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Candidalysin is a fungal peptide toxin critical for mucosal infection

Nature (London), 2016-04, Vol.532 (7597), p.64-68

2016

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Candidalysin Drives Epithelial Signaling, Neutrophil Recruitment, and Immunopathology at the Vaginal Mucosa

Infection and immunity, 2018-02, Vol.86 (2)

2018

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Candida albicans yeast and hyphae are discriminated by MAPK signaling in vaginal epithelial cells

PloS one, 2011-11, Vol.6 (11), p.e26580-e26580

2011

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Probing the Mobilome: Discoveries in the Dynamic Microbiome

Trends in microbiology (Regular ed.), 2021-02, Vol.29 (2), p.158-170

2021

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Abundance and diversity of resistomes differ between healthy human oral cavities and gut

Nature communications, 2020-02, Vol.11 (1), p.693-10, Article 693

2020

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New Insights in Candida albicans Innate Immunity at the Mucosa: Toxins, Epithelium, Metabolism, and Beyond

Frontiers in cellular and infection microbiology, 2020-03, Vol.10, p.81-81

2020

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The Role of ErbB Receptors in Infection

Trends in microbiology (Regular ed.), 2017-11, Vol.25 (11), p.942-952

2017

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Adaptive immune responses to Candida albicans infection

Virulence, 2015-05, Vol.6 (4), p.327-337

2015

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Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface

Virulence, 2015-05, Vol.6 (4), p.338-346

2015

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Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection

The Journal of infectious diseases, 2019-11, Vol.220 (9), p.1477-1488

2019

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Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor

Nature communications, 2019-05, Vol.10 (1), p.2297-2297, Article 2297

2019

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Candida innate immunity at the mucosa

Seminars in cell & developmental biology, 2019-05, Vol.89, p.58-70

2019

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Candida albicans interactions with epithelial cells and mucosal immunity

Microbes and infection, 2011-11, Vol.13 (12), p.963-976

2011

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Evaluation of the role of Candida albicans agglutinin-like sequence (Als) proteins in human oral epithelial cell interactions

PloS one, 2012-03, Vol.7 (3), p.e33362-e33362

2012

Volltextzugriff (PDF)

Candidalysin triggers epithelial cellular stresses that induce necrotic death

Cellular microbiology, 2021-10, Vol.23 (10), p.e13371-n/a

2021

Volltextzugriff (PDF)

Candidalysins Are a New Family of Cytolytic Fungal Peptide Toxins

mBio, 2022-02, Vol.13 (1), p.e0351021-e0351021

2022

Volltextzugriff (PDF)

Host-mycobiome metabolic interactions in health and disease

Gut microbes, 2022-12, Vol.14 (1), p.2121576-2121576

2022

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Correction: Oral and Vaginal Epithelial Cell Lines Bind and Transfer Cell-Free Infectious HIV-1 to Permissive Cells but Are Not Productively Infected

PloS one, 2020-02, Vol.15 (2), p.e0229553-e0229553

2020

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A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells

Cell host & microbe, 2010-09, Vol.8 (3), p.225-235

2010

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Albumin Neutralizes Hydrophobic Toxins and Modulates Candida albicans Pathogenicity

mBio, 2021-06, Vol.12 (3), p.e0053121-e0053121

2021

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Aspergillus fumigatus Drives Tissue Damage via Iterative Assaults upon Mucosal Integrity and Immune Homeostasis

Infection and immunity, 2023-02, Vol.91 (2), p.e0033322-e0033322

2023

Volltextzugriff (PDF)

Oral and vaginal epithelial cell lines bind and transfer cell-free infectious HIV-1 to permissive cells but are not productively infected

PloS one, 2014-05, Vol.9 (5), p.e98077-e98077

2014

Volltextzugriff (PDF)

Candida albicans increases the aerobic glycolysis and activates MAPK–dependent inflammatory response of liver sinusoidal endothelial cells

Microbes and infection, 2024-05, Vol.26 (4), p.105305, Article 105305

2024

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Comparative analysis of microbiome in coronal and root caries

BMC oral health, 2024-07, Vol.24 (1), p.869-9, Article 869

2024

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Candidalysin is a fungal peptide toxin critical for mucosal infection

Candidalysin Drives Epithelial Signaling, Neutrophil Recruitment, and Immunopathology at the Vaginal Mucosa

Candida albicans yeast and hyphae are discriminated by MAPK signaling in vaginal epithelial cells

Probing the Mobilome: Discoveries in the Dynamic Microbiome

Abundance and diversity of resistomes differ between healthy human oral cavities and gut

New Insights in Candida albicans Innate Immunity at the Mucosa: Toxins, Epithelium, Metabolism, and Beyond

The Role of ErbB Receptors in Infection

Adaptive immune responses to Candida albicans infection

Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface

Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection

Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor

Candida innate immunity at the mucosa

Candida albicans interactions with epithelial cells and mucosal immunity

Evaluation of the role of Candida albicans agglutinin-like sequence (Als) proteins in human oral epithelial cell interactions

Candidalysin triggers epithelial cellular stresses that induce necrotic death

Candidalysins Are a New Family of Cytolytic Fungal Peptide Toxins

Host-mycobiome metabolic interactions in health and disease

Candida albicans Enhances the Progression of Oral Squamous Cell Carcinoma In Vitro and In Vivo

Correction: Oral and Vaginal Epithelial Cell Lines Bind and Transfer Cell-Free Infectious HIV-1 to Permissive Cells but Are Not Productively Infected

A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells

Albumin Neutralizes Hydrophobic Toxins and Modulates Candida albicans Pathogenicity

Aspergillus fumigatus Drives Tissue Damage via Iterative Assaults upon Mucosal Integrity and Immune Homeostasis

Oral and vaginal epithelial cell lines bind and transfer cell-free infectious HIV-1 to permissive cells but are not productively infected

Candida albicans increases the aerobic glycolysis and activates MAPK–dependent inflammatory response of liver sinusoidal endothelial cells

Comparative analysis of microbiome in coronal and root caries

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