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Inhibition of MYC by the SMARCB1 tumor suppressor

Nature communications, 2019-05, Vol.10 (1), p.2014-2014, Article 2014

2019

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MYC regulates ribosome biogenesis and mitochondrial gene expression programs through its interaction with host cell factor-1

eLife, 2021-01, Vol.10

2021

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Displacement of WDR5 from Chromatin by a WIN Site Inhibitor with Picomolar Affinity

Cell reports (Cambridge), 2019-03, Vol.26 (11), p.2916-2928.e13

2019

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WIN site inhibition disrupts a subset of WDR5 function

Scientific reports, 2022-02, Vol.12 (1), p.1848-11, Article 1848

2022

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B cell responses to HIV antigen are a potent correlate of viremia in HIV-1 infection and improve with PD-1 blockade

PloS one, 2013-12, Vol.8 (12), p.e84185

2013

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Antiretroviral Therapy Reduces the Magnitude and T Cell Receptor Repertoire Diversity of HIV-Specific T Cell Responses without Changing T Cell Clonotype Dominance

Journal of Virology, 2012-04, Vol.86 (8), p.4213-4221

2012

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Synergistic action of WDR5 and HDM2 inhibitors in SMARCB1-deficient cancer cells

NAR cancer, 2022-03, Vol.4 (1)

2022

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Interaction of the oncoprotein transcription factor MYC with its chromatin cofactor WDR5 is essential for tumor maintenance

Proceedings of the National Academy of Sciences - PNAS, 2019-12, Vol.116 (50), p.25260-25268

2019

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WDR5 is a conserved regulator of protein synthesis gene expression

Nucleic acids research, 2020-04, Vol.48 (6), p.2924-2941

2020

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Enhancement of Human Immunodeficiency Virus (HIV)-Specific CD8+ T Cells in Cerebrospinal Fluid Compared to Those in Blood among Antiretroviral Therapy-Naïve HIV-Positive Subjects

Journal of Virology, 2008-11, Vol.82 (21), p.10418-10428

2008

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Multiple interactions of the oncoprotein transcription factor MYC with the SWI/SNF chromatin remodeler

Oncogene, 2021-05, Vol.40 (20), p.3593-3609

2021

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Structure-based discovery of potent WD repeat domain 5 inhibitors that demonstrate efficacy and safety in preclinical animal models

Proceedings of the National Academy of Sciences - PNAS, 2023-01, Vol.120 (1), p.e2211297120

2023

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Impact of WIN site inhibitor on the WDR5 interactome

Cell reports (Cambridge), 2021-01, Vol.34 (3), p.108636-108636, Article 108636

2021

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Enhanced PD-1 Expression by T Cells in Cerebrospinal Fluid Does Not Reflect Functional Exhaustion during Chronic Human Immunodeficiency Virus Type 1 Infection

Journal of Virology, 2010-01, Vol.84 (1), p.131-140

2010

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Clonal expansion and TCR-independent differentiation shape the HIV-specific CD8+ effector-memory T-cell repertoire in vivo

Blood, 2010-07, Vol.116 (3), p.396-405

2010

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Despite Biased TRBV Gene Usage against a Dominant HLA B57-Restricted Epitope, TCR Diversity Can Provide Recognition of Circulating Epitope Variants

The Journal of immunology (1950), 2008-10, Vol.181 (7), p.5137-5146

2008

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Dominant clonotypes within HIV-specific T cell responses are programmed death-1high and CD127low and display reduced variant cross-reactivity

The Journal of immunology (1950), 2011-06, Vol.186 (12), p.6871-6885

2011

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A common functional consequence of tumor-derived mutations within c-MYC

Oncogene, 2015-04, Vol.34 (18), p.2406-2409

2015

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Abstract LB-206: Inhibition of MYC by the SMARCB1 tumor suppressor

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.LB-206-LB-206

2018

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Autocrine Role of Macrophage Inflammatory Protein-1 β in Human T-Cell Lymphotropic Virus Type-I tax-Transfected Jurkat T-Cells

Biochemical and biophysical research communications, 2001-10, Vol.287 (4), p.910-913

2001

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Displacement of WDR5 from Chromatin by a WIN Site Inhibitor with Picomolar Affinity

Cell reports (Cambridge), 2019-03, Vol.26 (11)

2019

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Dominant clonotypes within HIV-specific T cell responses are PD-1hi and CD127low and display reduced variant cross-reactivity1

The Journal of immunology (1950), 2011-05, Vol.186 (12), p.6871-6885

2011

Volltextzugriff (PDF)

Enhancement of Human Immunodeficiency Virus (HIV)-Specific CD8 + T Cells in Cerebrospinal Fluid Compared to Those in Blood among Antiretroviral Therapy-Naïve HIV-Positive Subjects

Journal of virology, 2008-11, Vol.82 (21), p.10418-10428

2008

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Inhibition of MYC by the SMARCB1 tumor suppressor

MYC regulates ribosome biogenesis and mitochondrial gene expression programs through its interaction with host cell factor-1

Displacement of WDR5 from Chromatin by a WIN Site Inhibitor with Picomolar Affinity

WIN site inhibition disrupts a subset of WDR5 function

Ribosome subunit attrition and activation of the p53-MDM4 axis dominate the response of MLL-rearranged cancer cells to WDR5 WIN site inhibition

B cell responses to HIV antigen are a potent correlate of viremia in HIV-1 infection and improve with PD-1 blockade

Antiretroviral Therapy Reduces the Magnitude and T Cell Receptor Repertoire Diversity of HIV-Specific T Cell Responses without Changing T Cell Clonotype Dominance

Synergistic action of WDR5 and HDM2 inhibitors in SMARCB1-deficient cancer cells

Interaction of the oncoprotein transcription factor MYC with its chromatin cofactor WDR5 is essential for tumor maintenance

WDR5 is a conserved regulator of protein synthesis gene expression

Enhancement of Human Immunodeficiency Virus (HIV)-Specific CD8+ T Cells in Cerebrospinal Fluid Compared to Those in Blood among Antiretroviral Therapy-Naïve HIV-Positive Subjects

Multiple interactions of the oncoprotein transcription factor MYC with the SWI/SNF chromatin remodeler

Structure-based discovery of potent WD repeat domain 5 inhibitors that demonstrate efficacy and safety in preclinical animal models

Impact of WIN site inhibitor on the WDR5 interactome

Enhanced PD-1 Expression by T Cells in Cerebrospinal Fluid Does Not Reflect Functional Exhaustion during Chronic Human Immunodeficiency Virus Type 1 Infection

Clonal expansion and TCR-independent differentiation shape the HIV-specific CD8+ effector-memory T-cell repertoire in vivo

Despite Biased TRBV Gene Usage against a Dominant HLA B57-Restricted Epitope, TCR Diversity Can Provide Recognition of Circulating Epitope Variants

Dominant clonotypes within HIV-specific T cell responses are programmed death-1high and CD127low and display reduced variant cross-reactivity

A common functional consequence of tumor-derived mutations within c-MYC

Abstract LB-206: Inhibition of MYC by the SMARCB1 tumor suppressor

Ribosome subunit attrition and activation of the p53–MDM4 axis dominate the response of MLL-rearranged cancer cells to WDR5 WIN site inhibition

Autocrine Role of Macrophage Inflammatory Protein-1 β in Human T-Cell Lymphotropic Virus Type-I tax-Transfected Jurkat T-Cells

Displacement of WDR5 from Chromatin by a WIN Site Inhibitor with Picomolar Affinity

Dominant clonotypes within HIV-specific T cell responses are PD-1hi and CD127low and display reduced variant cross-reactivity1

Enhancement of Human Immunodeficiency Virus (HIV)-Specific CD8 + T Cells in Cerebrospinal Fluid Compared to Those in Blood among Antiretroviral Therapy-Naïve HIV-Positive Subjects

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