UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Suppression of Somatic Expansion Delays the Onset of Pathophysiology in a Mouse Model of Huntington's Disease

PLoS genetics, 2015-08, Vol.11 (8), p.e1005267

2015

Volltextzugriff (PDF)

Metabolic Reprogramming in Astrocytes Distinguishes Region-Specific Neuronal Susceptibility in Huntington Mice

Cell metabolism, 2019-06, Vol.29 (6), p.1258-1273.e11

2019

Volltextzugriff (PDF)

Close encounters: Moving along bumps, breaks, and bubbles on expanded trinucleotide tracts

DNA repair, 2017-08, Vol.56 (C), p.144-155

2017

Volltextzugriff (PDF)

Base excision repair and double strand break repair cooperate to modulate the formation of unrepaired double strand breaks in mouse brain

Nature communications, 2024-09, Vol.15 (1), p.7726-18, Article 7726

2024

Volltextzugriff (PDF)

Mechanisms of trinucleotide repeat instability during human development

Nature reviews. Genetics, 2010-11, Vol.11 (11), p.786-799

McMurray, Cynthia T

2010

Volltextzugriff (PDF)

Features of trinucleotide repeat instability in vivo

Cell research, 2008, Vol.18 (1), p.198-213

2008

Volltextzugriff (PDF)

Targeting of XJB-5-131 to Mitochondria Suppresses Oxidative DNA Damage and Motor Decline in a Mouse Model of Huntington’s Disease

Cell reports (Cambridge), 2012-11, Vol.2 (5), p.1137-1142

2012

Volltextzugriff (PDF)

XJB-5-131-mediated improvement in physiology and behaviour of the R6/2 mouse model of Huntington's disease is age- and sex- dependent

PloS one, 2018-04, Vol.13 (4), p.e0194580-e0194580

2018

Volltextzugriff (PDF)

Crosstalk between MSH2–MSH3 and polβ promotes trinucleotide repeat expansion during base excision repair

Nature communications, 2016-08, Vol.7 (1), p.12465-15, Article 12465

2016

Volltextzugriff (PDF)

An infrared spectral biomarker accurately predicts neurodegenerative disease class in the absence of overt symptoms

Scientific reports, 2021-08, Vol.11 (1), p.15598-15598, Article 15598

2021

Volltextzugriff (PDF)

Hijacking of the mismatch repair system to cause CAG expansion and cell death in neurodegenerative disease

DNA repair, 2008-07, Vol.7 (7), p.1121-1134

McMurray, Cynthia T.

2008

Volltextzugriff (PDF)

Conformational trapping of Mismatch Recognition Complex MSH2/MSH3 on repair-resistant DNA loops

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (42), p.E837-E844

2011

Volltextzugriff (PDF)

Mutant Huntingtin Impairs Axonal Trafficking in Mammalian Neurons In Vivo and In Vitro

Molecular and Cellular Biology, 2004-09, Vol.24 (18), p.8195-8209

2004

Volltextzugriff (PDF)

(CAG) n -hairpin DNA binds to Msh2-Msh3 and changes properties of mismatch recognition

Nature structural & molecular biology, 2005-08, Vol.12 (8), p.663-670

2005

Volltextzugriff (PDF)

Retinoic acid-induced differentiation increases the rate of oxygen consumption and enhances the spare respiratory capacity of mitochondria in SH-SY5Y cells

Mechanisms of ageing and development, 2012-04, Vol.133 (4), p.176-185

2012

Volltextzugriff (PDF)

The Rad50 zinc-hook is a structure joining Mre11 complexes in DNA recombination and repair

Nature (London), 2002-08, Vol.418 (6897), p.562-566

2002

Volltextzugriff (PDF)

Distinct pools of non-glycolytic substrates differentiate brain regions and prime region-specific responses of mitochondria

PloS one, 2013-07, Vol.8 (7), p.e68831-e68831

2013

Volltextzugriff (PDF)

DNA Secondary Structure: A Common and Causative Factor for Expansion in Human Disease

Proceedings of the National Academy of Sciences - PNAS, 1999-03, Vol.96 (5), p.1823-1825

McMurray, Cynthia T.

1999

Volltextzugriff (PDF)

A brief history of triplet repeat diseases

Methods in molecular biology (Clifton, N.J.), 2013, Vol.1010, p.3

2013

Volltextzugriff (PDF)

Nuclease-Deficient FEN-1 Blocks Rad51/BRCA1-Mediated Repair and Causes Trinucleotide Repeat Instability

Molecular and Cellular Biology, 2003-09, Vol.23 (17), p.6063-6074

2003

Volltextzugriff (PDF)

Perfluorooctane Sulfonate (PFOS) Produces Dopaminergic Neuropathology in Caenorhabditis elegans

Toxicological sciences, 2019-12, Vol.172 (2), p.417-434

2019

Volltextzugriff (PDF)

OGG1 initiates age-dependent CAG trinucleotide expansion in somatic cells

Nature, 2007-05, Vol.447 (7143), p.447-452

2007

Volltextzugriff (PDF)

Sculpting of DNA at Abasic Sites by DNA Glycosylase Homolog Mag2

Structure (London), 2013-01, Vol.21 (1), p.154-166

2013

Volltextzugriff (PDF)

Trinucleotide expansion in disease: why is there a length threshold?

Current opinion in genetics & development, 2014-06, Vol.26, p.131-140

2014

Volltextzugriff (PDF)

The chicken or the egg: mitochondrial dysfunction as a cause or consequence of toxicity in Huntington’s disease

Mechanisms of ageing and development, 2017-01, Vol.161 (Pt A), p.181-197

2017

Volltextzugriff (PDF)

1 – 25 von 172

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(147)
Mit Volltextzugriff
(138)

Publikationsform

Sprache

Englisch
(171)

Erscheinungsjahr

n.n

Thema

Science & Technology
(109)
Life Sciences & Biomedicine
(93)
Humans
(74)
Animals
(73)
Biochemistry & Molecular Biology
(45)
Mice
(41)
Biological And Medical Sciences
(38)
Male
(32)
Base Sequence
(29)
Female
(28)
Cell Biology
(23)
Fundamental And Applied Biological Sciences. Psychology
(22)
Genetics & Heredity
(22)
Molecular Sequence Data
(22)
Dna
(21)
Dna Repair
(21)
Nucleic Acid Conformation
(20)
Dna - Genetics
(18)
Dna - Metabolism
(17)
Huntington Disease - Genetics
(17)

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Suppression of Somatic Expansion Delays the Onset of Pathophysiology in a Mouse Model of Huntington's Disease

Metabolic Reprogramming in Astrocytes Distinguishes Region-Specific Neuronal Susceptibility in Huntington Mice

Close encounters: Moving along bumps, breaks, and bubbles on expanded trinucleotide tracts

Base excision repair and double strand break repair cooperate to modulate the formation of unrepaired double strand breaks in mouse brain

Mechanisms of trinucleotide repeat instability during human development

Features of trinucleotide repeat instability in vivo

Targeting of XJB-5-131 to Mitochondria Suppresses Oxidative DNA Damage and Motor Decline in a Mouse Model of Huntington’s Disease

XJB-5-131-mediated improvement in physiology and behaviour of the R6/2 mouse model of Huntington's disease is age- and sex- dependent

Crosstalk between MSH2–MSH3 and polβ promotes trinucleotide repeat expansion during base excision repair

An infrared spectral biomarker accurately predicts neurodegenerative disease class in the absence of overt symptoms

Hijacking of the mismatch repair system to cause CAG expansion and cell death in neurodegenerative disease

Conformational trapping of Mismatch Recognition Complex MSH2/MSH3 on repair-resistant DNA loops

Mutant Huntingtin Impairs Axonal Trafficking in Mammalian Neurons In Vivo and In Vitro

(CAG) n -hairpin DNA binds to Msh2-Msh3 and changes properties of mismatch recognition

Retinoic acid-induced differentiation increases the rate of oxygen consumption and enhances the spare respiratory capacity of mitochondria in SH-SY5Y cells

The Rad50 zinc-hook is a structure joining Mre11 complexes in DNA recombination and repair

Distinct pools of non-glycolytic substrates differentiate brain regions and prime region-specific responses of mitochondria

DNA Secondary Structure: A Common and Causative Factor for Expansion in Human Disease

A brief history of triplet repeat diseases

Nuclease-Deficient FEN-1 Blocks Rad51/BRCA1-Mediated Repair and Causes Trinucleotide Repeat Instability

Perfluorooctane Sulfonate (PFOS) Produces Dopaminergic Neuropathology in Caenorhabditis elegans

OGG1 initiates age-dependent CAG trinucleotide expansion in somatic cells

Sculpting of DNA at Abasic Sites by DNA Glycosylase Homolog Mag2

Trinucleotide expansion in disease: why is there a length threshold?

The chicken or the egg: mitochondrial dysfunction as a cause or consequence of toxicity in Huntington’s disease

Suchergebnisse filtern