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KRAS as a Therapeutic Target

Clinical cancer research, 2015-04, Vol.21 (8), p.1797-1801

McCormick, Frank

2015

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Targeting RAF kinases for cancer therapy: BRAF-mutated melanoma and beyond

Nature reviews. Cancer, 2014-07, Vol.14 (7), p.455-467

2014

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Complications and Reoperations During and After Hip Arthroscopy: A Systematic Review of 92 Studies and More Than 6,000 Patients

Arthroscopy, 2013-03, Vol.29 (3), p.589-595

2013

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The Metabolic Landscape of RAS-Driven Cancers from biology to therapy

Nature cancer, 2021-03, Vol.2 (3), p.271-283

2021

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The duality of human oncoproteins: drivers of cancer and congenital disorders

Nature reviews. Cancer, 2020-07, Vol.20 (7), p.383-397

2020

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Trends in the Surgical Treatment of Articular Cartilage Lesions in the United States: An Analysis of a Large Private-Payer Database Over a Period of 8 Years

Arthroscopy, 2014-02, Vol.30 (2), p.222-226

2014

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Long-Term Outcomes After Bankart Shoulder Stabilization

Arthroscopy, 2013-05, Vol.29 (5), p.920-933

2013

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Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition

Nature (London), 2017-11, Vol.551 (7679), p.247-250

2017

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Undermining Glutaminolysis Bolsters Chemotherapy While NRF2 Promotes Chemoresistance in KRAS-Driven Pancreatic Cancers

Cancer research (Chicago, Ill.), 2020-04, Vol.80 (8), p.1630-1643

2020

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Oncogenic and wild-type Ras play divergent roles in the regulation of mitogen-activated protein kinase signaling

Cancer discovery, 2013-01, Vol.3 (1), p.112-123

2013

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Trends in Meniscus Repair and Meniscectomy in the United States, 2005-2011

The American journal of sports medicine, 2013-10, Vol.41 (10), p.2333-2339

2013

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Cancer gene therapy: fringe or cutting edge?

Nature reviews. Cancer, 2001-11, Vol.1 (2), p.130-141

McCormick, Frank

2001

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K-Ras Promotes Tumorigenicity through Suppression of Non-canonical Wnt Signaling

Cell, 2015-11, Vol.163 (5), p.1237-1251

2015

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Use of Hip Arthroscopy and Risk of Conversion to Total Hip Arthroplasty: A Population-Based Analysis

Arthroscopy, 2016-04, Vol.32 (4), p.587-593

2016

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Blockade of leukemia inhibitory factor as a therapeutic approach to KRAS driven pancreatic cancer

Nature communications, 2019-07, Vol.10 (1), p.3055-10, Article 3055

2019

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KRAS G13D sensitivity to neurofibromin-mediated GTP hydrolysis

Proceedings of the National Academy of Sciences - PNAS, 2019-10, Vol.116 (44), p.22122-22131

2019

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KRAS interaction with RAF1 RAS-binding domain and cysteine-rich domain provides insights into RAS-mediated RAF activation

Nature communications, 2021-02, Vol.12 (1), p.1176-1176, Article 1176

2021

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Sticking it to KRAS: Covalent Inhibitors Enter the Clinic

Cancer cell, 2020-01, Vol.37 (1), p.3-4

McCormick, Frank

2020

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Progress in targeting RAS with small molecule drugs

Biochemical journal, 2019-01, Vol.476 (2), p.365-374

McCormick, Frank

2019

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EGFR inhibition evokes innate drug resistance in lung cancer cells by preventing Akt activity and thus inactivating Ets-1 function

Proceedings of the National Academy of Sciences - PNAS, 2015-07, Vol.112 (29), p.E3855-E3863

2015

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Aurora kinase A drives the evolution of resistance to third-generation EGFR inhibitors in lung cancer

Nature medicine, 2019-01, Vol.25 (1), p.111-118

2019

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Trends in Medial Ulnar Collateral Ligament Reconstruction in the United States: A Retrospective Review of a Large Private-Payer Database From 2007 to 2011

The American journal of sports medicine, 2015-07, Vol.43 (7), p.1770-1774

2015

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Ras-GTP dimers activate the Mitogen-Activated Protein Kinase (MAPK) pathway

Proceedings of the National Academy of Sciences - PNAS, 2015-06, Vol.112 (26), p.7996-8001

2015

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The RAS GTPase RIT1 compromises mitotic fidelity through spindle assembly checkpoint suppression

Current biology, 2021-09, Vol.31 (17), p.3915-3924.e9

2021

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Glutamine Sensitivity Analysis Identifies the xCT Antiporter as a Common Triple-Negative Breast Tumor Therapeutic Target

Cancer cell, 2013-10, Vol.24 (4), p.450-465

2013

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KRAS as a Therapeutic Target

Targeting RAF kinases for cancer therapy: BRAF-mutated melanoma and beyond

Complications and Reoperations During and After Hip Arthroscopy: A Systematic Review of 92 Studies and More Than 6,000 Patients

The Metabolic Landscape of RAS-Driven Cancers from biology to therapy

The duality of human oncoproteins: drivers of cancer and congenital disorders

Trends in the Surgical Treatment of Articular Cartilage Lesions in the United States: An Analysis of a Large Private-Payer Database Over a Period of 8 Years

Long-Term Outcomes After Bankart Shoulder Stabilization

Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition

Undermining Glutaminolysis Bolsters Chemotherapy While NRF2 Promotes Chemoresistance in KRAS-Driven Pancreatic Cancers

Oncogenic and wild-type Ras play divergent roles in the regulation of mitogen-activated protein kinase signaling

Trends in Meniscus Repair and Meniscectomy in the United States, 2005-2011

Cancer gene therapy: fringe or cutting edge?

K-Ras Promotes Tumorigenicity through Suppression of Non-canonical Wnt Signaling

Use of Hip Arthroscopy and Risk of Conversion to Total Hip Arthroplasty: A Population-Based Analysis

Blockade of leukemia inhibitory factor as a therapeutic approach to KRAS driven pancreatic cancer

KRAS G13D sensitivity to neurofibromin-mediated GTP hydrolysis

KRAS interaction with RAF1 RAS-binding domain and cysteine-rich domain provides insights into RAS-mediated RAF activation

Sticking it to KRAS: Covalent Inhibitors Enter the Clinic

Progress in targeting RAS with small molecule drugs

EGFR inhibition evokes innate drug resistance in lung cancer cells by preventing Akt activity and thus inactivating Ets-1 function

Aurora kinase A drives the evolution of resistance to third-generation EGFR inhibitors in lung cancer

Trends in Medial Ulnar Collateral Ligament Reconstruction in the United States: A Retrospective Review of a Large Private-Payer Database From 2007 to 2011

Ras-GTP dimers activate the Mitogen-Activated Protein Kinase (MAPK) pathway

The RAS GTPase RIT1 compromises mitotic fidelity through spindle assembly checkpoint suppression

Glutamine Sensitivity Analysis Identifies the xCT Antiporter as a Common Triple-Negative Breast Tumor Therapeutic Target

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