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P-05-1 A Penile Implant Patient: The Benefits of a Prosthetic Nurse

Journal of sexual medicine, 2020-06, Vol.17 (6), p.S206-S206

2020

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Cardiac complications of type 2 diabetes

Revue médicale de Bruxelles, 2000-06, Vol.21 (3), p.149

Lenaers, M

2000

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Anaphylactoid reactions during hemodialysis on AN69 membranes in patients receiving ACE inhibitors

Kidney international, 1990-11, Vol.38 (5), p.982-984

1990

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TRPV4 channels mediate the infrared laser-evoked response in sensory neurons

Journal of neurophysiology, 2012-06, Vol.107 (12), p.3227-3234

2012

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Multi-system neurological disease is common in patients with OPA1 mutations

Brain (London, England : 1878), 2010-03, Vol.133 (3), p.771-786

2010

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STochastic Optical Reconstruction Microscopy (STORM) reveals the nanoscale organization of pathological aggregates in human brain

Neuropathology and applied neurobiology, 2021-02, Vol.47 (1), p.127-142

2021

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Physiology of PNS axons relies on glycolytic metabolism in myelinating Schwann cells

PloS one, 2022-10, Vol.17 (10), p.e0272097-e0272097

2022

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Correlation between physical, electrical, and optical properties of Cu2ZnSnSe4 based solar cells

Applied physics letters, 2013-01, Vol.102 (1)

2013

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A case report of two Moroccan patients with hereditary neurological disorders and molecular modeling study on the S72L de novo PMP22 variant

Revue neurologique, 2023-10, Vol.179 (8), p.902-909

2023

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Mitochondrial Complex I Disruption Causes Broad Reorchestration of Plant Lipidome Including Chloroplast Lipids

International journal of molecular sciences, 2022-12, Vol.24 (1), p.453

2022

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Glycosylation as new pharmacological strategies for diseases associated with excessive angiogenesis

Pharmacology & therapeutics (Oxford), 2018-11, Vol.191, p.92-122

2018

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TEFM variants impair mitochondrial transcription causing childhood-onset neurological disease

Nature communications, 2023-02, Vol.14 (1), p.1009-1009, Article 1009

2023

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WFS1 in Optic Neuropathies: Mutation Findings in Nonsyndromic Optic Atrophy and Assessment of Clinical Severity

Ophthalmology (Rochester, Minn.), 2016-09, Vol.123 (9), p.1989-1998

2016

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Nuclear gene OPA1 , encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy

Nature genetics, 2000-10, Vol.26 (2), p.207-210

2000

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Tailoring Interlayer Charge Transfer Dynamics in 2D Perovskites with Electroactive Spacer Molecules

Journal of the American Chemical Society, 2023-10, Vol.145 (39), p.21330-21343

2023

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3-Methyl 5-{3-[(4-Methylbenzenesulfonyl)oxy]propyl} 4-(2,3-Dichlorophenyl)-2,6-dimethyl-1,4-dihydropyridine-3,5-dicarboxylate

MolBank, 2022-12, Vol.2022 (4), p.M1460

2022

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Milk Urea Nitrogen as a Tool to Monitor the Protein Nutrition of Dairy Cows

Journal of dairy science, 1997-12, Vol.80 (12), p.3333-3340

1997

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Mutation spectrum and splicing variants in the OPA1 gene

Human genetics, 2001-12, Vol.109 (6), p.584-591

2001

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3-Methyl 5-{3-[-2,6-dimethyl-1,4-dihydropyridine-3,5-dicarboxylate

MolBank, 2022-10, Vol.2022 (4)

2022

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GNE Myopathy and Duchenne Muscular Dystrophy in Two Moroccans Families

Muscles, Ligaments and Tendons Journal, 2023-10, Vol.13 (4), p.653

2023

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A Data Mining Metabolomics Exploration of Glaucoma

Metabolites, 2020-01, Vol.10 (2), p.49

2020

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Biallelic Variants in UBA5 Reveal that Disruption of the UFM1 Cascade Can Result in Early-Onset Encephalopathy

American journal of human genetics, 2016-09, Vol.99 (3), p.695-703

2016

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Expanding the FDXR-Associated Disease Phenotype: Retinal Dystrophy Is a Recurrent Ocular Feature

Investigative ophthalmology & visual science, 2021-05, Vol.62 (6), p.2-2

2021

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L’étude de l’ultrastructure des inclusions protéiques dans l’atrophie multi-systématisée suggère une propagation prion-like de l’α-synucléine au sein des oligodendrocytes et des neurones

Revue neurologique, 2023-04, Vol.179, p.S71-S72

2023

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P-05-1 A Penile Implant Patient: The Benefits of a Prosthetic Nurse

Cardiac complications of type 2 diabetes

Anaphylactoid reactions during hemodialysis on AN69 membranes in patients receiving ACE inhibitors

TRPV4 channels mediate the infrared laser-evoked response in sensory neurons

Multi-system neurological disease is common in patients with OPA1 mutations

OPA1 mutations induce mitochondrial DNA instability and optic atrophy ‘plus’ phenotypes

STochastic Optical Reconstruction Microscopy (STORM) reveals the nanoscale organization of pathological aggregates in human brain

Physiology of PNS axons relies on glycolytic metabolism in myelinating Schwann cells

Correlation between physical, electrical, and optical properties of Cu2ZnSnSe4 based solar cells

A case report of two Moroccan patients with hereditary neurological disorders and molecular modeling study on the S72L de novo PMP22 variant

Mitochondrial Complex I Disruption Causes Broad Reorchestration of Plant Lipidome Including Chloroplast Lipids

Glycosylation as new pharmacological strategies for diseases associated with excessive angiogenesis

TEFM variants impair mitochondrial transcription causing childhood-onset neurological disease

WFS1 in Optic Neuropathies: Mutation Findings in Nonsyndromic Optic Atrophy and Assessment of Clinical Severity

Nuclear gene OPA1 , encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy

Tailoring Interlayer Charge Transfer Dynamics in 2D Perovskites with Electroactive Spacer Molecules

3-Methyl 5-{3-[(4-Methylbenzenesulfonyl)oxy]propyl} 4-(2,3-Dichlorophenyl)-2,6-dimethyl-1,4-dihydropyridine-3,5-dicarboxylate

Milk Urea Nitrogen as a Tool to Monitor the Protein Nutrition of Dairy Cows

Mutation spectrum and splicing variants in the OPA1 gene

3-Methyl 5-{3-[-2,6-dimethyl-1,4-dihydropyridine-3,5-dicarboxylate

GNE Myopathy and Duchenne Muscular Dystrophy in Two Moroccans Families

A Data Mining Metabolomics Exploration of Glaucoma

Biallelic Variants in UBA5 Reveal that Disruption of the UFM1 Cascade Can Result in Early-Onset Encephalopathy

Expanding the FDXR-Associated Disease Phenotype: Retinal Dystrophy Is a Recurrent Ocular Feature

L’étude de l’ultrastructure des inclusions protéiques dans l’atrophie multi-systématisée suggère une propagation prion-like de l’α-synucléine au sein des oligodendrocytes et des neurones

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