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Sox2 antagonizes the Hippo pathway to maintain stemness in cancer cells

Nature communications, 2015-04, Vol.6 (1), p.6411-6411, Article 6411

2015

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Alternatively activated macrophages derived from monocytes and tissue macrophages are phenotypically and functionally distinct

Blood, 2014-05, Vol.123 (20), p.e110-e122

2014

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Mechanisms underlying differential responses to FGF signaling

Cytokine & growth factor reviews, 2005-04, Vol.16 (2), p.233-247

2005

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SOX2 Regulates YAP1 to Maintain Stemness and Determine Cell Fate in the Osteo-Adipo Lineage

Cell reports (Cambridge), 2013-06, Vol.3 (6), p.2075-2087

2013

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Osteoblast proliferation or differentiation is regulated by relative strengths of opposing signaling pathways

Journal of cellular physiology, 2008-05, Vol.215 (2), p.442-451

2008

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Distinct Functions of Sox2 Control Self-Renewal and Differentiation in the Osteoblast Lineage

Molecular and Cellular Biology, 2011-11, Vol.31 (22), p.4593-4608

2011

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Enhanced Paracrine FGF10 Expression Promotes Formation of Multifocal Prostate Adenocarcinoma and an Increase in Epithelial Androgen Receptor

Cancer cell, 2007-12, Vol.12 (6), p.572-585

2007

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Osteosarcoma cell proliferation and survival requires mGluR5 receptor activity and is blocked by Riluzole

PloS one, 2017-02, Vol.12 (2), p.e0171256-e0171256

2017

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Sox2 induction by FGF and FGFR2 activating mutations inhibits Wnt signaling and osteoblast differentiation

The Journal of cell biology, 2005-03, Vol.168 (7), p.1065-1076

2005

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Fibroblast Growth Factor Signaling Uses Multiple Mechanisms To Inhibit Wnt-Induced Transcription in Osteoblasts

Molecular and Cellular Biology, 2008-08, Vol.28 (15), p.4759-4771

2008

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Signaling by Fibroblast Growth Factors (FGF) and Fibroblast Growth Factor Receptor 2 (FGFR2)-Activating Mutations Blocks Mineralization and Induces Apoptosis in Osteoblasts

The Journal of cell biology, 2000-06, Vol.149 (6), p.1297-1308

2000

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Activation of the ERK1/2 and p38 Mitogen-activated Protein Kinase Pathways Mediates Fibroblast Growth Factor-induced Growth Arrest of Chondrocytes

The Journal of biological chemistry, 2004-01, Vol.279 (3), p.1747-1756

2004

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Sox2 is required for tumor development and cancer cell proliferation in osteosarcoma

Oncogene, 2018-08, Vol.37 (33), p.4626-4632

2018

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Perspectives on cancer stem cells in osteosarcoma

Cancer letters, 2013-09, Vol.338 (1), p.158-167

2013

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Endothelium-targeted overexpression of constitutively active FGF receptor induces cardioprotection in mice myocardial infarction

Journal of molecular and cellular cardiology, 2009-05, Vol.46 (5), p.663-673

2009

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Sox2 maintains self renewal of tumor-initiating cells in osteosarcomas

Oncogene, 2012-05, Vol.31 (18), p.2270-2282

2012

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Activation of FGF receptors by mutations in the transmembrane domain

Oncogene, 1997-03, Vol.14 (12), p.1397-1406

1997

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Sox2 is required for tumor development and cancer cells proliferation in osteosarcoma

Oncogene, 2018-05, Vol.37 (33), p.4626-4632

2018

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Sphere Formation (Osteosphere/Sarcopshere) Assay

Bio-protocol, 2012-12, Vol.2 (24)

2012

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Osteogenic and Adipogenic Differentiation of Osteosarcoma Cells

Bio-protocol, 2012-12, Vol.2 (24)

2012

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Myeloid Zinc Finger 1 and GA Binding Protein Co‐Operate with Sox2 in Regulating the Expression of Yes‐Associated Protein 1 in Cancer Cells

Stem cells (Dayton, Ohio), 2017-12, Vol.35 (12), p.2340-2350

2017

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The transcription factor Sox2 is required for osteoblast self-renewal

Cell death and differentiation, 2010-08, Vol.17 (8), p.1345-1353

2010

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Early onset of craniosynostosis in an Apert mouse model reveals critical features of this pathology

Developmental biology, 2009-04, Vol.328 (2), p.273-284

2009

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Two FGF Receptor Kinase Molecules Act in Concert to Recruit and Transphosphorylate Phospholipase Cγ

Molecular cell, 2016-01, Vol.61 (1), p.98-110

2016

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Sox2 antagonizes the Hippo pathway to maintain stemness in cancer cells

Alternatively activated macrophages derived from monocytes and tissue macrophages are phenotypically and functionally distinct

Mechanisms underlying differential responses to FGF signaling

SOX2 Regulates YAP1 to Maintain Stemness and Determine Cell Fate in the Osteo-Adipo Lineage

Osteoblast proliferation or differentiation is regulated by relative strengths of opposing signaling pathways

Distinct Functions of Sox2 Control Self-Renewal and Differentiation in the Osteoblast Lineage

Enhanced Paracrine FGF10 Expression Promotes Formation of Multifocal Prostate Adenocarcinoma and an Increase in Epithelial Androgen Receptor

Osteosarcoma cell proliferation and survival requires mGluR5 receptor activity and is blocked by Riluzole

Sox2 induction by FGF and FGFR2 activating mutations inhibits Wnt signaling and osteoblast differentiation

Fibroblast Growth Factor Signaling Uses Multiple Mechanisms To Inhibit Wnt-Induced Transcription in Osteoblasts

Signaling by Fibroblast Growth Factors (FGF) and Fibroblast Growth Factor Receptor 2 (FGFR2)-Activating Mutations Blocks Mineralization and Induces Apoptosis in Osteoblasts

miR-509-3p is clinically significant and strongly attenuates cellular migration and multi-cellular spheroids in ovarian cancer

Activation of the ERK1/2 and p38 Mitogen-activated Protein Kinase Pathways Mediates Fibroblast Growth Factor-induced Growth Arrest of Chondrocytes

Sox2 is required for tumor development and cancer cell proliferation in osteosarcoma

Perspectives on cancer stem cells in osteosarcoma

Endothelium-targeted overexpression of constitutively active FGF receptor induces cardioprotection in mice myocardial infarction

Sox2 maintains self renewal of tumor-initiating cells in osteosarcomas

Activation of FGF receptors by mutations in the transmembrane domain

Sox2 is required for tumor development and cancer cells proliferation in osteosarcoma

Sphere Formation (Osteosphere/Sarcopshere) Assay

Osteogenic and Adipogenic Differentiation of Osteosarcoma Cells

Myeloid Zinc Finger 1 and GA Binding Protein Co‐Operate with Sox2 in Regulating the Expression of Yes‐Associated Protein 1 in Cancer Cells

The transcription factor Sox2 is required for osteoblast self-renewal

Early onset of craniosynostosis in an Apert mouse model reveals critical features of this pathology

Two FGF Receptor Kinase Molecules Act in Concert to Recruit and Transphosphorylate Phospholipase Cγ

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