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Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications

The FEBS journal, 2019-01, Vol.286 (2), p.241-278

2019

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Stalling the Engine of Resistance: Targeting Cancer Metabolism to Overcome Therapeutic Resistance

Cancer research (Chicago, Ill.), 2013-05, Vol.73 (9), p.2709-2717

2013

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Starvation and antimetabolic therapy promote cytokine release and recruitment of immune cells

Proceedings of the National Academy of Sciences - PNAS, 2020-05, Vol.117 (18), p.9932-9941

2020

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Cell death induced by endoplasmic reticulum stress

The FEBS journal, 2016-07, Vol.283 (14), p.2640-2652

2016

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TRAIL receptors promote constitutive and inducible IL-8 secretion in non-small cell lung carcinoma

Cell death & disease, 2022-12, Vol.13 (12), p.1046-1046, Article 1046

2022

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Glycosylation defects, offset by PEPCK-M, drive entosis in breast carcinoma cells

Cell death & disease, 2022-08, Vol.13 (8), p.730-730, Article 730

2022

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Chemokine C-C motif ligand 2 overexpression drives tissue-specific metabolic responses in the liver and muscle of mice

Scientific reports, 2020-07, Vol.10 (1), p.11954-11954, Article 11954

2020

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ATF4 mediates necrosis induced by glucose deprivation and apoptosis induced by 2‐deoxyglucose in the same cells

The FEBS journal, 2015-09, Vol.282 (18), p.3647-3658

2015

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Gene Expression Profiling as a Potential Tool for Precision Oncology in Non-Small Cell Lung Cancer

Cancers, 2021-09, Vol.13 (19), p.4734

2021

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Different Mitochondrial Intermembrane Space Proteins Are Released during Apoptosis in a Manner That Is Coordinately Initiated but Can Vary in Duration

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (31), p.11573-11578

2006

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Negligible role of TRAIL death receptors in cell death upon endoplasmic reticulum stress in B-cell malignancies

Oncogenesis (New York, NY), 2023-02, Vol.12 (1), p.6-6, Article 6

2023

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Glucose Deprivation Induces ATF4-Mediated Apoptosis through TRAIL Death Receptors

Molecular and cellular biology, 2017-05, Vol.37 (10)

2017

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Disruption of the M80-Fe ligation stimulates the translocation of cytochrome c to the cytoplasm and nucleus in nonapoptotic cells

Proceedings of the National Academy of Sciences - PNAS, 2009-02, Vol.106 (8), p.2653-2658

2009

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Resistance to Caspase-Independent Cell Death Requires Persistence of Intact Mitochondria

Developmental cell, 2010-05, Vol.18 (5), p.802-813

2010

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Efficacy of CDK4/6 inhibitors in preclinical models of malignant pleural mesothelioma

British journal of cancer, 2021-11, Vol.125 (10), p.1365-1376

2021

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Correlated three-dimensional light and electron microscopy reveals transformation of mitochondria during apoptosis

Nature cell biology, 2007-09, Vol.9 (9), p.1057-1065

2007

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The importance of being dead: cell death mechanisms assessment in anti-sarcoma therapy

Frontiers in oncology, 2015-04, Vol.5, p.82-82

2015

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Glucose-starved Cells Do Not Engage in Prosurvival Autophagy

The Journal of biological chemistry, 2013-10, Vol.288 (42), p.30387-30398

2013

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Regulation of Cancer Metabolism by Oncogenes and Tumor Suppressors

Methods in Enzymology, 2014, Vol.542, p.59-80

2014

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Hematopoietic versus Solid Cancers and T Cell Dysfunction: Looking for Similarities and Distinctions

Cancers, 2021-01, Vol.13 (2), p.284

2021

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China's reviewers need unique IDs

Nature (London), 2019-01, Vol.565 (7738), p.161-161

Muñoz-Pinedo, Cristina

2019

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Mutations in the Antioxidant KEAP1/NRF2 Pathway Define an Aggressive Subset of NSCLC Resistant to Conventional Treatments

Journal of thoracic oncology, 2019-11, Vol.14 (11), p.1881-1883

2019

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A Transcriptomic Approach Outperforms Mutational Analysis to Identify KEAP1/NFE2L2 Pathway Activation in Patients With NSCLC

Journal of thoracic oncology, 2023-11, Vol.18 (11), p.1431-1433

2023

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Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications

Stalling the Engine of Resistance: Targeting Cancer Metabolism to Overcome Therapeutic Resistance

Starvation and antimetabolic therapy promote cytokine release and recruitment of immune cells

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Cell death induced by endoplasmic reticulum stress

TRAIL receptors promote constitutive and inducible IL-8 secretion in non-small cell lung carcinoma

Glycosylation defects, offset by PEPCK-M, drive entosis in breast carcinoma cells

Chemokine C-C motif ligand 2 overexpression drives tissue-specific metabolic responses in the liver and muscle of mice

ATF4 mediates necrosis induced by glucose deprivation and apoptosis induced by 2‐deoxyglucose in the same cells

Gene Expression Profiling as a Potential Tool for Precision Oncology in Non-Small Cell Lung Cancer

Different Mitochondrial Intermembrane Space Proteins Are Released during Apoptosis in a Manner That Is Coordinately Initiated but Can Vary in Duration

Negligible role of TRAIL death receptors in cell death upon endoplasmic reticulum stress in B-cell malignancies

Glucose Deprivation Induces ATF4-Mediated Apoptosis through TRAIL Death Receptors

Disruption of the M80-Fe ligation stimulates the translocation of cytochrome c to the cytoplasm and nucleus in nonapoptotic cells

The anti-cancer drug ABTL0812 induces ER stress-mediated cytotoxic autophagy by increasing dihydroceramide levels in cancer cells

Resistance to Caspase-Independent Cell Death Requires Persistence of Intact Mitochondria

Efficacy of CDK4/6 inhibitors in preclinical models of malignant pleural mesothelioma

Correlated three-dimensional light and electron microscopy reveals transformation of mitochondria during apoptosis

The importance of being dead: cell death mechanisms assessment in anti-sarcoma therapy

Glucose-starved Cells Do Not Engage in Prosurvival Autophagy

Regulation of Cancer Metabolism by Oncogenes and Tumor Suppressors

Hematopoietic versus Solid Cancers and T Cell Dysfunction: Looking for Similarities and Distinctions

China's reviewers need unique IDs

Mutations in the Antioxidant KEAP1/NRF2 Pathway Define an Aggressive Subset of NSCLC Resistant to Conventional Treatments

A Transcriptomic Approach Outperforms Mutational Analysis to Identify KEAP1/NFE2L2 Pathway Activation in Patients With NSCLC

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