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Obesity confers macrophage memory

Science (American Association for the Advancement of Science), 2023-01, Vol.379 (6627), p.28-29

2023

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Genetic and epigenetic regulation of abdominal aortic aneurysms

Clinical genetics, 2020-06, Vol.97 (6), p.815-826

2020

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29-OR: Diabetic Wound Keratinocytes Induce Macrophage JMJD3–Mediated Nlrp3 Expression

Diabetes (New York, N.Y.), 2023-06, Vol.72 (Supplement_1), p.1

2023

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458-P: Proinflammatory Macrophage Phenotype Modulates Vascular Smooth Muscle Cell Differentiation via the Histone Demethylase JMJD3

Diabetes (New York, N.Y.), 2023-06, Vol.72 (Supplement_1), p.1

2023

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Druggable targets in the Rho pathway and their promise for therapeutic control of blood pressure

Pharmacology & therapeutics (Oxford), 2019-01, Vol.193, p.121-134

2019

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Macrophage-specific inhibition of the histone demethylase JMJD3 decreases STING and pathologic inflammation in diabetic wound repair

Cellular & molecular immunology, 2022-11, Vol.19 (11), p.1251-1262

2022

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The Role of Epigenetic Modifications in Abdominal Aortic Aneurysm Pathogenesis

Biomolecules (Basel, Switzerland), 2022-01, Vol.12 (2), p.172

2022

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A novel familial truncating mutation in the filamin C gene associated with cardiac arrhythmias

European journal of medical genetics, 2019-04, Vol.62 (4), p.282-285

2019

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Transcriptional and posttranscriptional regulation of the SMC-selective blood pressure-associated gene, ARHGAP42

American journal of physiology. Heart and circulatory physiology, 2020-02, Vol.318 (2), p.H413-H424

2020

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Blood pressure-associated polymorphism controls ARHGAP42 expression via serum response factor DNA binding

The Journal of clinical investigation, 2017-02, Vol.127 (2), p.670-680

2017

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Prdm6 drives ductus arteriosus closure by promoting ductus arteriosus smooth muscle cell identity and contractility

JCI insight, 2023-03, Vol.8 (5)

2023

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Nuclear RhoA signaling regulates MRTF-dependent SMC-specific transcription

American journal of physiology. Heart and circulatory physiology, 2014-08, Vol.307 (3), p.H379-H390

2014

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Histone Demethylase JARID1c Regulates TH17 T-cells in Diabetic Wounds by Increasing IL-6 Expression in Plasmacytoid Dendritic Cells

JVS-vascular science, 2023, Vol.4, p.100140, Article 100140

2023

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The Histone Demethylase JMJD3 Regulates Smooth Muscle Differentiation and Is Associated With Blood Pressure

JVS-vascular science, 2023, Vol.4, p.100149, Article 100149

2023

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The histone methyltransferase Mixed-lineage-leukemia-1 drives T cell phenotype via Notch signaling in diabetic tissue repair

JCI insight, 2024-10, Vol.9 (19)

2024

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The STAT3/SETDB2 axis dictates NF-κB-mediated inflammation in macrophages during wound repair

JCI insight, 2024-10, Vol.9 (20)

2024

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Histone demethylase JARID1C/KDM5C regulates Th17 cells by increasing IL-6 expression in diabetic plasmacytoid dendritic cells

JCI insight, 2024-06, Vol.9 (12)

2024

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MOF Expression Regulates Interferon β in Diabetic Wound Macrophages and Impairs Tissue Repair

JVS-vascular science, 2022, Vol.3, p.408-408

2022

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Epigenetically Altered Diabetic Wound Plasmacytoid Dendritic Cells Direct Wound CD4 T-cells toward a Th17 Phenotype

JVS-vascular science, 2022, Vol.3, p.422-422

2022

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Mixed-lineage Leukemia 1 Regulates T-Cell Phenotype and Inflammation in Diabetic Wounds

JVS-vascular science, 2022, Vol.3, p.406-406

2022

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Spinal Manipulation for the Treatment of Hypertension: A Systematic Qualitative Literature Review

Journal of manipulative and physiological therapeutics, 2012-03, Vol.35 (3), p.235-243

2012

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Mixed-Lineage Leukemia 1 (Mll1) Drives Notch Signaling and Regulates T-Cell Phenotype and Inflammation in Diabetic Wounds

Journal of the American College of Surgeons, 2022-11, Vol.235 (5), p.S323-S324

2022

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Monocyte Toll-like Receptor 4 Signaling Is Essential for Post Thrombotic Urokinase Expression

JVS-vascular science, 2023, Vol.4, p.100135, Article 100135

2023

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Inflammatory Macrophages Dictate Fibroblast Function Via Epigenetic Reprogramming in Diabetic Wounds

JVS-vascular science, 2023, Vol.4, p.100143, Article 100143

2023

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Abstract 475: Histone Demethylase JARID1c Regulates T H 17 T-cells In Diabetic Wounds By Increasing IL-6 Expression In Plasmacytoid Dendritic Cells

Arteriosclerosis, thrombosis, and vascular biology, 2023-05, Vol.43 (Suppl_1)

2023

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Obesity confers macrophage memory

Genetic and epigenetic regulation of abdominal aortic aneurysms

29-OR: Diabetic Wound Keratinocytes Induce Macrophage JMJD3–Mediated Nlrp3 Expression

458-P: Proinflammatory Macrophage Phenotype Modulates Vascular Smooth Muscle Cell Differentiation via the Histone Demethylase JMJD3

Druggable targets in the Rho pathway and their promise for therapeutic control of blood pressure

Macrophage-specific inhibition of the histone demethylase JMJD3 decreases STING and pathologic inflammation in diabetic wound repair

The Role of Epigenetic Modifications in Abdominal Aortic Aneurysm Pathogenesis

A novel familial truncating mutation in the filamin C gene associated with cardiac arrhythmias

Transcriptional and posttranscriptional regulation of the SMC-selective blood pressure-associated gene, ARHGAP42

Blood pressure-associated polymorphism controls ARHGAP42 expression via serum response factor DNA binding

Prdm6 drives ductus arteriosus closure by promoting ductus arteriosus smooth muscle cell identity and contractility

Nuclear RhoA signaling regulates MRTF-dependent SMC-specific transcription

Histone Demethylase JARID1c Regulates TH17 T-cells in Diabetic Wounds by Increasing IL-6 Expression in Plasmacytoid Dendritic Cells

The Histone Demethylase JMJD3 Regulates Smooth Muscle Differentiation and Is Associated With Blood Pressure

The histone methyltransferase Mixed-lineage-leukemia-1 drives T cell phenotype via Notch signaling in diabetic tissue repair

The STAT3/SETDB2 axis dictates NF-κB-mediated inflammation in macrophages during wound repair

Histone demethylase JARID1C/KDM5C regulates Th17 cells by increasing IL-6 expression in diabetic plasmacytoid dendritic cells

MOF Expression Regulates Interferon β in Diabetic Wound Macrophages and Impairs Tissue Repair

Epigenetically Altered Diabetic Wound Plasmacytoid Dendritic Cells Direct Wound CD4 T-cells toward a Th17 Phenotype

Mixed-lineage Leukemia 1 Regulates T-Cell Phenotype and Inflammation in Diabetic Wounds

Spinal Manipulation for the Treatment of Hypertension: A Systematic Qualitative Literature Review

Mixed-Lineage Leukemia 1 (Mll1) Drives Notch Signaling and Regulates T-Cell Phenotype and Inflammation in Diabetic Wounds

Monocyte Toll-like Receptor 4 Signaling Is Essential for Post Thrombotic Urokinase Expression

Inflammatory Macrophages Dictate Fibroblast Function Via Epigenetic Reprogramming in Diabetic Wounds

Abstract 475: Histone Demethylase JARID1c Regulates T H 17 T-cells In Diabetic Wounds By Increasing IL-6 Expression In Plasmacytoid Dendritic Cells

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