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The contribution of intrinsically disordered regions to protein function, cellular complexity, and human disease

Biochemical Society transactions, 2016-10, Vol.44 (5), p.1185-1200

Babu, M Madan

2016

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Mechanisms of signalling and biased agonism in G protein-coupled receptors

Nature reviews. Molecular cell biology, 2018-10, Vol.19 (10), p.638-653

2018

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Cells alter their tRNA abundance to selectively regulate protein synthesis during stress conditions

Science signaling, 2018-09, Vol.11 (546)

2018

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GPCR activation mechanisms across classes and macro/microscales

Nature structural & molecular biology, 2021-11, Vol.28 (11), p.879-888

2021

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Data-driven design of orthogonal protein-protein interactions

Science signaling, 2023-02, Vol.16 (774), p.eabm4484

2023

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Common activation mechanism of class A GPCRs

eLife, 2019-12, Vol.8

2019

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Molecular signatures of G-protein-coupled receptors

Nature (London), 2013-02, Vol.494 (7436), p.185-194

2013

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Context-Specific Striatal Astrocyte Molecular Responses Are Phenotypically Exploitable

Neuron (Cambridge, Mass.), 2020-12, Vol.108 (6), p.1146-1162.e10

2020

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A community effort to bring structure to disorder

Nature methods, 2021-05, Vol.18 (5), p.454-455

2021

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Genomic analysis of regulatory network dynamics reveals large topological changes

Nature, 2004-09, Vol.431 (7006), p.308-312

2004

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A Million Peptide Motifs for the Molecular Biologist

Molecular cell, 2014-07, Vol.55 (2), p.161-169

2014

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An Expanded Oct4 Interaction Network: Implications for Stem Cell Biology, Development, and Disease

Cell stem cell, 2010-04, Vol.6 (4), p.382-395

2010

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Discovery and Genetic Code Expansion of a Polyethylene Terephthalate (PET) Hydrolase from the Human Saliva Metagenome for the Degradation and Bio‐Functionalization of PET

Angewandte Chemie International Edition, 2022-09, Vol.61 (37), p.e202203061-n/a

International ed. in English, 2022

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Promiscuity as a functional trait: intrinsically disordered regions as central players of interactomes

Biochemical journal, 2013-09, Vol.454 (3), p.361-369

2013

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Biological impact of mutually exclusive exon switching

PLoS computational biology, 2021-03, Vol.17 (3), p.e1008708-e1008708

2021

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Structural polymorphism in the N-terminal oligomerization domain of NPM1

Proceedings of the National Academy of Sciences - PNAS, 2014-03, Vol.111 (12), p.4466-4471

2014

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Molecular mechanism of modulating arrestin conformation by GPCR phosphorylation

Nature structural & molecular biology, 2018-06, Vol.25 (6), p.538-545

2018

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Visualization and analysis of non-covalent contacts using the Protein Contacts Atlas

Nature structural & molecular biology, 2018-02, Vol.25 (2), p.185-194

2018

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Tissue-Specific Splicing of Disordered Segments that Embed Binding Motifs Rewires Protein Interaction Networks

Molecular cell, 2012-06, Vol.46 (6), p.871-883

2012

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Intrinsically disordered proteins: regulation and disease

Current opinion in structural biology, 2011-06, Vol.21 (3), p.432-440

2011

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Use of Host-like Peptide Motifs in Viral Proteins Is a Prevalent Strategy in Host-Virus Interactions

Cell reports (Cambridge), 2014-06, Vol.7 (5), p.1729-1739

2014

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Classification of Intrinsically Disordered Regions and Proteins

Chemical reviews, 2014-07, Vol.114 (13), p.6589-6631

2014

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Tight Regulation of Unstructured Proteins: From Transcript Synthesis to Protein Degradation

Science (American Association for the Advancement of Science), 2008-11, Vol.322 (5906), p.1365-1368

2008

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Molecular determinants of ligand efficacy and potency in GPCR signaling

Science (American Association for the Advancement of Science), 2023-12, Vol.382 (6677), p.eadh1859-eadh1859

2023

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Diverse activation pathways in class A GPCRs converge near the G-protein-coupling region

Nature (London), 2016-08, Vol.536 (7617), p.484-487

2016

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The contribution of intrinsically disordered regions to protein function, cellular complexity, and human disease

Mechanisms of signalling and biased agonism in G protein-coupled receptors

Cells alter their tRNA abundance to selectively regulate protein synthesis during stress conditions

GPCR activation mechanisms across classes and macro/microscales

Data-driven design of orthogonal protein-protein interactions

Common activation mechanism of class A GPCRs

Molecular signatures of G-protein-coupled receptors

Context-Specific Striatal Astrocyte Molecular Responses Are Phenotypically Exploitable

A community effort to bring structure to disorder

Genomic analysis of regulatory network dynamics reveals large topological changes

A Million Peptide Motifs for the Molecular Biologist

An Expanded Oct4 Interaction Network: Implications for Stem Cell Biology, Development, and Disease

Discovery and Genetic Code Expansion of a Polyethylene Terephthalate (PET) Hydrolase from the Human Saliva Metagenome for the Degradation and Bio‐Functionalization of PET

Promiscuity as a functional trait: intrinsically disordered regions as central players of interactomes

Biological impact of mutually exclusive exon switching

Structural polymorphism in the N-terminal oligomerization domain of NPM1

Molecular mechanism of modulating arrestin conformation by GPCR phosphorylation

Visualization and analysis of non-covalent contacts using the Protein Contacts Atlas

Tissue-Specific Splicing of Disordered Segments that Embed Binding Motifs Rewires Protein Interaction Networks

Intrinsically disordered proteins: regulation and disease

Use of Host-like Peptide Motifs in Viral Proteins Is a Prevalent Strategy in Host-Virus Interactions

Classification of Intrinsically Disordered Regions and Proteins

Tight Regulation of Unstructured Proteins: From Transcript Synthesis to Protein Degradation

Molecular determinants of ligand efficacy and potency in GPCR signaling

Diverse activation pathways in class A GPCRs converge near the G-protein-coupling region

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