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Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain

PloS one, 2014-09, Vol.9 (9), p.e105895-e105895

2014

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Crystal structures of human glycine receptor α3 bound to a novel class of analgesic potentiators

Nature structural & molecular biology, 2017-02, Vol.24 (2), p.108-113

2017

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Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain

PloS one, 2015-09, Vol.10 (9), p.e0138140

2015

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Rare germline variants in individuals diagnosed with schizophrenia within multiplex families

Psychiatry research, 2021-09, Vol.303, p.114038-114038, Article 114038

2021

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Pharmacological effects of nonselective and subtype-selective nicotinic acetylcholine receptor agonists in animal models of persistent pain

Pain (Amsterdam), 2010-04, Vol.149 (1), p.33-49

2010

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Gating modifier toxins of voltage-gated calcium channels

Toxicon (Oxford), 2007-02, Vol.49 (2), p.202-212

McDonough, Stefan I.

2007

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Engineering Potent and Selective Analogues of GpTx-1, a Tarantula Venom Peptide Antagonist of the NaV1.7 Sodium Channel

Journal of medicinal chemistry, 2015-03, Vol.58 (5), p.2299-2314

2015

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Engineering potent and selective analogues of GpTx-1, a tarantula venom peptide antagonist of the Na(V)1.7 sodium channel

Journal of medicinal chemistry, 2015-03, Vol.58 (5), p.2299-2314

2015

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Single Residue Substitutions That Confer Voltage-Gated Sodium Ion Channel Subtype Selectivity in the NaV1.7 Inhibitory Peptide GpTx‑1

Journal of medicinal chemistry, 2016-03, Vol.59 (6), p.2704-2717

2016

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Sustained inhibition of the NaV1.7 sodium channel by engineered dimers of the domain II binding peptide GpTx-1

Bioorganic & medicinal chemistry letters, 2015-11, Vol.25 (21), p.4866-4871

2015

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Peptides targeting voltage-gated calcium channels

Current pharmaceutical design, 2008-08, Vol.14 (24), p.2480-2491

2008

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The discovery of benzoxazine sulfonamide inhibitors of NaV1.7: Tools that bridge efficacy and target engagement

Bioorganic & medicinal chemistry letters, 2017-08, Vol.27 (15), p.3477-3485

2017

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Identification of a Potent, State-Dependent Inhibitor of Nav1.7 with Oral Efficacy in the Formalin Model of Persistent Pain

Journal of medicinal chemistry, 2011-07, Vol.54 (13), p.4427-4445

2011

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Calcium ion channels: challenges and successes in drug discovery

Wiley interdisciplinary reviews. Membrane transport and signaling, 2013-03, Vol.2 (2), p.85-104

McDonough, Stefan I.

2013

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The discovery of benzoxazine sulfonamide inhibitors of Na V 1.7: Tools that bridge efficacy and target engagement

Bioorganic & medicinal chemistry letters, 2017-08, Vol.27 (15), p.3477-3485

2017

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The discovery of aminopyrazines as novel, potent Na(v)1.7 antagonists: hit-to-lead identification and SAR

Bioorganic & medicinal chemistry letters, 2012-03, Vol.22 (5), p.2033-2042

2012

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Voltage-gated calcium channels as targets for the treatment of chronic pain

Current drug targets. CNS & neurological disorders, 2004-12, Vol.3 (6), p.457-478

2004

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Mibefradil Inhibition of T-Type Calcium Channels in Cerebellar Purkinje Neurons

Molecular pharmacology, 1998-12, Vol.54 (6), p.1080-1087

1998

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Discovery and optimization of aminopyrimidinones as potent and state-dependent Nav1.7 antagonists

Bioorganic & medicinal chemistry letters, 2012-01, Vol.22 (2), p.1055-1060

2012

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Discovery and optimization of substituted piperidines as potent, selective, CNS-penetrant alpha 4 beta 2 nicotinic acetylcholine receptor potentiators

Bioorganic & medicinal chemistry, 2008-10, Vol.18 (19), p.5209-5212

2008

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Discovery and optimization of substituted piperidines as potent, selective, CNS-penetrant α4β2 nicotinic acetylcholine receptor potentiators

Bioorganic & medicinal chemistry letters, 2008-10, Vol.18 (19), p.5209-5212

2008

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Discovery and hit-to-lead optimization of pyrrolopyrimidines as potent, state-dependent Nav1.7 antagonists

Bioorganic & medicinal chemistry letters, 2012-03, Vol.22 (5), p.2052-2062

2012

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The discovery of aminopyrazines as novel, potent Nav1.7 antagonists: Hit-to-lead identification and SAR

Bioorganic & medicinal chemistry letters, 2012-03, Vol.22 (5), p.2033-2042

2012

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Interactions among toxins that inhibit N-type and P-type calcium channels

The Journal of general physiology, 2002-04, Vol.119 (4), p.313-328

2002

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Synthesis and activity of substituted carbamates as potentiators of the α4β2 nicotinic acetylcholine receptor

Bioorganic & medicinal chemistry letters, 2008-10, Vol.18 (20), p.5643-5647

2008

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Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain

Crystal structures of human glycine receptor α3 bound to a novel class of analgesic potentiators

Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain

Rare germline variants in individuals diagnosed with schizophrenia within multiplex families

Pharmacological effects of nonselective and subtype-selective nicotinic acetylcholine receptor agonists in animal models of persistent pain

Gating modifier toxins of voltage-gated calcium channels

Engineering Potent and Selective Analogues of GpTx-1, a Tarantula Venom Peptide Antagonist of the NaV1.7 Sodium Channel

Engineering potent and selective analogues of GpTx-1, a tarantula venom peptide antagonist of the Na(V)1.7 sodium channel

Single Residue Substitutions That Confer Voltage-Gated Sodium Ion Channel Subtype Selectivity in the NaV1.7 Inhibitory Peptide GpTx‑1

Sustained inhibition of the NaV1.7 sodium channel by engineered dimers of the domain II binding peptide GpTx-1

Peptides targeting voltage-gated calcium channels

The discovery of benzoxazine sulfonamide inhibitors of NaV1.7: Tools that bridge efficacy and target engagement

Identification of a Potent, State-Dependent Inhibitor of Nav1.7 with Oral Efficacy in the Formalin Model of Persistent Pain

Calcium ion channels: challenges and successes in drug discovery

The discovery of benzoxazine sulfonamide inhibitors of Na V 1.7: Tools that bridge efficacy and target engagement

The discovery of aminopyrazines as novel, potent Na(v)1.7 antagonists: hit-to-lead identification and SAR

Voltage-gated calcium channels as targets for the treatment of chronic pain

Mibefradil Inhibition of T-Type Calcium Channels in Cerebellar Purkinje Neurons

Discovery and optimization of aminopyrimidinones as potent and state-dependent Nav1.7 antagonists

Discovery and optimization of substituted piperidines as potent, selective, CNS-penetrant alpha 4 beta 2 nicotinic acetylcholine receptor potentiators

Discovery and optimization of substituted piperidines as potent, selective, CNS-penetrant α4β2 nicotinic acetylcholine receptor potentiators

Discovery and hit-to-lead optimization of pyrrolopyrimidines as potent, state-dependent Nav1.7 antagonists

The discovery of aminopyrazines as novel, potent Nav1.7 antagonists: Hit-to-lead identification and SAR

Interactions among toxins that inhibit N-type and P-type calcium channels

Synthesis and activity of substituted carbamates as potentiators of the α4β2 nicotinic acetylcholine receptor

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