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Lipotoxicity and Diabetic Nephropathy: Novel Mechanistic Insights and Therapeutic Opportunities

International journal of molecular sciences, 2020-04, Vol.21 (7), p.2632

2020

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Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease

PloS one, 2010-09, Vol.5 (9), p.e12859-e12859

2010

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Pathogenic Pathways and Therapeutic Approaches Targeting Inflammation in Diabetic Nephropathy

International journal of molecular sciences, 2020-05, Vol.21 (11), p.3798

2020

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Targeting inflammation in diabetic nephropathy: a tale of hope

Expert opinion on investigational drugs, 2018-11, Vol.27 (11), p.917-930

2018

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Lyso-Gb3 activates Notch1 in human podocytes

Human molecular genetics, 2015-10, Vol.24 (20), p.5720-5732

2015

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Unveiling the Clinical Benefits of High-Volume Hemodiafiltration: Optimizing the Removal of Medium-Weight Uremic Toxins and Beyond

Toxins, 2023-08, Vol.15 (9), p.531

2023

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Angiotensin II and Renal Fibrosis

Hypertension (Dallas, Tex. 1979), 2001-09, Vol.38 (3, Part 2 Suppl), p.635-638

2001

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Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation

The Journal of pathology, 2018-02, Vol.244 (2), p.227-241

2018

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Gremlin regulates renal inflammation via the vascular endothelial growth factor receptor 2 pathway

The Journal of pathology, 2015-08, Vol.236 (4), p.407-420

2015

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Mitogen-Activated Protein Kinase 14 Promotes AKI

Journal of the American Society of Nephrology, 2017-03, Vol.28 (3), p.823-836

2017

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Angiotensin II activates the Smad pathway during epithelial mesenchymal transdifferentiation

Kidney international, 2008-09, Vol.74 (5), p.585-595

2008

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Gremlin, A Potential Urinary Biomarker of Anca-Associated Crescentic Glomerulonephritis

Scientific reports, 2019-05, Vol.9 (1), p.6867-6867, Article 6867

2019

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Interleukin 17A Participates in Renal Inflammation Associated to Experimental and Human Hypertension

Frontiers in pharmacology, 2019-09, Vol.10, p.1015-1015

2019

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Targeting NF-κB by the Cell-Permeable NEMO-Binding Domain Peptide Improves Albuminuria and Renal Lesions in an Experimental Model of Type 2 Diabetic Nephropathy

International journal of molecular sciences, 2020-06, Vol.21 (12), p.4225

2020

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CTGF Promotes Inflammatory Cell Infiltration of the Renal Interstitium by Activating NF-κB

Journal of the American Society of Nephrology, 2009-07, Vol.20 (7), p.1513-1526

2009

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Angiotensin II regulates the synthesis of proinflammatory cytokines and chemokines in the kidney

Kidney international, 2002-12, Vol.62 (S82), p.S12-S22

2002

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Angiotensin-(1-7) and the g protein-coupled receptor MAS are key players in renal inflammation

PloS one, 2009-04, Vol.4 (4), p.e5406

2009

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Gremlin Regulates Tubular Epithelial to Mesenchymal Transition via VEGFR2: Potential Role in Renal Fibrosis

Frontiers in pharmacology, 2018-10, Vol.9, p.1195-1195

2018

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TWEAK (tumor necrosis factor–like weak inducer of apoptosis) activates CXCL16 expression during renal tubulointerstitial inflammation

Kidney international, 2012-06, Vol.81 (11), p.1098-1107

2012

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Tubular overexpression of gremlin induces renal damage susceptibility in mice

PloS one, 2014-07, Vol.9 (7), p.e101879

2014

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Epigenetic Modulation of Gremlin-1/NOTCH Pathway in Experimental Crescentic Immune-Mediated Glomerulonephritis

Pharmaceuticals (Basel, Switzerland), 2022-01, Vol.15 (2), p.121

2022

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Effect of CYP3A4 , CYP3A5 , MDR1 and POR Genetic Polymorphisms in Immunosuppressive Treatment in Chilean Kidney Transplanted Patients

Frontiers in pharmacology, 2021-12, Vol.12, p.674117-674117

2021

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Statins inhibit angiotensin II/Smad pathway and related vascular fibrosis, by a TGF-β-independent process

PloS one, 2010-11, Vol.5 (11), p.e14145-e14145

2010

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HSP27/HSPB1 as an adaptive podocyte antiapoptotic protein activated by high glucose and angiotensin II

Laboratory investigation, 2012-01, Vol.92 (1), p.32-45

2012

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The Evolution of Hemodialysis in Chile: Is Hemodiafiltration the Next Step?

Revista medíca de Chile, 2023-12, Vol.151 (12), p.1576

2023

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Lipotoxicity and Diabetic Nephropathy: Novel Mechanistic Insights and Therapeutic Opportunities

Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease

Pathogenic Pathways and Therapeutic Approaches Targeting Inflammation in Diabetic Nephropathy

Targeting inflammation in diabetic nephropathy: a tale of hope

Lyso-Gb3 activates Notch1 in human podocytes

Unveiling the Clinical Benefits of High-Volume Hemodiafiltration: Optimizing the Removal of Medium-Weight Uremic Toxins and Beyond

Angiotensin II and Renal Fibrosis

Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation

Gremlin regulates renal inflammation via the vascular endothelial growth factor receptor 2 pathway

Mitogen-Activated Protein Kinase 14 Promotes AKI

Angiotensin II activates the Smad pathway during epithelial mesenchymal transdifferentiation

Gremlin, A Potential Urinary Biomarker of Anca-Associated Crescentic Glomerulonephritis

Interleukin 17A Participates in Renal Inflammation Associated to Experimental and Human Hypertension

Targeting NF-κB by the Cell-Permeable NEMO-Binding Domain Peptide Improves Albuminuria and Renal Lesions in an Experimental Model of Type 2 Diabetic Nephropathy

CTGF Promotes Inflammatory Cell Infiltration of the Renal Interstitium by Activating NF-κB

Angiotensin II regulates the synthesis of proinflammatory cytokines and chemokines in the kidney

Angiotensin-(1-7) and the g protein-coupled receptor MAS are key players in renal inflammation

Gremlin Regulates Tubular Epithelial to Mesenchymal Transition via VEGFR2: Potential Role in Renal Fibrosis

TWEAK (tumor necrosis factor–like weak inducer of apoptosis) activates CXCL16 expression during renal tubulointerstitial inflammation

Tubular overexpression of gremlin induces renal damage susceptibility in mice

Epigenetic Modulation of Gremlin-1/NOTCH Pathway in Experimental Crescentic Immune-Mediated Glomerulonephritis

Effect of CYP3A4 , CYP3A5 , MDR1 and POR Genetic Polymorphisms in Immunosuppressive Treatment in Chilean Kidney Transplanted Patients

Statins inhibit angiotensin II/Smad pathway and related vascular fibrosis, by a TGF-β-independent process

HSP27/HSPB1 as an adaptive podocyte antiapoptotic protein activated by high glucose and angiotensin II

The Evolution of Hemodialysis in Chile: Is Hemodiafiltration the Next Step?

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