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Von Willebrand factor delays liver repair after acetaminophen-induced acute liver injury in mice

Journal of hepatology, 2020-01, Vol.72 (1), p.146-155

2020

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Intrinsic versus idiosyncratic drug-induced hepatotoxicity--two villains or one?

The Journal of pharmacology and experimental therapeutics, 2010-03, Vol.332 (3), p.692-697

2010

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Inflammatory stress and idiosyncratic hepatotoxicity: hints from animal models

Pharmacological reviews, 2009-09, Vol.61 (3), p.262-282

2009

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Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in mice

Blood, 2015-10, Vol.126 (15), p.1835-1843

2015

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Complement activation in acetaminophen-induced liver injury in mice

The Journal of pharmacology and experimental therapeutics, 2012-05, Vol.341 (2), p.377-385

2012

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Lipopolysaccharide and Trovafloxacin Coexposure in Mice Causes Idiosyncrasy-Like Liver Injury Dependent on Tumor Necrosis Factor-Alpha

Toxicological sciences, 2007-11, Vol.100 (1), p.259-266

2007

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Amiodarone Exposure During Modest Inflammation Induces Idiosyncrasy-like Liver Injury in Rats: Role of Tumor Necrosis Factor-alpha

Toxicological sciences, 2012-01, Vol.125 (1), p.126-133

2012

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Molecular mechanisms of hepatocellular apoptosis induced by trovafloxacin-tumor necrosis factor-alpha interaction

Toxicological sciences, 2014-01, Vol.137 (1), p.91-101

2014

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Metformin Prevents Alcohol-Induced Liver Injury in the Mouse: Critical Role of Plasminogen Activator Inhibitor-1

Gastroenterology (New York, N.Y. 1943), 2006-06, Vol.130 (7), p.2099-2112

2006

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Modest inflammation enhances diclofenac hepatotoxicity in rats: role of neutrophils and bacterial translocation

The Journal of pharmacology and experimental therapeutics, 2006-12, Vol.319 (3), p.1191-1199

2006

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Role of the coagulation system in acetaminophen‐induced hepatotoxicity in mice

Hepatology (Baltimore, Md.), 2007-10, Vol.46 (4), p.1177-1186

2007

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Trovafloxacin-induced replication stress sensitizes HepG2 cells to tumor necrosis factor-alpha-induced cytotoxicity mediated by extracellular signal-regulated kinase and ataxia telangiectasia and Rad3-related

Toxicology (Amsterdam), 2015-05, Vol.331, p.35-46

2015

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The Role of Tumor Necrosis Factor Alpha in Lipopolysaccharide/Ranitidine-Induced Inflammatory Liver Injury

Toxicological sciences, 2007-11, Vol.100 (1), p.267-280

2007

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Tumor necrosis factor-alpha potentiates the cytotoxicity of amiodarone in Hepa1c1c7 cells: roles of caspase activation and oxidative stress

Toxicological sciences, 2013-01, Vol.131 (1), p.164-178

2013

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Microarray analysis of lipopolysaccharide potentiation of trovafloxacin-induced liver injury in rats suggests a role for proinflammatory chemokines and neutrophils

The Journal of pharmacology and experimental therapeutics, 2006-03, Vol.316 (3), p.1080-1087

2006

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Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide

Toxicological sciences, 2013-11, Vol.136 (1), p.51-62

2013

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Trovafloxacin enhances the inflammatory response to a Gram-negative or a Gram-positive bacterial stimulus, resulting in neutrophil-dependent liver injury in mice

The Journal of pharmacology and experimental therapeutics, 2009-07, Vol.330 (1), p.72-78

2009

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Coexposure of Mice to Trovafloxacin and Lipopolysaccharide, a Model of Idiosyncratic Hepatotoxicity, Results in a Unique Gene Expression Profile and Interferon Gamma–Dependent Liver Injury

Toxicological sciences, 2009-01, Vol.107 (1), p.270-280

2009

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Adverse hepatic drug reactions: inflammatory episodes as consequence and contributor

Chemico-biological interactions, 2004-11, Vol.150 (1), p.35-51

2004

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Oxidative stress is important in the pathogenesis of liver injury induced by sulindac and lipopolysaccharide cotreatment

Toxicology (Amsterdam), 2010-06, Vol.272 (1), p.32-38

2010

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Ranitidine treatment during a modest inflammatory response precipitates idiosyncrasy-like liver injury in rats

The Journal of pharmacology and experimental therapeutics, 2003-10, Vol.307 (1), p.9-16

2003

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Neutrophil migration during endotoxemia

Journal of leukocyte biology, 1999-07, Vol.66 (1), p.10-24

1999

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Bacterial- and Viral-Induced Inflammation Increases Sensitivity to Acetaminophen Hepatotoxicity

Journal of Toxicology and Environmental Health, Part A, 2010-01, Vol.73 (1), p.58-73

2010

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Key Characteristics of Human Hepatotoxicants as a Basis for Identification and Characterization of the Causes of Liver Toxicity

Hepatology (Baltimore, Md.), 2021-12, Vol.74 (6), p.3486-3496

2021

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Pharmacokinetic and toxicodynamic concepts in idiosyncratic, drug-induced liver injury

Expert opinion on drug metabolism & toxicology, 2022-08, Vol.18 (7-8), p.469-481

2022

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Von Willebrand factor delays liver repair after acetaminophen-induced acute liver injury in mice

Intrinsic versus idiosyncratic drug-induced hepatotoxicity--two villains or one?

Inflammatory stress and idiosyncratic hepatotoxicity: hints from animal models

Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in mice

Complement activation in acetaminophen-induced liver injury in mice

Lipopolysaccharide and Trovafloxacin Coexposure in Mice Causes Idiosyncrasy-Like Liver Injury Dependent on Tumor Necrosis Factor-Alpha

Amiodarone Exposure During Modest Inflammation Induces Idiosyncrasy-like Liver Injury in Rats: Role of Tumor Necrosis Factor-alpha

Molecular mechanisms of hepatocellular apoptosis induced by trovafloxacin-tumor necrosis factor-alpha interaction

Metformin Prevents Alcohol-Induced Liver Injury in the Mouse: Critical Role of Plasminogen Activator Inhibitor-1

Modest inflammation enhances diclofenac hepatotoxicity in rats: role of neutrophils and bacterial translocation

Role of the coagulation system in acetaminophen‐induced hepatotoxicity in mice

Trovafloxacin-induced replication stress sensitizes HepG2 cells to tumor necrosis factor-alpha-induced cytotoxicity mediated by extracellular signal-regulated kinase and ataxia telangiectasia and Rad3-related

The Role of Tumor Necrosis Factor Alpha in Lipopolysaccharide/Ranitidine-Induced Inflammatory Liver Injury

Tumor necrosis factor-alpha potentiates the cytotoxicity of amiodarone in Hepa1c1c7 cells: roles of caspase activation and oxidative stress

Microarray analysis of lipopolysaccharide potentiation of trovafloxacin-induced liver injury in rats suggests a role for proinflammatory chemokines and neutrophils

Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide

Trovafloxacin enhances the inflammatory response to a Gram-negative or a Gram-positive bacterial stimulus, resulting in neutrophil-dependent liver injury in mice

Coexposure of Mice to Trovafloxacin and Lipopolysaccharide, a Model of Idiosyncratic Hepatotoxicity, Results in a Unique Gene Expression Profile and Interferon Gamma–Dependent Liver Injury

Adverse hepatic drug reactions: inflammatory episodes as consequence and contributor

Oxidative stress is important in the pathogenesis of liver injury induced by sulindac and lipopolysaccharide cotreatment

Ranitidine treatment during a modest inflammatory response precipitates idiosyncrasy-like liver injury in rats

Neutrophil migration during endotoxemia

Bacterial- and Viral-Induced Inflammation Increases Sensitivity to Acetaminophen Hepatotoxicity

Key Characteristics of Human Hepatotoxicants as a Basis for Identification and Characterization of the Causes of Liver Toxicity

Pharmacokinetic and toxicodynamic concepts in idiosyncratic, drug-induced liver injury

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