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Reduced calreticulin levels link endoplasmic reticulum stress and Fas-triggered cell death in motoneurons vulnerable to ALS

The Journal of neuroscience, 2012-04, Vol.32 (14), p.4901-4912

2012

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Calreticulin levels determine onset of early muscle denervation by fast motoneurons of ALS model mice

Neurobiology of disease, 2015-01, Vol.73, p.130-136

2015

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Optimised and rapid pre-clinical screening in the SOD1(G93A) transgenic mouse model of amyotrophic lateral sclerosis (ALS)

PloS one, 2011, Vol.6 (8), p.e23244

2011

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Collapsin Response Mediator Protein 4a (CRMP4a) Is Upregulated in Motoneurons of Mutant SOD1 Mice and Can Trigger Motoneuron Axonal Degeneration and Cell Death

The Journal of neuroscience, 2010-01, Vol.30 (2), p.785-796

2010

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Programmed Cell Death of Embryonic Motoneurons Triggered through the Fas Death Receptor

The Journal of cell biology, 1999-11, Vol.147 (5), p.1049-1061

1999

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Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

BMC neuroscience, 2004-11, Vol.5 (1), p.48-48

2004

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Signaling by death receptors in the nervous system

Current opinion in neurobiology, 2008-06, Vol.18 (3), p.284-291

2008

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IFNγ triggers a LIGHT-dependent selective death of motoneurons contributing to the non-cell-autonomous effects of mutant SOD1

Cell death and differentiation, 2011-05, Vol.18 (5), p.754-768

2011

Volltextzugriff (PDF)

Neuronal Cell Death

Neuron, 1998-04, Vol.20 (4), p.633-647

1998

Volltextzugriff (PDF)

Repair of the injured spinal cord by implantation of a synthetic degradable block copolymer in rat

Biomaterials, 2014-08, Vol.35 (24), p.6248-6258

2014

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Motoneuron Death Triggered by a Specific Pathway Downstream of Fas: Potentiation by ALS-Linked SOD1 Mutations

Neuron (Cambridge, Mass.), 2002-09, Vol.35 (6), p.1067-1083

2002

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FGF9: A motoneuron survival factor expressed by medial thoracic and sacral motoneurons

Journal of neuroscience research, 2000-04, Vol.60 (1), p.1-9

2000

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Trim17, a novel E3 ubiquitin-ligase, initiates neuronal apoptosis

Cell death and differentiation, 2010-12, Vol.17 (12), p.1928-1941

2010

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Motoneuron Death Triggered by a Specific Pathway Downstream of Fas

Neuron (Cambridge, Mass.), 2002-09, Vol.35 (6), p.1067-1083

2002

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A novel p.E121G SOD1 mutation in slowly progressive form of amyotrophic lateral sclerosis induces cytoplasmic aggregates in cultured motor neurons and reduces cell viability

Amyotrophic lateral sclerosis and frontotemporal degeneration, 2015-03, Vol.16 (1-2), p.131-134

2015

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A Rare Motor Neuron Deleterious Missense Mutation in the DPYSL3 (CRMP4) Gene is Associated with ALS

Human mutation, 2013-07, Vol.34 (7), p.953-960

2013

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Active killing of neurons during development and following stress: a role for p75(NTR) and Fas?

Current opinion in neurobiology, 2000-02, Vol.10 (1), p.111-117

2000

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Optimised and Rapid Pre-clinical Screening in the SOD1.sup.G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS).(Research Article)

PloS one, 2011-08, Vol.6 (8), p.e23244

2011

Volltextzugriff (PDF)

Cardiotrophin-1 requires LIFRβ to promote survival of mouse motoneurons purified by a novel technique

Journal of neuroscience research, 1999-01, Vol.55 (1), p.119-126

1999

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Death Receptors in the Selective Degeneration of Motoneurons in Amyotrophic Lateral Sclerosis

Journal of neurodegenerative diseases, 2013-01, Vol.2013, p.1-11

2013

Volltextzugriff (PDF)

Calpain inhibitors, but not caspase inhibitors, prevent actin proteolysis and DNA fragmentation during apoptosis

Journal of cell science, 1998-03, Vol.111 ( Pt 6) (6), p.713-722

1998

Volltextzugriff (PDF)

Fas/Tumor Necrosis Factor Receptor Death Signaling Is Required for Axotomy-Induced Death of Motoneurons In Vivo

The Journal of neuroscience, 2003-09, Vol.23 (24), p.8526-8531

2003

Volltextzugriff (PDF)

Killer wiles: growing interest in Fas

Nature cell biology, 2003-02, Vol.5 (2), p.91

2003

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Post-translational modifications in the survival motor neuron protein

Biochemical and biophysical research communications, 2004-11, Vol.324 (1), p.288-293

2004

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Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS)

PloS one, 2011-08, Vol.6 (8), p.e23244

2011

Volltextzugriff (PDF)

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Reduced calreticulin levels link endoplasmic reticulum stress and Fas-triggered cell death in motoneurons vulnerable to ALS

Calreticulin levels determine onset of early muscle denervation by fast motoneurons of ALS model mice

Optimised and rapid pre-clinical screening in the SOD1(G93A) transgenic mouse model of amyotrophic lateral sclerosis (ALS)

Collapsin Response Mediator Protein 4a (CRMP4a) Is Upregulated in Motoneurons of Mutant SOD1 Mice and Can Trigger Motoneuron Axonal Degeneration and Cell Death

Programmed Cell Death of Embryonic Motoneurons Triggered through the Fas Death Receptor

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

Signaling by death receptors in the nervous system

IFNγ triggers a LIGHT-dependent selective death of motoneurons contributing to the non-cell-autonomous effects of mutant SOD1

Neuronal Cell Death

Repair of the injured spinal cord by implantation of a synthetic degradable block copolymer in rat

Motoneuron Death Triggered by a Specific Pathway Downstream of Fas: Potentiation by ALS-Linked SOD1 Mutations

FGF9: A motoneuron survival factor expressed by medial thoracic and sacral motoneurons

Trim17, a novel E3 ubiquitin-ligase, initiates neuronal apoptosis

Motoneuron Death Triggered by a Specific Pathway Downstream of Fas

A novel p.E121G SOD1 mutation in slowly progressive form of amyotrophic lateral sclerosis induces cytoplasmic aggregates in cultured motor neurons and reduces cell viability

A Rare Motor Neuron Deleterious Missense Mutation in the DPYSL3 (CRMP4) Gene is Associated with ALS

Active killing of neurons during development and following stress: a role for p75(NTR) and Fas?

Optimised and Rapid Pre-clinical Screening in the SOD1.sup.G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS).(Research Article)

Cardiotrophin-1 requires LIFRβ to promote survival of mouse motoneurons purified by a novel technique

Death Receptors in the Selective Degeneration of Motoneurons in Amyotrophic Lateral Sclerosis

Calpain inhibitors, but not caspase inhibitors, prevent actin proteolysis and DNA fragmentation during apoptosis

Fas/Tumor Necrosis Factor Receptor Death Signaling Is Required for Axotomy-Induced Death of Motoneurons In Vivo

Killer wiles: growing interest in Fas

Post-translational modifications in the survival motor neuron protein

Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS)

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