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The substrate repertoire of γ-secretase/presenilin

Seminars in cell & developmental biology, 2020-09, Vol.105, p.27-42

2020

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The alpha secretase ADAM10: A metalloprotease with multiple functions in the brain

Progress in neurobiology, 2015-12, Vol.135, p.1-20

2015

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Proteolytic ectodomain shedding of membrane proteins in mammals—hardware, concepts, and recent developments

The EMBO journal, 2018-08, Vol.37 (15), p.n/a

2018

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Alpha-secretase in Alzheimer's disease: molecular identity, regulation and therapeutic potential

Journal of neurochemistry, 2011-01, Vol.116 (1), p.10-21

Lichtenthaler, Stefan F

2011

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SHEDDING OF TREM2 AND APP AS DRUG TARGETS IN ALZHEIMER’S DISEASE

IBRO neuroscience reports, 2023-10, Vol.15, p.S53-S53

Lichtenthaler, Stefan

2023

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BACE1 Physiological Functions May Limit Its Use as Therapeutic Target for Alzheimer's Disease

Trends in neurosciences (Regular ed.), 2016-03, Vol.39 (3), p.158-169

2016

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The case for low-level BACE1 inhibition for the prevention of Alzheimer disease

Nature reviews. Neurology, 2021-11, Vol.17 (11), p.703

2021

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Alpha-secretase cleavage of the amyloid precursor protein: proteolysis regulated by signaling pathways and protein trafficking

Current Alzheimer research, 2012-02, Vol.9 (2), p.165

Lichtenthaler, Stefan F

2012

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Functions of ‘A disintegrin and metalloproteases (ADAMs)’ in the mammalian nervous system

Cellular and molecular life sciences : CMLS, 2019-08, Vol.76 (16), p.3055-3081

2019

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Regulated intramembrane proteolysis – lessons from amyloid precursor protein processing

Journal of neurochemistry, 2011-06, Vol.117 (5), p.779-796

2011

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Secretases in Alzheimer's disease: Novel insights into proteolysis of APP and TREM2

Current opinion in neurobiology, 2022-02, Vol.72, p.101-110

2022

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An Alzheimer‐associated TREM2 variant occurs at the ADAM cleavage site and affects shedding and phagocytic function

EMBO molecular medicine, 2017-10, Vol.9 (10), p.1356-1365

2017

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Cell-type-specific profiling of brain mitochondria reveals functional and molecular diversity

Nature neuroscience, 2019-10, Vol.22 (10), p.1731

2019

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Function, therapeutic potential and cell biology of BACE proteases: current status and future prospects

Journal of neurochemistry, 2014-07, Vol.130 (1), p.4-28

2014

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To cut or not to cut: New rules for proteolytic shedding of membrane proteins

The Journal of biological chemistry, 2020-08, Vol.295 (35), p.12353-12355

2020

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The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO

Molecular cell, 2013-03, Vol.49 (5), p.908-921

2013

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ADAM10 and ADAM17 promote SARS‐CoV‐2 cell entry and spike protein‐mediated lung cell fusion

EMBO reports, 2022-06, Vol.23 (6), p.e54305-n/a

2022

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Degradome of soluble ADAM10 and ADAM17 metalloproteases

Cellular and molecular life sciences : CMLS, 2020-01, Vol.77 (2), p.331-350

2020

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Shedding of BAFF/APRIL Receptors Controls B Cells

Trends in immunology, 2018-09, Vol.39 (9), p.673-676

2018

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Highly efficient intercellular spreading of protein misfolding mediated by viral ligand-receptor interactions

Nature communications, 2021-10, Vol.12 (1), p.5739-5739, Article 5739

2021

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Amyloid-beta mediates homeostatic synaptic plasticity

The Journal of neuroscience, 2021-06, Vol.41 (24), p.5157-5172

2021

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ADAM10 is the physiologically relevant, constitutive α-secretase of the amyloid precursor protein in primary neurons

The EMBO journal, 2010-09, Vol.29 (17), p.3020-3032

2010

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Loss of NPC1 enhances phagocytic uptake and impairs lipid trafficking in microglia

Nature communications, 2021-02, Vol.12 (1), p.1158-1158, Article 1158

2021

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Pro-inflammatory activation following demyelination is required for myelin clearance and oligodendrogenesis

The Journal of experimental medicine, 2020-05, Vol.217 (5)

2020

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The substrate repertoire of γ-secretase/presenilin

The alpha secretase ADAM10: A metalloprotease with multiple functions in the brain

Proteolytic ectodomain shedding of membrane proteins in mammals—hardware, concepts, and recent developments

Alpha-secretase in Alzheimer's disease: molecular identity, regulation and therapeutic potential

SHEDDING OF TREM2 AND APP AS DRUG TARGETS IN ALZHEIMER’S DISEASE

BACE1 Physiological Functions May Limit Its Use as Therapeutic Target for Alzheimer's Disease

The case for low-level BACE1 inhibition for the prevention of Alzheimer disease

Alpha-secretase cleavage of the amyloid precursor protein: proteolysis regulated by signaling pathways and protein trafficking

Functions of ‘A disintegrin and metalloproteases (ADAMs)’ in the mammalian nervous system

Regulated intramembrane proteolysis – lessons from amyloid precursor protein processing

Secretases in Alzheimer's disease: Novel insights into proteolysis of APP and TREM2

An Alzheimer‐associated TREM2 variant occurs at the ADAM cleavage site and affects shedding and phagocytic function

Cell-type-specific profiling of brain mitochondria reveals functional and molecular diversity

Function, therapeutic potential and cell biology of BACE proteases: current status and future prospects

To cut or not to cut: New rules for proteolytic shedding of membrane proteins

The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO

ADAM10 and ADAM17 promote SARS‐CoV‐2 cell entry and spike protein‐mediated lung cell fusion

Degradome of soluble ADAM10 and ADAM17 metalloproteases

Shedding of BAFF/APRIL Receptors Controls B Cells

Highly efficient intercellular spreading of protein misfolding mediated by viral ligand-receptor interactions

Amyloid-beta mediates homeostatic synaptic plasticity

ADAM10 is the physiologically relevant, constitutive α-secretase of the amyloid precursor protein in primary neurons

Fibrillar Aβ triggers microglial proteome alterations and dysfunction in Alzheimer mouse models

Loss of NPC1 enhances phagocytic uptake and impairs lipid trafficking in microglia

Pro-inflammatory activation following demyelination is required for myelin clearance and oligodendrogenesis

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