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Vascular smooth muscle cells in atherosclerosis

Nature reviews cardiology, 2019-12, Vol.16 (12), p.727-744

2019

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Temporal inhibition of autophagy reveals segmental reversal of ageing with increased cancer risk

Nature communications, 2020-01, Vol.11 (1), p.307-307, Article 307

2020

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Alternative Pathways of IL-1 Activation, and Its Role in Health and Disease

Frontiers in immunology, 2020-12, Vol.11, p.613170-613170

2020

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IL‐1α cleavage by inflammatory caspases of the noncanonical inflammasome controls the senescence‐associated secretory phenotype

Aging cell, 2019-06, Vol.18 (3), p.e12946-n/a

2019

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The Coagulation and Immune Systems Are Directly Linked through the Activation of Interleukin-1α by Thrombin

Immunity (Cambridge, Mass.), 2019-04, Vol.50 (4), p.1033-1042.e6

2019

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Mitochondrial DNA Damage Can Promote Atherosclerosis Independently of Reactive Oxygen Species Through Effects on Smooth Muscle Cells and Monocytes and Correlates With Higher-Risk Plaques in Humans

Circulation (New York, N.Y.), 2013-08, Vol.128 (7), p.702-712

2013

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Intracellular Interleukin-1 Receptor 2 Binding Prevents Cleavage and Activity of Interleukin-1α, Controlling Necrosis-Induced Sterile Inflammation

Immunity (Cambridge, Mass.), 2013-02, Vol.38 (2), p.285-295

2013

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Chronic Apoptosis of Vascular Smooth Muscle Cells Accelerates Atherosclerosis and Promotes Calcification and Medial Degeneration

Circulation research, 2008-06, Vol.102 (12), p.1529-1538

2008

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Vascular Smooth Muscle Cell Apoptosis Induces Interleukin-1–Directed Inflammation: Effects of Hyperlipidemia-Mediated Inhibition of Phagocytosis

Circulation research, 2010-02, Vol.106 (2), p.363-372

2010

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Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis

Nature medicine, 2006-09, Vol.12 (9), p.1075-1080

2006

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Death Is Coming and the Clot Thickens, as Pyroptosis Feeds the Fire

Immunity (Cambridge, Mass.), 2019-06, Vol.50 (6), p.1339-1341

2019

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Smooth muscle cell apoptosis promotes vessel remodeling and repair via activation of cell migration, proliferation, and collagen synthesis

Arteriosclerosis, thrombosis, and vascular biology, 2011-11, Vol.31 (11), p.2402-2409

2011

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Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors

European journal of immunology, 2020-11, Vol.50 (11), p.1663-1675

2020

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Cigarette Smoke Prevents Apoptosis through Inhibition of Caspase Activation and Induces Necrosis

American journal of respiratory cell and molecular biology, 2003-11, Vol.29 (5), p.562-570

2003

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Cell death, damage-associated molecular patterns, and sterile inflammation in cardiovascular disease

Arteriosclerosis, thrombosis, and vascular biology, 2011-12, Vol.31 (12), p.2781-2786

2011

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Thrombin-activated interleukin-1α drives atherogenesis, but also promotes vascular smooth muscle cell proliferation and collagen production

Cardiovascular research, 2023-10, Vol.119 (12), p.2179-2189

2023

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Expansion of fibroblast-like cells may explain the CANTOS meta-analysis findings for patients with clonal hematopoiesis

Nature Cardiovascular Research, 2024-01, Vol.3 (1), p.23-25

2024

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Basic research: Killing the old: cell senescence in atherosclerosis

Nature reviews cardiology, 2017-01, Vol.14 (1), p.8

2017

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Interleukin-1α Activity in Necrotic Endothelial Cells Is Controlled by Caspase-1 Cleavage of Interleukin-1 Receptor-2: IMPLICATIONS FOR ALLOGRAFT REJECTION

The Journal of biological chemistry, 2015-10, Vol.290 (41), p.25188-25196

2015

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Evidence of a genetic background predisposing to complex regional pain syndrome type 1

Journal of medical genetics, 2024-02, Vol.61 (2), p.163-170

2024

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Platelet Isolation and Activation Assays

Bio-protocol, 2019-10, Vol.9 (20), p.e3405-e3405

2019

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The CCR5 chemokine receptor mediates vasoconstriction and stimulates intimal hyperplasia in human vessels in vitro

Cardiovascular research, 2014-03, Vol.101 (3), p.513-521

2014

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Compartmentalized Megakaryocyte Death Generates Functional Platelets Committed to Caspase-Independent Death

The Journal of cell biology, 2003-02, Vol.160 (4), p.577-587

2003

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Cause or consequence: what does macrophage apoptosis do in atherosclerosis?

Arteriosclerosis, thrombosis, and vascular biology, 2009-02, Vol.29 (2), p.153-155

2009

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Response to Letter Regarding Article, “Mitochondrial DNA Damage Can Promote Atherosclerosis Independently of Reactive Oxygen Species Through Effects on Smooth Muscle Cells and Monocytes and Correlates With Higher-Risk Plaques in Humans”

Circulation (New York, N.Y.), 2014-03, Vol.129 (12), p.e408-e408

2014

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Vascular smooth muscle cells in atherosclerosis

Temporal inhibition of autophagy reveals segmental reversal of ageing with increased cancer risk

Alternative Pathways of IL-1 Activation, and Its Role in Health and Disease

IL‐1α cleavage by inflammatory caspases of the noncanonical inflammasome controls the senescence‐associated secretory phenotype

The Coagulation and Immune Systems Are Directly Linked through the Activation of Interleukin-1α by Thrombin

Mitochondrial DNA Damage Can Promote Atherosclerosis Independently of Reactive Oxygen Species Through Effects on Smooth Muscle Cells and Monocytes and Correlates With Higher-Risk Plaques in Humans

Intracellular Interleukin-1 Receptor 2 Binding Prevents Cleavage and Activity of Interleukin-1α, Controlling Necrosis-Induced Sterile Inflammation

Chronic Apoptosis of Vascular Smooth Muscle Cells Accelerates Atherosclerosis and Promotes Calcification and Medial Degeneration

Vascular Smooth Muscle Cell Apoptosis Induces Interleukin-1–Directed Inflammation: Effects of Hyperlipidemia-Mediated Inhibition of Phagocytosis

Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis

Death Is Coming and the Clot Thickens, as Pyroptosis Feeds the Fire

Smooth muscle cell apoptosis promotes vessel remodeling and repair via activation of cell migration, proliferation, and collagen synthesis

Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors

Cigarette Smoke Prevents Apoptosis through Inhibition of Caspase Activation and Induces Necrosis

Cell death, damage-associated molecular patterns, and sterile inflammation in cardiovascular disease

Thrombin-activated interleukin-1α drives atherogenesis, but also promotes vascular smooth muscle cell proliferation and collagen production

Expansion of fibroblast-like cells may explain the CANTOS meta-analysis findings for patients with clonal hematopoiesis

Basic research: Killing the old: cell senescence in atherosclerosis

Interleukin-1α Activity in Necrotic Endothelial Cells Is Controlled by Caspase-1 Cleavage of Interleukin-1 Receptor-2: IMPLICATIONS FOR ALLOGRAFT REJECTION

Evidence of a genetic background predisposing to complex regional pain syndrome type 1

Platelet Isolation and Activation Assays

The CCR5 chemokine receptor mediates vasoconstriction and stimulates intimal hyperplasia in human vessels in vitro

Compartmentalized Megakaryocyte Death Generates Functional Platelets Committed to Caspase-Independent Death

Cause or consequence: what does macrophage apoptosis do in atherosclerosis?

Response to Letter Regarding Article, “Mitochondrial DNA Damage Can Promote Atherosclerosis Independently of Reactive Oxygen Species Through Effects on Smooth Muscle Cells and Monocytes and Correlates With Higher-Risk Plaques in Humans”

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