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RIP3 Induces Apoptosis Independent of Pronecrotic Kinase Activity

Molecular cell, 2014-11, Vol.56 (4), p.481-495

2014

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NLRP12 Suppresses Colon Inflammation and Tumorigenesis through the Negative Regulation of Noncanonical NF-κB Signaling

Immunity (Cambridge, Mass.), 2012-05, Vol.36 (5), p.742-754

2012

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Autolytic Proteolysis within the Function to Find Domain (FIIND) Is Required for NLRP1 Inflammasome Activity

The Journal of biological chemistry, 2012-07, Vol.287 (30), p.25030-25037

2012

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NLRX1 is a regulator of mitochondrial antiviral immunity

Nature, 2008-01, Vol.451 (7178), p.573-577

2008

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Identification of a RIP1 Kinase Inhibitor Clinical Candidate (GSK3145095) for the Treatment of Pancreatic Cancer

ACS medicinal chemistry letters, 2019-06, Vol.10 (6), p.857-862

2019

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Cutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivity

The Journal of immunology (1950), 2010-10, Vol.185 (8), p.4515-4519

2010

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Characterization of NLRP12 during the in vivo host immune response to Klebsiella pneumoniae and Mycobacterium tuberculosis

PloS one, 2013-04, Vol.8 (4), p.e60842-e60842

2013

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ATP Binding by Monarch-1/NLRP12 Is Critical for Its Inhibitory Function

Molecular and Cellular Biology, 2008-03, Vol.28 (5), p.1841-1850

2008

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Characterization of NLRP12 during the development of allergic airway disease in mice

PloS one, 2012-01, Vol.7 (1), p.e30612

2012

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Cutting Edge: ASC Mediates the Induction of Multiple Cytokines by Porphyromonas gingivalis via Caspase-1-Dependent and -Independent Pathways

The Journal of immunology (1950), 2006-10, Vol.177 (7), p.4252-4256

2006

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Semaphorin 6D regulates the late phase of CD4⁺ T cell primary immune responses

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (35), p.13015-13020

2008

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Criteria for effective design, construction, and gene knockdown by shRNA vectors

BMC biotechnology, 2006-01, Vol.6 (1), p.7-7, Article 7

2006

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G-CSF drives autoinflammation in APLAID

Nature immunology, 2023-05, Vol.24 (5), p.814-826

2023

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Discovery of Small Molecule RIP1 Kinase Inhibitors for the Treatment of Pathologies Associated with Necroptosis

ACS medicinal chemistry letters, 2013-12, Vol.4 (12), p.1238-1243

2013

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Cutting Edge: Monarch-1 Suppresses Non-Canonical NF-κB Activation and p52-Dependent Chemokine Expression in Monocytes

Journal of Immunology, 2007-02, Vol.178 (3), p.1256-1260

2007

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The CATERPILLER Protein Monarch-1 Is an Antagonist of Toll-like Receptor-, Tumor Necrosis Factor α-, and Mycobacterium tuberculosis-induced Pro-inflammatory Signals

The Journal of biological chemistry, 2005-12, Vol.280 (48), p.39914-39924

2005

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Cutting Edge: Editing of Recycling Class II:Peptide Complexes by HLA-DM

The Journal of immunology (1950), 2001-07, Vol.167 (2), p.632-635

2001

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Proteomic profiling drug-induced apoptosis in non-small cell lung carcinoma: Identification of RS/DJ-1 and RhoGDIα

Cancer research (Chicago, Ill.), 2003-10, Vol.63 (20), p.6928-6934

2003

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Heat Shock Protein 90 Associates with Monarch-1 and Regulates Its Ability to Promote Degradation of NF-κB-Inducing Kinase

The Journal of immunology (1950), 2007-11, Vol.179 (9), p.6291-6296

2007

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Monarch-1/ PYPAF7 and other CATERPILLER (CLR, NOD, NLR) proteins with negative regulatory functions

Microbes and infection, 2007-04, Vol.9 (5), p.672-676

2007

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Cytoplasmic processing is a prerequisite for presentation of an endogenous antigen by major histocompatibility complex class II proteins

The Journal of experimental medicine, 2000-05, Vol.191 (9), p.1513-1524

2000

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Correction: Characterization of NLRP12 during the In Vivo Host Immune Response to Klebsiella pneumoniae and Mycobacterium tuberculosis

PloS one, 2013-11, Vol.8 (11)

2013

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Autolytic proteolysis within the function to find domain (FIIND) is required for NLRP1 inflammasome activity

The Journal of biological chemistry, 2012-09, Vol.287 (37), p.31456-31456

2012

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RIP3 Induces Apoptosis Independent of Pronecrotic Kinase Activity

NLRP12 Suppresses Colon Inflammation and Tumorigenesis through the Negative Regulation of Noncanonical NF-κB Signaling

Autolytic Proteolysis within the Function to Find Domain (FIIND) Is Required for NLRP1 Inflammasome Activity

NLRX1 is a regulator of mitochondrial antiviral immunity

Identification of a RIP1 Kinase Inhibitor Clinical Candidate (GSK3145095) for the Treatment of Pancreatic Cancer

Cutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivity

Characterization of NLRP12 during the in vivo host immune response to Klebsiella pneumoniae and Mycobacterium tuberculosis

ATP Binding by Monarch-1/NLRP12 Is Critical for Its Inhibitory Function

Characterization of NLRP12 during the development of allergic airway disease in mice

RIP1 Kinase Drives Macrophage-Mediated Adaptive Immune Tolerance in Pancreatic Cancer

Cutting Edge: ASC Mediates the Induction of Multiple Cytokines by Porphyromonas gingivalis via Caspase-1-Dependent and -Independent Pathways

Semaphorin 6D regulates the late phase of CD4⁺ T cell primary immune responses

Criteria for effective design, construction, and gene knockdown by shRNA vectors

G-CSF drives autoinflammation in APLAID

RIP1 Kinase Drives Macrophage-Mediated Adaptive Immune Tolerance in Pancreatic Cancer

Discovery of Small Molecule RIP1 Kinase Inhibitors for the Treatment of Pathologies Associated with Necroptosis

Cutting Edge: Monarch-1 Suppresses Non-Canonical NF-κB Activation and p52-Dependent Chemokine Expression in Monocytes

The CATERPILLER Protein Monarch-1 Is an Antagonist of Toll-like Receptor-, Tumor Necrosis Factor α-, and Mycobacterium tuberculosis-induced Pro-inflammatory Signals

Cutting Edge: Editing of Recycling Class II:Peptide Complexes by HLA-DM

Proteomic profiling drug-induced apoptosis in non-small cell lung carcinoma: Identification of RS/DJ-1 and RhoGDIα

Heat Shock Protein 90 Associates with Monarch-1 and Regulates Its Ability to Promote Degradation of NF-κB-Inducing Kinase

Monarch-1/ PYPAF7 and other CATERPILLER (CLR, NOD, NLR) proteins with negative regulatory functions

Cytoplasmic processing is a prerequisite for presentation of an endogenous antigen by major histocompatibility complex class II proteins

Correction: Characterization of NLRP12 during the In Vivo Host Immune Response to Klebsiella pneumoniae and Mycobacterium tuberculosis

Autolytic proteolysis within the function to find domain (FIIND) is required for NLRP1 inflammasome activity

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