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Induction of heat shock proteins for protection against oxidative stress

Advanced drug delivery reviews, 2009-04, Vol.61 (4), p.310-318

2009

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Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS

Nature communications, 2018-05, Vol.9 (1), p.2010-15, Article 2010

2018

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The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol

Pharmacology & therapeutics (Oxford), 2014, Vol.141 (1), p.40-54

2014

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Cellular Chaperones As Therapeutic Targets in ALS to Restore Protein Homeostasis and Improve Cellular Function

Frontiers in molecular neuroscience, 2017-09, Vol.10, p.251-251

2017

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Circulating miR-181 is a prognostic biomarker for amyotrophic lateral sclerosis

Nature neuroscience, 2021-11, Vol.24 (11), p.1534-1541

2021

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Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis

Brain (London, England : 1878), 2019-09, Vol.142 (9), p.2572-2580

2019

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Deficits in axonal transport precede ALS symptoms in vivo

Proceedings of the National Academy of Sciences - PNAS, 2010-11, Vol.107 (47), p.20523-20528

2010

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Optical Control of Muscle Function by Transplantation of Stem Cell–Derived Motor Neurons in Mice

Science (American Association for the Advancement of Science), 2014-04, Vol.344 (6179), p.94-97

2014

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Enhanced Expression of TRAP1 Protects Mitochondrial Function in Motor Neurons under Conditions of Oxidative Stress

International journal of molecular sciences, 2022-02, Vol.23 (3), p.1789

2022

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c-Jun Reprograms Schwann Cells of Injured Nerves to Generate a Repair Cell Essential for Regeneration

Neuron (Cambridge, Mass.), 2012-08, Vol.75 (4), p.633-647

2012

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Astrocytes display cell autonomous and diverse early reactive states in familial amyotrophic lateral sclerosis

Brain (London, England : 1878), 2022-04, Vol.145 (2), p.481-489

2022

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Humanized mutant FUS drives progressive motor neuron degeneration without aggregation in ‘FUSDelta14’ knockin mice

Brain (London, England : 1878), 2017-11, Vol.140 (11), p.2797-2805

2017

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Inhibiting p38 MAPK alpha rescues axonal retrograde transport defects in a mouse model of ALS

Cell death & disease, 2018-05, Vol.9 (6), p.596-16, Article 596

2018

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Overexpression of human wild-type FUS causes progressive motor neuron degeneration in an age- and dose-dependent fashion

Acta neuropathologica, 2013-02, Vol.125 (2), p.273-288

2013

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An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

eLife, 2024-01, Vol.12

2024

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Plasma neurofilament heavy chain levels and disease progression in amyotrophic lateral sclerosis: insights from a longitudinal study

Journal of neurology, neurosurgery and psychiatry, 2015-05, Vol.86 (5), p.565-573

2015

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Hereditary sensory neuropathy type 1-associated deoxysphingolipids cause neurotoxicity, acute calcium handling abnormalities and mitochondrial dysfunction in vitro

Neurobiology of disease, 2018-09, Vol.117, p.1-14

2018

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Targeting protein homeostasis in sporadic inclusion body myositis

Science translational medicine, 2016-03, Vol.8 (331), p.331ra41

2016

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Nidogens are therapeutic targets for the prevention of tetanus

Science (American Association for the Advancement of Science), 2014-11, Vol.346 (6213), p.1118-1123

2014

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Opinion: more mouse models and more translation needed for ALS

Molecular neurodegeneration, 2023-05, Vol.18 (1), p.30-30, Article 30

2023

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Senataxin mutations elicit motor neuron degeneration phenotypes and yield TDP-43 mislocalization in ALS4 mice and human patients

Acta neuropathologica, 2018-09, Vol.136 (3), p.425-443

2018

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Gene expression analysis reveals early dysregulation of disease pathways and links Chmp7 to pathogenesis of spinal and bulbar muscular atrophy

Scientific reports, 2019-03, Vol.9 (1), p.3539-3539, Article 3539

2019

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Correlation of clinical and molecular features in spinal bulbar muscular atrophy

Neurology, 2014-06, Vol.82 (23), p.2077-2084

2014

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Systemic inflammatory response and neuromuscular involvement in amyotrophic lateral sclerosis

Neurology : neuroimmunology & neuroinflammation, 2016-08, Vol.3 (4), p.e244-e244

2016

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Neuromuscular Junction Formation in Tissue-Engineered Skeletal Muscle Augments Contractile Function and Improves Cytoskeletal Organization

Tissue engineering. Part A, 2015-10, Vol.21 (19-20), p.2595-2604

2015

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Induction of heat shock proteins for protection against oxidative stress

Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS

The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol

Cellular Chaperones As Therapeutic Targets in ALS to Restore Protein Homeostasis and Improve Cellular Function

Circulating miR-181 is a prognostic biomarker for amyotrophic lateral sclerosis

Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis

Deficits in axonal transport precede ALS symptoms in vivo

Optical Control of Muscle Function by Transplantation of Stem Cell–Derived Motor Neurons in Mice

Enhanced Expression of TRAP1 Protects Mitochondrial Function in Motor Neurons under Conditions of Oxidative Stress

c-Jun Reprograms Schwann Cells of Injured Nerves to Generate a Repair Cell Essential for Regeneration

Astrocytes display cell autonomous and diverse early reactive states in familial amyotrophic lateral sclerosis

Humanized mutant FUS drives progressive motor neuron degeneration without aggregation in ‘FUSDelta14’ knockin mice

Inhibiting p38 MAPK alpha rescues axonal retrograde transport defects in a mouse model of ALS

Overexpression of human wild-type FUS causes progressive motor neuron degeneration in an age- and dose-dependent fashion

An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

Plasma neurofilament heavy chain levels and disease progression in amyotrophic lateral sclerosis: insights from a longitudinal study

Hereditary sensory neuropathy type 1-associated deoxysphingolipids cause neurotoxicity, acute calcium handling abnormalities and mitochondrial dysfunction in vitro

Targeting protein homeostasis in sporadic inclusion body myositis

Nidogens are therapeutic targets for the prevention of tetanus

Opinion: more mouse models and more translation needed for ALS

Senataxin mutations elicit motor neuron degeneration phenotypes and yield TDP-43 mislocalization in ALS4 mice and human patients

Gene expression analysis reveals early dysregulation of disease pathways and links Chmp7 to pathogenesis of spinal and bulbar muscular atrophy

Correlation of clinical and molecular features in spinal bulbar muscular atrophy

Systemic inflammatory response and neuromuscular involvement in amyotrophic lateral sclerosis

Neuromuscular Junction Formation in Tissue-Engineered Skeletal Muscle Augments Contractile Function and Improves Cytoskeletal Organization

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