Delayed Lymphoid Repopulation with Defects in IL-4–Driven Responses Produced by Inactivation of NF-ATc

Immunity (Cambridge, Mass.), 1998, Vol.8 (1), p.125-134

1998

Volltextzugriff (PDF)

Pristane-Accelerated Autoimmune Disease in

PloS one, 2016-10, Vol.11 (10), p.e0164423

2016

Volltextzugriff (PDF)

Hyperproliferation and dysregulation of IL-4 expression in NF-ATp-deficient mice

Immunity (Cambridge, Mass.), 1996-04, Vol.4 (4), p.397-405

1996

Volltextzugriff (PDF)

The nuclear factor of activated T cells (NFAT) transcription factor NFATp (NFATc2) is a repressor of chondrogenesis

The Journal of experimental medicine, 2000-01, Vol.191 (1), p.9-22

2000

Volltextzugriff (PDF)

Anti‐ BDCA 2 monoclonal antibody inhibits plasmacytoid dendritic cell activation through Fc‐dependent and Fc‐independent mechanisms

EMBO molecular medicine, 2015-04, Vol.7 (4), p.464-476

2015

Volltextzugriff (PDF)

Hyperproliferation and Dysregulation ofIL-4 Expression in NF-ATp-Deficient Mice

Immunity (Cambridge, Mass.), 1996-04, Vol.4 (4), p.397-405

1996

Volltextzugriff (PDF)

A Phase 1 Study of Two Investigational Agents, ACTR087, an Autologous T Cell Product Expressing an Antibody-Coupled T Cell Receptor, in Combination with SEA-BCMA, a Novel Non-Fucosylated Monoclonal Antibody, in Subjects with Relapsed or Refractory Multiple Myeloma

Blood, 2018-11, Vol.132 (Supplement 1), p.1997-1997

2018

Volltextzugriff (PDF)

ACTR087, Autologous T Lymphocytes Expressing Antibody Coupled T-Cell Receptors (ACTR), Induces Complete Responses in Patients with Relapsed or Refractory CD20-Positive B-Cell Lymphoma, in Combination with Rituximab

Blood, 2017-12, Vol.130 (Suppl_1), p.580-580

2017

Volltextzugriff (PDF)

Clinical Application of a Modular Genomics Technique in Systemic Lupus Erythematosus: Progress towards Precision Medicine

International journal of genomics, 2016-01, Vol.2016, p.1-7

2016

Volltextzugriff (PDF)

Recombinant human interleukin 10 in the treatment of patients with mild to moderately active Crohn's disease

Gastroenterology (New York, N.Y. 1943), 2000-12, Vol.119 (6), p.1473-1482

2000

Volltextzugriff (PDF)

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Pristane-Accelerated Autoimmune Disease in (SWR X NZB) F1 Mice Leads to Prominent Tubulointerstitial Inflammation and Human Lupus Nephritis-Like Fibrosis

Bad-Deficient Mice Develop Diffuse Large B Cell Lymphoma

Survival Factor-Mediated BAD Phosphorylation Raises the Mitochondrial Threshold for Apoptosis

Activation of BAD by Therapeutic Inhibition of Epidermal Growth Factor Receptor and Transactivation by Insulin-like Growth Factor Receptor

Repeated administration of dapirolizumab pegol in a randomised phase I study is well tolerated and accompanied by improvements in several composite measures of systemic lupus erythematosus disease activity and changes in whole blood transcriptomic profiles

The transcription factor NF-ATc is essential for cardiac valve formation

B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: Application to autoimmune disease therapy

Anti‐BDCA2 monoclonal antibody inhibits plasmacytoid dendritic cell activation through Fc‐dependent and Fc‐independent mechanisms

BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

BAD Is a Pro-survival Factor Prior to Activation of Its Pro-apoptotic Function

B7–2 (CD86) is essential for the development of IL-4-producing T cells

BAFF (B cell activating factor) transcript level in peripheral blood of patients with SLE is associated with same-day disease activity as well as global activity over the next year

NFATc1 and NFATc2 Together Control Both T and B Cell Activation and Differentiation

Mouse models of cell death

Inhibitory Function of Two NFAT Family Members in Lymphoid Homeostasis and Th2 Development

Delayed Lymphoid Repopulation with Defects in IL-4–Driven Responses Produced by Inactivation of NF-ATc

Pristane-Accelerated Autoimmune Disease in

Hyperproliferation and dysregulation of IL-4 expression in NF-ATp-deficient mice

The nuclear factor of activated T cells (NFAT) transcription factor NFATp (NFATc2) is a repressor of chondrogenesis

Anti‐ BDCA 2 monoclonal antibody inhibits plasmacytoid dendritic cell activation through Fc‐dependent and Fc‐independent mechanisms

Hyperproliferation and Dysregulation ofIL-4 Expression in NF-ATp-Deficient Mice

A Phase 1 Study of Two Investigational Agents, ACTR087, an Autologous T Cell Product Expressing an Antibody-Coupled T Cell Receptor, in Combination with SEA-BCMA, a Novel Non-Fucosylated Monoclonal Antibody, in Subjects with Relapsed or Refractory Multiple Myeloma

ACTR087, Autologous T Lymphocytes Expressing Antibody Coupled T-Cell Receptors (ACTR), Induces Complete Responses in Patients with Relapsed or Refractory CD20-Positive B-Cell Lymphoma, in Combination with Rituximab

Clinical Application of a Modular Genomics Technique in Systemic Lupus Erythematosus: Progress towards Precision Medicine

Recombinant human interleukin 10 in the treatment of patients with mild to moderately active Crohn's disease

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