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Altered distributions of Gemini of coiled bodies and mitochondria in motor neurons of TDP-43 transgenic mice

Proceedings of the National Academy of Sciences - PNAS, 2010-09, Vol.107 (37), p.16325-16330

2010

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Arc/Arg3.1 Regulates an Endosomal Pathway Essential for Activity-Dependent β-Amyloid Generation

Cell, 2011-10, Vol.147 (3), p.615-628

2011

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Deletion of TDP-43 down-regulates Tbc1d1, a gene linked to obesity, and alters body fat metabolism

Proceedings of the National Academy of Sciences - PNAS, 2010-09, Vol.107 (37), p.16320-16324

2010

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Aggregation Promoting C-Terminal Truncation of α-Synuclein Is a Normal Cellular Process and Is Enhanced by the Familial Parkinson's Disease-Linked Mutations

Proceedings of the National Academy of Sciences - PNAS, 2005-02, Vol.102 (6), p.2162-2167

2005

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Motor Neuron Disease Occurring in a Mutant Dynactin Mouse Model Is Characterized by Defects in Vesicular Trafficking

The Journal of neuroscience, 2008-02, Vol.28 (9), p.1997-2005

2008

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Episodic-like memory deficits in the APPswe/PS1dE9 mouse model of Alzheimer's disease: Relationships to β-amyloid deposition and neurotransmitter abnormalities

Neurobiology of disease, 2005-04, Vol.18 (3), p.602-617

2005

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Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid β peptide in APPswe transgenic mice

Neurobiology of disease, 2003-10, Vol.14 (1), p.133-145

2003

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Morphologies, metastability, and coarsening of quantum nanoislands on the surfaces of the annealed Ag(110) and Pb(111) thin films

Journal of applied physics, 2018-11, Vol.124 (17)

2018

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Histological Evidence of Protein Aggregation in Mutant SOD1 Transgenic Mice and in Amyotrophic Lateral Sclerosis Neural Tissues

Neurobiology of disease, 2001-12, Vol.8 (6), p.933-941

2001

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Axonal Transport, Amyloid Precursor Protein, Kinesin-1, and the Processing Apparatus: Revisited

The Journal of neuroscience, 2005-03, Vol.25 (9), p.2386-2395

2005

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Mutant genes in familial Alzheimer's disease and transgenic models

Annual review of neuroscience, 1998-01, Vol.21 (1), p.479-505

1998

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Neuropathologic studies of the Baltimore Longitudinal Study of Aging (BLSA)

Journal of Alzheimer's disease, 2009-01, Vol.18 (3), p.665

2009

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Increased expression of PS1 is sufficient to elevate the level and activity of γ-secretase in vivo

PloS one, 2011-11, Vol.6 (11), p.e28179-e28179

2011

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Progressive behavioral deficits in DJ-1 -deficient mice are associated with normal nigrostriatal function

Neurobiology of disease, 2008-03, Vol.29 (3), p.505-514

2008

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The Etiopathogenesis of Parkinson Disease and Suggestions for Future Research. Part I

Journal of neuropathology and experimental neurology, 2007-04, Vol.66 (4), p.251-257

2007

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Rodent models of TDP-43: Recent advances

Brain research, 2012-06, Vol.1462, p.26-39

2012

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Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins

Neuron (Cambridge, Mass.), 1997-10, Vol.19 (4), p.939-945

1997

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Parkinson's disease alpha-synuclein transgenic mice develop neuronal mitochondrial degeneration and cell death

The Journal of neuroscience, 2006-01, Vol.26 (1), p.41-50

2006

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ALZHEIMER'S DISEASE: Genetic Studies and Transgenic Models

Annual review of genetics, 1998-01, Vol.32 (1), p.461-493

1998

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Presenilin 1 is required for Notch 1 and Dll1 expression in the paraxial mesoderm

Nature (London), 1997-05, Vol.387 (6630), p.288-292

1997

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Genetic Neurodegenerative Diseases: The Human Illness and Transgenic Models

Science (American Association for the Advancement of Science), 1998-11, Vol.282 (5391), p.1079-1083

1998

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Effects of PS1 deficiency on membrane protein trafficking in neurons

Neuron (Cambridge, Mass.), 1998-11, Vol.21 (5), p.1213-1221

1998

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Amyotrophic Lateral Sclerosis 2-Deficiency Leads to Neuronal Degeneration in Amyotrophic Lateral Sclerosis through Altered AMPA Receptor Trafficking

The Journal of neuroscience, 2006-11, Vol.26 (45), p.11798-11806

2006

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An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

Neuron (Cambridge, Mass.), 1995-06, Vol.14 (6), p.1105-1116

1995

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Endoproteolysis of Presenilin 1 and Accumulation of Processed Derivatives In Vivo

Neuron (Cambridge, Mass.), 1996-07, Vol.17 (1), p.181-190

1996

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Altered distributions of Gemini of coiled bodies and mitochondria in motor neurons of TDP-43 transgenic mice

Arc/Arg3.1 Regulates an Endosomal Pathway Essential for Activity-Dependent β-Amyloid Generation

Deletion of TDP-43 down-regulates Tbc1d1, a gene linked to obesity, and alters body fat metabolism

Aggregation Promoting C-Terminal Truncation of α-Synuclein Is a Normal Cellular Process and Is Enhanced by the Familial Parkinson's Disease-Linked Mutations

Motor Neuron Disease Occurring in a Mutant Dynactin Mouse Model Is Characterized by Defects in Vesicular Trafficking

Episodic-like memory deficits in the APPswe/PS1dE9 mouse model of Alzheimer's disease: Relationships to β-amyloid deposition and neurotransmitter abnormalities

Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid β peptide in APPswe transgenic mice

Morphologies, metastability, and coarsening of quantum nanoislands on the surfaces of the annealed Ag(110) and Pb(111) thin films

Histological Evidence of Protein Aggregation in Mutant SOD1 Transgenic Mice and in Amyotrophic Lateral Sclerosis Neural Tissues

Axonal Transport, Amyloid Precursor Protein, Kinesin-1, and the Processing Apparatus: Revisited

Mutant genes in familial Alzheimer's disease and transgenic models

Neuropathologic studies of the Baltimore Longitudinal Study of Aging (BLSA)

Increased expression of PS1 is sufficient to elevate the level and activity of γ-secretase in vivo

Progressive behavioral deficits in DJ-1 -deficient mice are associated with normal nigrostriatal function

The Etiopathogenesis of Parkinson Disease and Suggestions for Future Research. Part I

Rodent models of TDP-43: Recent advances

Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins

Parkinson's disease alpha-synuclein transgenic mice develop neuronal mitochondrial degeneration and cell death

ALZHEIMER'S DISEASE: Genetic Studies and Transgenic Models

Presenilin 1 is required for Notch 1 and Dll1 expression in the paraxial mesoderm

Genetic Neurodegenerative Diseases: The Human Illness and Transgenic Models

Effects of PS1 deficiency on membrane protein trafficking in neurons

Amyotrophic Lateral Sclerosis 2-Deficiency Leads to Neuronal Degeneration in Amyotrophic Lateral Sclerosis through Altered AMPA Receptor Trafficking

An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

Endoproteolysis of Presenilin 1 and Accumulation of Processed Derivatives In Vivo

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