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Both fallopian tube and ovarian surface epithelium are cells-of-origin for high-grade serous ovarian carcinoma

Nature communications, 2019-11, Vol.10 (1), p.5367-16, Article 5367

2019

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Functional Genomic Landscape of Human Breast Cancer Drivers, Vulnerabilities, and Resistance

Cell, 2016-01, Vol.164 (1-2), p.293-309

2016

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NMR-based functional profiling of RASopathies and oncogenic RAS mutations

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (12), p.4574-4579

2013

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Leptin and Insulin Act on POMC Neurons to Promote the Browning of White Fat

Cell, 2015-01, Vol.160 (1-2), p.88-104

2015

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SHP2 Inhibition Prevents Adaptive Resistance to MEK Inhibitors in Multiple Cancer Models

Cancer discovery, 2018-10, Vol.8 (10), p.1237-1249

2018

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Restoration of TET2 Function Blocks Aberrant Self-Renewal and Leukemia Progression

Cell, 2017-09, Vol.170 (6), p.1079-1095.e20

2017

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SHP2 inhibition diminishes KRASG12C cycling and promotes tumor microenvironment remodeling

The Journal of experimental medicine, 2021-01, Vol.218 (1)

2021

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The tyrosine phosphatase Shp2 (PTPN11) in cancer

Cancer and metastasis reviews, 2008-06, Vol.27 (2), p.179-192

2008

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Genome-wide CRISPR/Cas9 screens reveal shared and cell-specific mechanisms of resistance to SHP2 inhibition

The Journal of experimental medicine, 2023-05, Vol.220 (5)

2023

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Protein-tyrosine phosphatase 1B is required for HER2/Neu-induced breast cancer

Cancer research (Chicago, Ill.), 2007-03, Vol.67 (6), p.2420-2424

2007

Volltextzugriff (PDF)

Global Proteomic Assessment of the Classical Protein-Tyrosine Phosphatome and “Redoxome”

Cell, 2011-09, Vol.146 (5), p.826-840

2011

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The spliceosome is a therapeutic vulnerability in MYC-driven cancer

Nature (London), 2015-09, Vol.525 (7569), p.384-388

2015

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Selective and noncovalent targeting of RAS mutants for inhibition and degradation

Nature communications, 2021-05, Vol.12 (1), p.2656-2656, Article 2656

2021

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SHP2 Tyrosine Phosphatase Converts Parafibromin/Cdc73 from a Tumor Suppressor to an Oncogenic Driver

Molecular cell, 2011-07, Vol.43 (1), p.45-56

2011

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The ‘Shp'ing news: SH2 domain-containing tyrosine phosphatases in cell signaling

Trends in biochemical sciences (Amsterdam. Regular ed.), 2003-06, Vol.28 (6), p.284-293

2003

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Oncogenic and RASopathy-associated K-RAS mutations relieve membrane-dependent occlusion of the effector-binding site

Proceedings of the National Academy of Sciences - PNAS, 2015-05, Vol.112 (21), p.6625-6630

2015

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Vitamin C in Stem Cell Reprogramming and Cancer

Trends in cell biology, 2018-09, Vol.28 (9), p.698-708

2018

Volltextzugriff (PDF)

Distinct Roles for Neutrophils and Dendritic Cells in Inflammation and Autoimmunity in motheaten Mice

Immunity (Cambridge, Mass.), 2013-03, Vol.38 (3), p.489-501

2013

Volltextzugriff (PDF)

Endocytic vesicles act as vehicles for glucose uptake in response to growth factor stimulation

Nature communications, 2024-04, Vol.15 (1), p.2843-2843, Article 2843

2024

Volltextzugriff (PDF)

Affinity purification mass spectrometry analysis of PD-1 uncovers SAP as a new checkpoint inhibitor

Proceedings of the National Academy of Sciences - PNAS, 2018-01, Vol.115 (3), p.E468-E477

2018

Volltextzugriff (PDF)

Hepatic Oxidative Stress Promotes Insulin-STAT-5 Signaling and Obesity by Inactivating Protein Tyrosine Phosphatase N2

Cell metabolism, 2014-07, Vol.20 (1), p.85-102

2014

Volltextzugriff (PDF)

A Global Analysis of the Receptor Tyrosine Kinase-Protein Phosphatase Interactome

Molecular cell, 2017-01, Vol.65 (2), p.347-360

2017

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Regulation of CD133 by HDAC6 Promotes β-Catenin Signaling to Suppress Cancer Cell Differentiation

Cell reports (Cambridge), 2012-10, Vol.2 (4), p.951-963

2012

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Elevated Hypothalamic TCPTP in Obesity Contributes to Cellular Leptin Resistance

Cell metabolism, 2011-11, Vol.14 (5), p.684-699

2011

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Off‐target inhibition by active site‐targeting SHP2 inhibitors

FEBS open bio, 2018-09, Vol.8 (9), p.1405-1411

2018

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Both fallopian tube and ovarian surface epithelium are cells-of-origin for high-grade serous ovarian carcinoma

Functional Genomic Landscape of Human Breast Cancer Drivers, Vulnerabilities, and Resistance

NMR-based functional profiling of RASopathies and oncogenic RAS mutations

Leptin and Insulin Act on POMC Neurons to Promote the Browning of White Fat

SHP2 Inhibition Prevents Adaptive Resistance to MEK Inhibitors in Multiple Cancer Models

Restoration of TET2 Function Blocks Aberrant Self-Renewal and Leukemia Progression

SHP2 inhibition diminishes KRASG12C cycling and promotes tumor microenvironment remodeling

The tyrosine phosphatase Shp2 (PTPN11) in cancer

Genome-wide CRISPR/Cas9 screens reveal shared and cell-specific mechanisms of resistance to SHP2 inhibition

Protein-tyrosine phosphatase 1B is required for HER2/Neu-induced breast cancer

Global Proteomic Assessment of the Classical Protein-Tyrosine Phosphatome and “Redoxome”

The spliceosome is a therapeutic vulnerability in MYC-driven cancer

Selective and noncovalent targeting of RAS mutants for inhibition and degradation

SHP2 Tyrosine Phosphatase Converts Parafibromin/Cdc73 from a Tumor Suppressor to an Oncogenic Driver

The ‘Shp'ing news: SH2 domain-containing tyrosine phosphatases in cell signaling

Oncogenic and RASopathy-associated K-RAS mutations relieve membrane-dependent occlusion of the effector-binding site

Vitamin C in Stem Cell Reprogramming and Cancer

Distinct Roles for Neutrophils and Dendritic Cells in Inflammation and Autoimmunity in motheaten Mice

Endocytic vesicles act as vehicles for glucose uptake in response to growth factor stimulation

Affinity purification mass spectrometry analysis of PD-1 uncovers SAP as a new checkpoint inhibitor

Hepatic Oxidative Stress Promotes Insulin-STAT-5 Signaling and Obesity by Inactivating Protein Tyrosine Phosphatase N2

A Global Analysis of the Receptor Tyrosine Kinase-Protein Phosphatase Interactome

Regulation of CD133 by HDAC6 Promotes β-Catenin Signaling to Suppress Cancer Cell Differentiation

Elevated Hypothalamic TCPTP in Obesity Contributes to Cellular Leptin Resistance

Off‐target inhibition by active site‐targeting SHP2 inhibitors

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