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Intracellular amyloid hypothesis for ultra‐early phase pathology of Alzheimer's disease

Neuropathology, 2021-04, Vol.41 (2), p.93-98

Okazawa, Hitoshi

2021

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PQBP1, an intrinsically disordered/denatured protein at the crossroad of intellectual disability and neurodegenerative diseases

Neurochemistry international, 2018-10, Vol.119, p.17-25

Okazawa, Hitoshi

2018

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Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation

Nature communications, 2021-11, Vol.12 (1), p.6565-6565, Article 6565

2021

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PQBP1: The Key to Intellectual Disability, Neurodegenerative Diseases, and Innate Immunity

International journal of molecular sciences, 2022-06, Vol.23 (11), p.6227

2022

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Ultra-Early Phase pathologies of Alzheimer’s disease and other neurodegenerative diseases

Proceedings of the Japan Academy, Series B, 2017/06/09, Vol.93(6), pp.361-377

OKAZAWA, Hitoshi

2017

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Comprehensive behavioral and molecular characterization of a new knock-in mouse model of Huntington's disease: zQ175

PloS one, 2012-12, Vol.7 (12), p.e49838-e49838

2012

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Infant brain atlases from neonates to 1- and 2-year-olds

PloS one, 2011-04, Vol.6 (4), p.e18746-e18746

2011

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YAP-dependent necrosis occurs in early stages of Alzheimer’s disease and regulates mouse model pathology

Nature communications, 2020-01, Vol.11 (1), p.507-507, Article 507

2020

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Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease

International journal of molecular sciences, 2024-04, Vol.25 (7), p.3636

2024

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Characterization of neurophysiological and behavioral changes, MRI brain volumetry and 1H MRS in zQ175 knock-in mouse model of Huntington's disease

PloS one, 2012-12, Vol.7 (12), p.e50717-e50717

2012

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Fasting activates macroautophagy in neurons of Alzheimer’s disease mouse model but is insufficient to degrade amyloid-beta

Scientific reports, 2015-07, Vol.5 (1), p.12115-12115, Article 12115

2015

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Predicting protein phenotypes based on protein-protein interaction network

PloS one, 2011-03, Vol.6 (3), p.e17668-e17668

2011

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Anti-Aβ drug screening platform using human iPS cell-derived neurons for the treatment of Alzheimer's disease

PloS one, 2011-09, Vol.6 (9), p.e25788

2011

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SIRT2 ablation has no effect on tubulin acetylation in brain, cholesterol biosynthesis or the progression of Huntington's disease phenotypes in vivo

PloS one, 2012-04, Vol.7 (4), p.e34805-e34805

2012

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Frameshift PQBP-1 mutants K192Sfs7 and R153Sfs41 implicated in X-linked intellectual disability form stable dimers

Journal of structural biology, 2019-06, Vol.206 (3), p.305-313

2019

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The intellectual disability gene PQBP1 rescues Alzheimer’s disease pathology

Molecular psychiatry, 2018-10, Vol.23 (10), p.2090-2110

2018

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PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions

Nature communications, 2023-01, Vol.14 (1), p.9-9, Article 9

2023

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In-frame Dystrophin Following Exon 51-Skipping Improves Muscle Pathology and Function in the Exon 52–Deficient mdx Mouse

Molecular therapy, 2010-11, Vol.18 (11), p.1995-2005

2010

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Role of the Drosophila YATA protein in the proper subcellular localization of COPI revealed by in vivo analysis

Genes & Genetic Systems, 2020/12/01, Vol.95(6), pp.303-314

2020

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Drebrin-like (Dbnl) Controls Neuronal Migration via Regulating N-Cadherin Expression in the Developing Cerebral Cortex

The Journal of neuroscience, 2019-01, Vol.39 (4), p.678-691

2019

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Mutant α-synuclein propagates via the lymphatic system of the brain in the monomeric state

Cell reports (Cambridge), 2023-08, Vol.42 (8), p.112962-112962, Article 112962

2023

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Methods to Image Macroautophagy in the Brain In Vivo

Methods in molecular biology (Clifton, N.J.), 2019, Vol.1880, p.529

2019

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PQBP1, an intellectual disability causative gene, affects bone development and growth

Biochemical and biophysical research communications, 2020-03, Vol.523 (4), p.894-899

2020

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Formation of polyglutamine inclusions in a wide range of non-CNS tissues in the HdhQ150 knock-in mouse model of Huntington's disease

PloS one, 2009-11, Vol.4 (11), p.e8025-e8025

2009

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Intracellular amyloid hypothesis for ultra‐early phase pathology of Alzheimer's disease

PQBP1, an intrinsically disordered/denatured protein at the crossroad of intellectual disability and neurodegenerative diseases

Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation

PQBP1: The Key to Intellectual Disability, Neurodegenerative Diseases, and Innate Immunity

Ultra-Early Phase pathologies of Alzheimer’s disease and other neurodegenerative diseases

Comprehensive behavioral and molecular characterization of a new knock-in mouse model of Huntington's disease: zQ175

Infant brain atlases from neonates to 1- and 2-year-olds

YAP-dependent necrosis occurs in early stages of Alzheimer’s disease and regulates mouse model pathology

Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease

Characterization of neurophysiological and behavioral changes, MRI brain volumetry and 1H MRS in zQ175 knock-in mouse model of Huntington's disease

Fasting activates macroautophagy in neurons of Alzheimer’s disease mouse model but is insufficient to degrade amyloid-beta

Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration

Predicting protein phenotypes based on protein-protein interaction network

Anti-Aβ drug screening platform using human iPS cell-derived neurons for the treatment of Alzheimer's disease

SIRT2 ablation has no effect on tubulin acetylation in brain, cholesterol biosynthesis or the progression of Huntington's disease phenotypes in vivo

Frameshift PQBP-1 mutants K192Sfs7 and R153Sfs41 implicated in X-linked intellectual disability form stable dimers

The intellectual disability gene PQBP1 rescues Alzheimer’s disease pathology

PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions

In-frame Dystrophin Following Exon 51-Skipping Improves Muscle Pathology and Function in the Exon 52–Deficient mdx Mouse

Role of the Drosophila YATA protein in the proper subcellular localization of COPI revealed by in vivo analysis

Drebrin-like (Dbnl) Controls Neuronal Migration via Regulating N-Cadherin Expression in the Developing Cerebral Cortex

Mutant α-synuclein propagates via the lymphatic system of the brain in the monomeric state

Methods to Image Macroautophagy in the Brain In Vivo

PQBP1, an intellectual disability causative gene, affects bone development and growth

Formation of polyglutamine inclusions in a wide range of non-CNS tissues in the HdhQ150 knock-in mouse model of Huntington's disease

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