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The Ca2+ influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance

Nature communications, 2017-09, Vol.8 (1), p.1-14, Article 475

2017

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From α1s splicing to γ1 function: A new twist in subunit modulation of the skeletal muscle L-type Ca2+ channel

The Journal of general physiology, 2022-09, Vol.154 (9), p.1

Melzer, Werner

2022

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Voltage modulates halothane-triggered Ca2+ release in malignant hyperthermia-susceptible muscle

The Journal of general physiology, 2018-01, Vol.150 (1), p.111-125

2018

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ECC meets CEU-New focus on the backdoor for calcium ions in skeletal muscle cells

The Journal of general physiology, 2020-10, Vol.152 (10), p.1

Melzer, Werner

2020

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Fast-to-Slow Transition of Skeletal Muscle Contractile Function and Corresponding Changes in Myosin Heavy and Light Chain Formation in the R6/2 Mouse Model of Huntington's Disease

PloS one, 2016-11, Vol.11 (11), p.e0166106-e0166106

2016

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No voltage change at skeletal muscle SR membrane during Ca2+ release—just Mermaids on acid

The Journal of general physiology, 2018-08, Vol.150 (8), p.1055-1058

Melzer, Werner

2018

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When Sparks Get Old

The Journal of cell biology, 2006-08, Vol.174 (5), p.613-614

Melzer, Werner

2006

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retrograde signal from RyR1 alters DHP receptor inactivation and limits window Ca²⁺ release in muscle fibers of Y522S RyR1 knock-in mice

Proceedings of the National Academy of Sciences - PNAS, 2009-03, Vol.106 (11), p.4531-4536

2009

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Skeletal muscle fibers: Inactivated or depleted after long depolarizations?

The Journal of general physiology, 2013-05, Vol.141 (5), p.517-520

Melzer, Werner

2013

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No voltage change at skeletal muscle SR membrane during Ca 2+ release-just Mermaids on acid

The Journal of general physiology, 2018-08, Vol.150 (8), p.1055

Melzer, Werner

2018

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Loss of S100A1 expression leads to Ca2+ release potentiation in mutant mice with disrupted CaM and S100A1 binding to CaMBD2 of RyR1

Physiological reports, 2018-08, Vol.6 (15), p.e13822-n/a

2018

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Calcium transients in developing mouse skeletal muscle fibres

The Journal of physiology, 2005-04, Vol.564 (2), p.451-464

2005

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Local calcium signals induced by hyper-osmotic stress in mammalian skeletal muscle cells

Journal of muscle research and cell motility, 2009-06, Vol.30 (3-4), p.97-109

2009

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Modulation of DHPR Inactivation by RyR1 Activity in Mouse Skeletal Muscle Fibers

Biophysical journal, 2014-01, Vol.106 (2), p.125a-125a

2014

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Muscle weakness in Ryr1I4895T/WT knock-in mice as a result of reduced ryanodine receptor Ca2+ ion permeation and release from the sarcoplasmic reticulum

The Journal of general physiology, 2011-01, Vol.137 (1), p.43-57

2011

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The Auxiliary Subunit γ₁ of the Skeletal Muscle L-Type Ca²⁺ Channel Is an Endogenous Ca²⁺ Antagonist

Proceedings of the National Academy of Sciences - PNAS, 2007-11, Vol.104 (45), p.17885-17890

2007

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Cross-Influence of Halothane and Voltage on Intracellular Ca2+ in Isolated Muscle Fibers of Mice Expressing Human RyR1 Mutation Y522S

Biophysical journal, 2018-02, Vol.114 (3), p.470a-470a

2018

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Voltage modulates halothane-triggered Ca^sup 2+^ release in malignant hyperthermia-susceptible muscle

The Journal of general physiology, 2018-01, Vol.150 (1), p.111

2018

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Altered Ca2+ signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington’s disease

The Journal of general physiology, 2014-11, Vol.144 (5), p.393-413

2014

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Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's disease

The Journal of general physiology, 2014-11, Vol.144 (5), p.393-413

2014

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The DHPR Calcium Current in Mammalian Skeletal Muscle: Physiological Necessity or Tolerated Evolutionary Remnant?

Biophysical journal, 2014-01, Vol.106 (2), p.126a-126a

2014

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A mutation in the GABAA receptor α1-subunit is associated with absence epilepsy

Annals of neurology, 2006-06, Vol.59 (6), p.983-987

2006

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A mutation in the GABA(A) receptor alpha(1)-subunit is associated with absence epilepsy

Annals of neurology, 2006-06, Vol.59 (6), p.983-987

2006

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Altered Inactivation of Ca2+ Current and Ca2+ Release in Mouse Muscle Fibers Deficient in the DHP receptor γ1 subunit

The Journal of general physiology, 2004-11, Vol.124 (5), p.605-618

2004

Volltextzugriff (PDF)

Altered Ca^sup 2+^ signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's disease

The Journal of general physiology, 2014-11, Vol.144 (5), p.393

2014

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The Ca2+ influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance

From α1s splicing to γ1 function: A new twist in subunit modulation of the skeletal muscle L-type Ca2+ channel

Voltage modulates halothane-triggered Ca2+ release in malignant hyperthermia-susceptible muscle

ECC meets CEU-New focus on the backdoor for calcium ions in skeletal muscle cells

Fast-to-Slow Transition of Skeletal Muscle Contractile Function and Corresponding Changes in Myosin Heavy and Light Chain Formation in the R6/2 Mouse Model of Huntington's Disease

No voltage change at skeletal muscle SR membrane during Ca2+ release—just Mermaids on acid

When Sparks Get Old

retrograde signal from RyR1 alters DHP receptor inactivation and limits window Ca²⁺ release in muscle fibers of Y522S RyR1 knock-in mice

Skeletal muscle fibers: Inactivated or depleted after long depolarizations?

No voltage change at skeletal muscle SR membrane during Ca 2+ release-just Mermaids on acid

Loss of S100A1 expression leads to Ca2+ release potentiation in mutant mice with disrupted CaM and S100A1 binding to CaMBD2 of RyR1

Calcium transients in developing mouse skeletal muscle fibres

Local calcium signals induced by hyper-osmotic stress in mammalian skeletal muscle cells

Modulation of DHPR Inactivation by RyR1 Activity in Mouse Skeletal Muscle Fibers

Muscle weakness in Ryr1I4895T/WT knock-in mice as a result of reduced ryanodine receptor Ca2+ ion permeation and release from the sarcoplasmic reticulum

The Auxiliary Subunit γ₁ of the Skeletal Muscle L-Type Ca²⁺ Channel Is an Endogenous Ca²⁺ Antagonist

Cross-Influence of Halothane and Voltage on Intracellular Ca2+ in Isolated Muscle Fibers of Mice Expressing Human RyR1 Mutation Y522S

Voltage modulates halothane-triggered Ca^sup 2+^ release in malignant hyperthermia-susceptible muscle

Altered Ca2+ signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington’s disease

Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's disease

The DHPR Calcium Current in Mammalian Skeletal Muscle: Physiological Necessity or Tolerated Evolutionary Remnant?

A mutation in the GABAA receptor α1-subunit is associated with absence epilepsy

A mutation in the GABA(A) receptor alpha(1)-subunit is associated with absence epilepsy

Altered Inactivation of Ca2+ Current and Ca2+ Release in Mouse Muscle Fibers Deficient in the DHP receptor γ1 subunit

Altered Ca^sup 2+^ signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's disease

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