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Lipid droplet formation in Mycobacterium tuberculosis infected macrophages requires IFN-γ/HIF-1α signaling and supports host defense

PLoS pathogens, 2018-01, Vol.14 (1), p.e1006874-e1006874

2018

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MicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection

PLoS pathogens, 2017-05, Vol.13 (5), p.e1006410-e1006410

2017

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Early alveolar macrophage response and IL-1R-dependent T cell priming determine transmissibility of Mycobacterium tuberculosis strains

Nature communications, 2022-02, Vol.13 (1), p.884-884, Article 884

2022

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Cytosolic access of Mycobacterium tuberculosis: critical impact of phagosomal acidification control and demonstration of occurrence in vivo

PLoS pathogens, 2015-02, Vol.11 (2), p.e1004650-e1004650

2015

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A Quantitative Analysis of Complexity of Human Pathogen-Specific CD4 T Cell Responses in Healthy M. tuberculosis Infected South Africans

PLoS pathogens, 2016-07, Vol.12 (7), p.e1005760-e1005760

2016

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Concurrent infection with Mycobacterium tuberculosis confers robust protection against secondary infection in macaques

PLoS pathogens, 2018-10, Vol.14 (10), p.e1007305-e1007305

2018

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TnSeq of Mycobacterium tuberculosis clinical isolates reveals strain-specific antibiotic liabilities

PLoS pathogens, 2018-03, Vol.14 (3), p.e1006939

2018

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Memory T cells in latent Mycobacterium tuberculosis infection are directed against three antigenic islands and largely contained in a CXCR3+CCR6+ Th1 subset

PLoS pathogens, 2013-01, Vol.9 (1), p.e1003130-e1003130

2013

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Mice infected with Mycobacterium tuberculosis are resistant to acute disease caused by secondary infection with SARS-CoV-2

PLoS pathogens, 2022-03, Vol.18 (3), p.e1010093-e1010093

2022

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Inferring Mycobacterium bovis transmission between cattle and badgers using isolates from the Randomised Badger Culling Trial

PLoS pathogens, 2021-11, Vol.17 (11), p.e1010075-e1010075

2021

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Quest for correlates of protection against tuberculosis

Clinical and vaccine immunology, 2015-03, Vol.22 (3), p.258-266

2015

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Mycobacterial infection-induced miR-206 inhibits protective neutrophil recruitment via the CXCL12/CXCR4 signalling axis

PLoS pathogens, 2021-04, Vol.17 (4), p.e1009186-e1009186

2021

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Systems genetics uncover new loci containing functional gene candidates in Mycobacterium tuberculosis-infected Diversity Outbred mice

PLoS pathogens, 2024-06, Vol.20 (6), p.e1011915

2024

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Exposure to Mycobacterium remodels alveolar macrophages and the early innate response to Mycobacterium tuberculosis infection

PLoS pathogens, 2024-01, Vol.20 (1), p.e1011871-e1011871

2024

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SIV and Mycobacterium tuberculosis synergy within the granuloma accelerates the reactivation pattern of latent tuberculosis

PLoS pathogens, 2020-07, Vol.16 (7), p.e1008413-e1008413

2020

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A protective role for type I interferon signaling following infection with Mycobacterium tuberculosis carrying the rifampicin drug resistance-conferring RpoB mutation H445Y

PLoS pathogens, 2024-04, Vol.20 (4), p.e1012137-e1012137

2024

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Alternate splicing of transcripts shape macrophage response to Mycobacterium tuberculosis infection

PLoS pathogens, 2017-03, Vol.13 (3), p.e1006236

2017

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Assessing vaccine-mediated protection in an ultra-low dose Mycobacterium tuberculosis murine model

PLoS pathogens, 2023-11, Vol.19 (11), p.e1011825-e1011825

2023

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MAIT cells are functionally impaired in a Mauritian cynomolgus macaque model of SIV and Mtb co-infection

PLoS pathogens, 2020-05, Vol.16 (5), p.e1008585-e1008585

2020

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Multiple genetic loci influence vaccine-induced protection against Mycobacterium tuberculosis in genetically diverse mice

PLoS pathogens, 2024-03, Vol.20 (3), p.e1012069-e1012069

2024

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Deficiency in Galectin-3, -8, and -9 impairs immunity to chronic Mycobacterium tuberculosis infection but not acute infection with multiple intracellular pathogens

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011088-e1011088

2023

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Engaging the CD40-CD40L pathway augments T-helper cell responses and improves control of Mycobacterium tuberculosis infection

PLoS pathogens, 2017-08, Vol.13 (8), p.e1006530-e1006530

2017

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Preexisting helminth infection induces inhibition of innate pulmonary anti-tuberculosis defense by engaging the IL-4 receptor pathway

The Journal of experimental medicine, 2011-08, Vol.208 (9), p.1863-1874

2011

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Lipid droplet formation in Mycobacterium tuberculosis infected macrophages requires IFN-γ/HIF-1α signaling and supports host defense

MicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection

Early alveolar macrophage response and IL-1R-dependent T cell priming determine transmissibility of Mycobacterium tuberculosis strains

Cytosolic access of Mycobacterium tuberculosis: critical impact of phagosomal acidification control and demonstration of occurrence in vivo

A Quantitative Analysis of Complexity of Human Pathogen-Specific CD4 T Cell Responses in Healthy M. tuberculosis Infected South Africans

Concurrent infection with Mycobacterium tuberculosis confers robust protection against secondary infection in macaques

TnSeq of Mycobacterium tuberculosis clinical isolates reveals strain-specific antibiotic liabilities

Memory T cells in latent Mycobacterium tuberculosis infection are directed against three antigenic islands and largely contained in a CXCR3+CCR6+ Th1 subset

Mice infected with Mycobacterium tuberculosis are resistant to acute disease caused by secondary infection with SARS-CoV-2

Inferring Mycobacterium bovis transmission between cattle and badgers using isolates from the Randomised Badger Culling Trial

Quest for correlates of protection against tuberculosis

Enhanced control of Mycobacterium tuberculosis extrapulmonary dissemination in mice by an arabinomannan-protein conjugate vaccine

Mycobacterial infection-induced miR-206 inhibits protective neutrophil recruitment via the CXCL12/CXCR4 signalling axis

Systems genetics uncover new loci containing functional gene candidates in Mycobacterium tuberculosis-infected Diversity Outbred mice

Exposure to Mycobacterium remodels alveolar macrophages and the early innate response to Mycobacterium tuberculosis infection

SIV and Mycobacterium tuberculosis synergy within the granuloma accelerates the reactivation pattern of latent tuberculosis

A protective role for type I interferon signaling following infection with Mycobacterium tuberculosis carrying the rifampicin drug resistance-conferring RpoB mutation H445Y

Alternate splicing of transcripts shape macrophage response to Mycobacterium tuberculosis infection

Assessing vaccine-mediated protection in an ultra-low dose Mycobacterium tuberculosis murine model

Fatty acid oxidation of alternatively activated macrophages prevents foam cell formation, but Mycobacterium tuberculosis counteracts this process via HIF-1α activation

MAIT cells are functionally impaired in a Mauritian cynomolgus macaque model of SIV and Mtb co-infection

Multiple genetic loci influence vaccine-induced protection against Mycobacterium tuberculosis in genetically diverse mice

Deficiency in Galectin-3, -8, and -9 impairs immunity to chronic Mycobacterium tuberculosis infection but not acute infection with multiple intracellular pathogens

Engaging the CD40-CD40L pathway augments T-helper cell responses and improves control of Mycobacterium tuberculosis infection

Preexisting helminth infection induces inhibition of innate pulmonary anti-tuberculosis defense by engaging the IL-4 receptor pathway

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