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Loss of activating transcription factor 3 prevents KRAS-mediated pancreatic cancer

Oncogene, 2021-04, Vol.40 (17), p.3118-3135

2021

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Oncogenic gene expression and epigenetic remodeling of cis-regulatory elements in ASXL1-mutant chronic myelomonocytic leukemia

Nature communications, 2022-03, Vol.13 (1), p.1434-1434, Article 1434

2022

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Naringenin Prevents Obesity, Hepatic Steatosis, and Glucose Intolerance in Male Mice Independent of Fibroblast Growth Factor 21

Endocrinology (Philadelphia), 2015-06, Vol.156 (6), p.2087-2102

2015

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Fibroblast Growth Factor 21 Reduces the Severity of Cerulein-Induced Pancreatitis in Mice

Gastroenterology (New York, N.Y. 1943), 2009-11, Vol.137 (5), p.1795-1804

2009

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Deletion of Panx3 Prevents the Development of Surgically Induced Osteoarthritis

Journal of molecular medicine (Berlin, Germany), 2015-08, Vol.93 (8), p.845-856

2015

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Transcriptional Profiling of Antigen-Dependent Murine B Cell Differentiation and Memory Formation

The Journal of immunology (1950), 2007-11, Vol.179 (10), p.6808-6819

2007

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A Fibroblast Growth Factor 21-Pregnane X Receptor Pathway Downregulates Hepatic CYP3A4 in Nonalcoholic Fatty Liver Disease

Molecular pharmacology, 2016-10, Vol.90 (4), p.437-446

2016

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Epigenetic reprogramming in Mist1(-/-) mice predicts the molecular response to cerulein-induced pancreatitis

PloS one, 2014-01, Vol.9 (1), p.e84182

2014

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The absence of MIST1 leads to increased ethanol sensitivity and decreased activity of the unfolded protein response in mouse pancreatic acinar cells

PloS one, 2011-12, Vol.6 (12), p.e28863-e28863

2011

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New Aspects of the Epigenetics of Pancreatic Carcinogenesis

Epigenomes, 2020-09, Vol.4 (3), p.18

2020

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The bHLH Transcription Factor Mist1 Is Required to Maintain Exocrine Pancreas Cell Organization and Acinar Cell Identity

The Journal of cell biology, 2001-11, Vol.155 (4), p.519-530

2001

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Acinar cell reprogramming: a clinically important target in pancreatic disease

Epigenomics, 2015-04, Vol.7 (2), p.267-281

2015

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MIST1 regulates the pancreatic acinar cell expression of Atp2c2, the gene encoding secretory pathway calcium ATPase 2

Experimental cell research, 2010-10, Vol.316 (17), p.2859-2870

2010

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Heterogeneity of Diabetes: β-Cells, Phenotypes, and Precision Medicine: Proceedings of an International Symposium of the Canadian Institutes of Health Research's Institute of Nutrition, Metabolism and Diabetes and the U.S. National Institutes of Health's National Institute of Diabetes and Digestive and Kidney Diseases

Diabetes care, 2022-01, Vol.45 (1), p.3-22

2022

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Epigenetics of gastrointestinal diseases: notes from a workshop

Epigenetics, 2018-01, Vol.13 (4), p.449-457

2018

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Mice lacking the transcription factor Mist1 exhibit an altered stress response and increased sensitivity to caerulein-induced pancreatitis

American journal of physiology: Gastrointestinal and liver physiology, 2007-04, Vol.292 (4), p.G1123-G1132

2007

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The unfolded protein response: An emerging therapeutic target for pancreatitis and pancreatic ductal adenocarcinoma

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2022-01, Vol.22 (1), p.148-159

2022

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The pancreas-specific form of secretory pathway calcium ATPase 2 regulates multiple pathways involved in calcium homeostasis

Biochimica et biophysica acta. Molecular cell research, 2020-01, Vol.1867 (1), p.118567-118567, Article 118567

2020

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Activating transcription factor 3 promotes loss of the acinar cell phenotype in response to cerulein-induced pancreatitis in mice

Molecular biology of the cell, 2017-09, Vol.28 (18), p.2347-2359

2017

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Silencing of the Fibroblast growth factor 21 gene is an underlying cause of acinar cell injury in mice lacking MIST1

American journal of physiology: endocrinology and metabolism, 2014-04, Vol.306 (8), p.E916-E928

2014

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Exocrine specific expression of Connexin32 is dependent on the basic helix-loop-helix transcription factor Mist1

Journal of cell science, 2003-08, Vol.116 (Pt 16), p.3315-3325

2003

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Heterogeneity of Diabetes: β-Cells, Phenotypes, and Precision Medicine: Proceedings of an International Symposium of the Canadian Institutes of Health Research's Institute of Nutrition, Metabolism and Diabetes and the U.S. National Institutes of Health's National Institute of Diabetes and Digestive and Kidney Diseases

Diabetes (New York, N.Y.), 2022-01, Vol.71 (1), p.1-22

2022

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Embryo collection induces transient activation of XBP1 arm of the ER stress response while embryo vitrification does not

Molecular human reproduction, 2012-05, Vol.18 (5), p.229-242

2012

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Atp2c2 Is Transcribed From a Unique Transcriptional Start Site in Mouse Pancreatic Acinar Cells

Journal of cellular physiology, 2016-12, Vol.231 (12), p.2768-2778

2016

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18 F-Labeled PET Probe Targeting Enhancer of Zeste Homologue 2 (EZH2) for Cancer Imaging

ACS medicinal chemistry letters, 2019-03, Vol.10 (3), p.334-340

2019

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Loss of activating transcription factor 3 prevents KRAS-mediated pancreatic cancer

Oncogenic gene expression and epigenetic remodeling of cis-regulatory elements in ASXL1-mutant chronic myelomonocytic leukemia

Naringenin Prevents Obesity, Hepatic Steatosis, and Glucose Intolerance in Male Mice Independent of Fibroblast Growth Factor 21

Fibroblast Growth Factor 21 Reduces the Severity of Cerulein-Induced Pancreatitis in Mice

Deletion of Panx3 Prevents the Development of Surgically Induced Osteoarthritis

Transcriptional Profiling of Antigen-Dependent Murine B Cell Differentiation and Memory Formation

A Fibroblast Growth Factor 21-Pregnane X Receptor Pathway Downregulates Hepatic CYP3A4 in Nonalcoholic Fatty Liver Disease

Epigenetic reprogramming in Mist1(-/-) mice predicts the molecular response to cerulein-induced pancreatitis

The absence of MIST1 leads to increased ethanol sensitivity and decreased activity of the unfolded protein response in mouse pancreatic acinar cells

New Aspects of the Epigenetics of Pancreatic Carcinogenesis

The bHLH Transcription Factor Mist1 Is Required to Maintain Exocrine Pancreas Cell Organization and Acinar Cell Identity

Acinar cell reprogramming: a clinically important target in pancreatic disease

MIST1 regulates the pancreatic acinar cell expression of Atp2c2, the gene encoding secretory pathway calcium ATPase 2

Epigenetics of gastrointestinal diseases: notes from a workshop

Mice lacking the transcription factor Mist1 exhibit an altered stress response and increased sensitivity to caerulein-induced pancreatitis

The unfolded protein response: An emerging therapeutic target for pancreatitis and pancreatic ductal adenocarcinoma

The pancreas-specific form of secretory pathway calcium ATPase 2 regulates multiple pathways involved in calcium homeostasis

Activating transcription factor 3 promotes loss of the acinar cell phenotype in response to cerulein-induced pancreatitis in mice

Silencing of the Fibroblast growth factor 21 gene is an underlying cause of acinar cell injury in mice lacking MIST1

Exocrine specific expression of Connexin32 is dependent on the basic helix-loop-helix transcription factor Mist1

Embryo collection induces transient activation of XBP1 arm of the ER stress response while embryo vitrification does not

Atp2c2 Is Transcribed From a Unique Transcriptional Start Site in Mouse Pancreatic Acinar Cells

18 F-Labeled PET Probe Targeting Enhancer of Zeste Homologue 2 (EZH2) for Cancer Imaging

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